What is the relationship between Delta-9-tetrahydrocannabinol (Delta-9-THC) and seizure activity?

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Last updated: February 26, 2025View editorial policy

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From the Guidelines

Delta-9-tetrahydrocannabinol (Delta-9 THC) should be avoided in individuals with seizure disorders as it may potentially trigger seizures in susceptible individuals. The relationship between Delta-9 THC and seizure activity is complex, and the current evidence suggests that THC can alter neuronal excitability in the brain, potentially worsening seizure control 1.

Key Considerations

  • Delta-9 THC may trigger seizures in susceptible individuals, making it essential to avoid recreational cannabis products containing this compound if you have a seizure disorder.
  • CBD (cannabidiol), another cannabis compound, has shown promise for seizure control and is available as Epidiolex, an FDA-approved medication for certain severe forms of epilepsy at doses of 5-20 mg/kg/day.
  • The distinction between Delta-9 THC and CBD is crucial, as CBD appears to have anticonvulsant properties by modulating calcium channels and reducing neuronal hyperexcitability.

Recommendations

  • If you have a seizure disorder, consult with a neurologist who specializes in epilepsy to discuss appropriate options, as individual responses can vary significantly based on seizure type, existing medications, and other health factors.
  • Avoid using marijuana, including Delta-9 THC, as it may increase anesthetic requirements and inhibit platelet aggregation in vitro, with tetrahydrocannabinol exhibiting a more potent effect 1.
  • Consider FDA-approved formulations of CBD, such as Epidiolex, for seizure control under the guidance of a qualified healthcare professional.

From the Research

Relationship Between Delta-9-tetrahydrocannabinol (Delta-9-THC) and Seizure Activity

  • The relationship between Delta-9-tetrahydrocannabinol (Delta-9-THC) and seizure activity is complex and has been studied in various research papers 2, 3, 4, 5, 6.
  • Delta-9-THC is a psychoactive constituent of Cannabis sativa, and its effects on seizure activity have been investigated in preclinical models of epilepsy 2.
  • Some studies suggest that Delta-9-THC may have anticonvulsant effects, although its mechanisms of action are not fully understood 5.
  • In contrast to cannabidiol (CBD), which has been shown to have significant anticonvulsant effects, Delta-9-THC has been found to have partial agonist activity at the endocannabinoid receptors CB1 and CB2 5.
  • A recent study found that combining low-dose CBD with Delta-9-THC resulted in significant anticonvulsant effects, suggesting a potential synergistic effect between the two compounds 6.

Mechanisms of Action

  • The mechanisms of action of Delta-9-THC in relation to seizure activity are not fully understood, but it is thought to involve modulation of neurotransmitter release and inflammation 2.
  • Delta-9-THC has been found to bind to other targets, including TRPV1, voltage-gated potassium and sodium channels, and GPR55, which may contribute to its anticonvulsant effects 5.
  • A recent study found that CBD exerts its anticonvulsant effects, at least in part, through the 5-HT1A receptor in the neocortex of mice 6.

Clinical Implications

  • The use of Delta-9-THC and other cannabinoids in the treatment of epilepsy is still in its early stages, and more research is needed to fully understand their potential benefits and risks 3, 4.
  • Clinicians have expressed concerns about the potential risks of using Delta-9-THC in patients with epilepsy, particularly in relation to its psychoactive effects and potential interactions with other medications 3.
  • Further studies are needed to determine the optimal dosing and formulation of Delta-9-THC and other cannabinoids for the treatment of epilepsy, as well as their potential interactions with other medications 5, 6.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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