What are the different types of Bartter syndrome?

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From the Guidelines

Bartter syndrome is classified into five main types based on genetic mutations and clinical features, with each type presenting with varying severity of hypokalemia, metabolic alkalosis, and increased urinary excretion of sodium, potassium, and chloride, as reported in the most recent study by 1.

Types of Bartter Syndrome

The five types of Bartter syndrome are:

  • Type I: caused by mutations in the SLC12A1 gene affecting the sodium-potassium-chloride cotransporter
  • Type II: involves mutations in the KCNJ1 gene affecting potassium channels
  • Type III: results from mutations in the CLCNKB gene affecting chloride channels
  • Type IV: characterized by mutations in either the BSND gene or both CLCNKA and CLCNKB genes, often causing sensorineural deafness along with renal symptoms
  • Type V: the rarest form, involves mutations in the CASR gene that regulates calcium sensing

Clinical Features and Treatment

Each type presents with varying severity of the classic symptoms, including hypokalemia, metabolic alkalosis, normal blood pressure despite elevated renin and aldosterone levels, and increased urinary excretion of sodium, potassium, and chloride, as described in 1. The age of onset differs among types, with Types I and II typically manifesting prenatally or in early infancy, Type III appearing in childhood, Type IV presenting at birth with additional hearing impairment, and Type V showing hypocalcemia alongside the typical features. Treatment generally includes potassium supplementation, prostaglandin inhibitors like indomethacin, and potassium-sparing diuretics, with specific approaches tailored to each type's unique characteristics.

Key Considerations

It is essential to distinguish Bartter syndrome from other salt-losing tubulopathies and to consider the potential risks and benefits of prenatal intervention, as discussed in 1. Genetic testing is crucial for diagnosis, and a minimal diagnostic panel should include genes underlying Bartter syndrome, as well as Gitelman syndrome, which can be difficult to distinguish clinically from BS3, as recommended in 1.

From the Research

Types of Bartter Syndrome

Bartter syndrome can be classified into different types based on the underlying mutant gene and clinical presentation. The main types of Bartter syndrome are:

  • Antenatal or neonatal Bartter syndrome (aBS)
  • Classic Bartter syndrome (cBS)
  • Five subtypes based on the underlying mutant gene:
    • Type I: SLC12A1 gene mutation 2, 3, 4
    • Type II: KCNJ1 gene mutation 2, 5, 4
    • Type III: CLCNKB gene mutation 2, 3, 4
    • Type IV: BSND gene mutation 2, 4, 6
    • Type V: CASR gene mutation 2, 4

Clinical Presentation

The clinical presentation of Bartter syndrome can vary depending on the type and underlying gene mutation. Some common features include:

  • Renal salt wasting
  • Hypokalemic metabolic alkalosis
  • Hypercalciuria
  • Hyperreninaemic hyperaldosteronism
  • Nephrocalcinosis
  • Sensorineural deafness (in some types, such as Type IV) 5, 6
  • Growth failure 3
  • Prematurity (in antenatal Bartter syndrome) 3

Genotype-Phenotype Correlation

The genotype-phenotype correlation in Bartter syndrome is not always clear-cut, and patients with the same gene mutation can present with different phenotypes 2, 3. However, some studies have identified specific mutations and their corresponding clinical features, which can aid in diagnosis and genetic counseling 3, 6.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Genetic basis of Bartter syndrome in Korea.

Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association, 2012

Research

Bartter syndrome.

Current opinion in nephrology and hypertension, 2003

Research

Nephrocalcinosis as adult presentation of Bartter syndrome type II.

The Netherlands journal of medicine, 2014

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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