QRS Duration in Supraventricular Tachycardia
In supraventricular tachycardia (SVT), the QRS complex duration is typically narrow, defined as less than 120 milliseconds. 1
Narrow QRS Complex: The Hallmark of SVT
When the QRS duration is less than 120 ms, the tachycardia is almost always supraventricular in origin. 1
This narrow QRS complex reflects normal ventricular activation through the His-Purkinje system, distinguishing SVT from ventricular tachycardia in most cases. 2
The narrow complex definition applies to all major SVT subtypes including AVNRT (atrioventricular nodal reentrant tachycardia), AVRT (atrioventricular reciprocating tachycardia), and atrial tachycardia. 1
Important Exceptions: Wide QRS Complex in SVT
SVT can present with a wide QRS complex (≥120 ms) in specific circumstances, which creates diagnostic challenges:
Three Mechanisms for Wide QRS in SVT:
Pre-existing or rate-related bundle branch block (BBB) 1
- Bundle branch block may exist prior to tachycardia or develop during SVT when one bundle branch becomes refractory due to the rapid rate
- Most rate-related BBB occurs due to long-short sequence initiation
- QRS width >140 ms with RBBB pattern or >160 ms with LBBB pattern favors ventricular tachycardia over SVT with aberrancy 1
Anterograde conduction over an accessory pathway (pre-excitation) 1
- Occurs during atrial tachycardia, atrial flutter, atrial fibrillation, AVNRT, or antidromic AVRT
- In pre-excited tachycardias, the QRS is generally wider (more pre-excited) compared to sinus rhythm 1
Antiarrhythmic drug effects 1
- Class Ic or Class Ia antiarrhythmic drugs can widen the QRS complex during SVT
- QRS width criteria become less helpful for differentiating SVT from VT in patients taking these medications 1
Clinical Implications
The critical diagnostic threshold is 120 ms: narrow QRS (<120 ms) strongly suggests SVT, while wide QRS (≥120 ms) requires careful differentiation from ventricular tachycardia. 1
When encountering wide QRS tachycardia and the diagnosis cannot be definitively established, the patient should be treated as having ventricular tachycardia due to the potentially life-threatening consequences of misdiagnosis. 1
Intravenous medications for SVT (particularly verapamil or diltiazem) may precipitate hemodynamic collapse if administered to a patient with ventricular tachycardia. 1