What is the role of Chimeric Antigen Receptor (CAR) T cells in treating Graft-Versus-Host Disease (GVHD)?

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Last updated: February 25, 2025View editorial policy

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From the Guidelines

CAR-T cell therapy is not currently approved for treating Graft-versus-Host Disease (GVHD), though research in this area is ongoing. The primary concern with using CAR-T cells in GVHD is that they might exacerbate the condition by increasing inflammatory responses 1. GVHD is typically treated with corticosteroids (like prednisone at 1-2 mg/kg/day), calcineurin inhibitors (tacrolimus or cyclosporine), antithymocyte globulin, or JAK inhibitors (ruxolitinib) 1.

Current Treatment Approaches

  • Corticosteroids are the mainstay of initial treatment for GVHD, often used in conjunction with a T-cell inhibitor (cyclosporine or tacrolimus) 1.
  • In cases of corticosteroid-refractory GVHD, numerous therapies have been studied, including cyclophosphamide, biologics, and photopheresis, with varied success depending on the tissues involved and the severity of the disease 1.
  • Management of GVHD can be optimized by providing coordinated care from a multidisciplinary team, preferably in medical centers with access to specialized transplant services 1.

Experimental Approaches

  • Some research explores using regulatory T cells (Tregs) modified with CARs to suppress GVHD, potentially targeting specific tissues affected by GVHD while preserving beneficial graft-versus-tumor effects 1.
  • The use of allogeneic CAR-T cells, which could offer an 'off-the-shelf' approach, is also being investigated, but graft-versus-host disease and host rejection remain major barriers 1.
  • Genome editing strategies, such as CRISPR-Cas, could potentially be used to prevent or delay the rejection of CAR-T cells by the recipient's immune system or to boost CAR-T-cell efficacy by knocking out T cell inhibitory receptors or signaling molecules 1.

Clinical Practice

  • Patients with GVHD should continue to receive standard therapies while these novel approaches are being investigated.
  • Clinical trials are needed to establish the safety and efficacy of CAR-T cell therapy in GVHD before it can be recommended for clinical practice.

From the Research

Role of CAR T cells in Treating GVHD

  • CAR T cells can be used to prevent and treat Graft-Versus-Host Disease (GVHD) by targeting specific cells involved in the disease pathogenesis, such as CD83+ cells 2.
  • Human CD83-targeted CAR T cells have been shown to eradicate pathogenic CD83+ target cells, increase the ratio of regulatory T cells to allo-activated Tconvs, and provide durable prevention of xenogeneic GVHD 2.
  • CAR T-cell therapy can also trigger GVHD, especially in patients who have undergone allogeneic hematopoietic cell transplantation (allo-HCT), highlighting the need for careful management and monitoring 3, 4.

Alternative Approaches to Prevent GVHD

  • Developing methods to disrupt endogenous TCR of allogeneic CAR-T cells using gene editing tools to prevent GVHD is an active area of research, but it also raises concerns about off-target activity and genotoxicities 5.
  • Alternative cell types, such as γδ-T, iNKT, virus-specific T, double negative T cells, and MAIT cells, killer cells (NK and CIK cells), non-lymphocytic cells (neutrophils and macrophages), stem/progenitor cells, and cell-free extracellular vesicles, are being explored as potential substitutes for αβ-T cells to reduce the risk of GVHD 5.

Treatment of GVHD after CAR T-cell Therapy

  • Extracorporeal photopheresis (ECP) has been shown to be a promising strategy for the treatment of GVHD after CAR T-cell therapy, with the ability to reduce alloreactivity without hampering CAR T-cell functionality 3, 6.
  • ECP can selectively induce apoptosis, enrich naive and central memory CAR-T cells, and modulate the cytokine milieu to promote immune tolerance, making it a potential therapeutic approach for GVHD after CAR T-cell therapy 6.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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