Can hypokalemia (low potassium levels) occur after a myocardial infarction (MI)?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: May 21, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

From the FDA Drug Label

Table 2. Hypokalemia (<3.5 mEq/L) or Hyperkalemia (>5.5 or ≥6.0 mEq/L) in EPHESUS Potassium (mEq/L) Eplerenone (N=3,251) n (%) Placebo (N=3,237) n (%) <3.5 273 (8.4) 424 (13.1)

5.5 508 (15.6) 363 (11.2) ≥6.0 180 (5.5) 126 (3. 9)

The answer to the question is yes, hypokalemia can occur after an MI.

  • The frequency of hypokalemia (<3.5 mEq/L) in the eplerenone group was 8.4% compared to 13.1% in the placebo group 1.
  • This suggests that while hypokalemia is possible after an MI, the use of eplerenone may actually decrease its frequency.

From the Research

Yes, hypokalemia can occur after a myocardial infarction (MI). Potassium levels often drop during the acute phase of an MI due to several mechanisms, including stress-induced catecholamine release, which drives potassium into cells, lowering serum levels 2. Additionally, common treatments for MI, such as beta-agonists, insulin-glucose infusions, and diuretics, especially loop diuretics like furosemide, can further deplete potassium. This is concerning because hypokalemia increases the risk of dangerous arrhythmias in post-MI patients, particularly ventricular fibrillation and torsades de pointes.

The most recent and highest quality study, published in 2022, highlights the importance of monitoring potassium levels in patients taking diuretics, as diuretic-induced hypokalaemia is a common and potentially life-threatening adverse drug reaction 3. The study emphasizes that reducing diuretic dose and potassium supplementation are the most direct and effective therapies for hypokalaemia.

Key points to consider:

  • Hypokalemia is a common problem in patients with acute myocardial infarction, with a prevalence of 14% at admission 4.
  • The presence of hypokalemia is associated with an increased frequency of ventricular tachycardia and ventricular fibrillation 4.
  • Mortality, both short and long term, and the occurrence of ventricular arrhythmias in patients with acute myocardial infarction seem to be negatively associated with hypokalemic serum potassium concentration 2.
  • Clinicians should monitor potassium levels closely after MI and maintain them between 4.0-5.0 mEq/L, which is slightly higher than the normal range.
  • Replacement is usually done with oral potassium chloride supplements for mild deficiency, while severe hypokalemia may require IV potassium at rates not exceeding 10-20 mEq/hour to prevent complications.
  • Magnesium levels should also be checked and corrected if low, as magnesium deficiency can make potassium replacement less effective.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.