What are the causes of metabolic alkalosis?

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From the Guidelines

Metabolic alkalosis is primarily caused by excessive loss of hydrogen ions or gain of bicarbonate in the body, with the main causes including diuretic use, vomiting or nasogastric suction, excessive alkali administration, severe potassium depletion, and certain endocrine disorders. The causes of metabolic alkalosis can be categorized into several key areas, including:

  • Diuretic use, particularly loop and thiazide diuretics like furosemide and hydrochlorothiazide, which increase renal bicarbonate reabsorption 1
  • Vomiting or nasogastric suction, which removes acidic gastric contents
  • Excessive alkali administration, such as sodium bicarbonate or antacids containing calcium carbonate
  • Severe potassium depletion, which promotes hydrogen ion movement into cells
  • Certain endocrine disorders like hyperaldosteronism or Cushing's syndrome
  • Contraction alkalosis, which can occur with volume depletion, as the kidneys retain sodium and bicarbonate
  • Milk-alkali syndrome from excessive calcium and absorbable alkali intake
  • Bartter and Gitelman syndromes, genetic disorders affecting renal tubular function, can lead to chronic metabolic alkalosis 1 It's worth noting that the body typically compensates for metabolic alkalosis through decreased respiratory rate to retain carbon dioxide, but severe cases may require addressing the underlying cause, administering chloride-containing solutions like normal saline, correcting potassium deficiencies, or using carbonic anhydrase inhibitors like acetazolamide in specific situations. Some rare tubulopathies exhibiting metabolic alkalosis are listed in Table 4 of the study by 1, and urinary chloride excretion assessed by either fractional chloride excretion or urinary sodium/chloride ratio is helpful to distinguish renal from extrarenal salt losses. In patients with suspected Bartter syndrome, genetic analysis has surpassed diuretic tests, and routine tubular function testing is not recommended 1.

From the FDA Drug Label

Supplemental potassium chloride and, if required, an aldosterone antagonist are helpful in preventing hypokalemia and metabolic alkalosis The causes of metabolic alkalosis include:

  • Hypokalemia
  • Aldosterone excess 2

From the Research

Causes of Metabolic Alkalosis

Metabolic alkalosis is a condition characterized by an increase in plasma bicarbonate levels and blood arterial pH. The causes of metabolic alkalosis can be divided into several categories:

  • Generation of alkalosis: This can occur due to excessive hydrogen ion loss through the gastrointestinal tract (e.g., vomiting) or the kidney (e.g., use of loop diuretics), or due to exogenous base gain 3, 4, 5.
  • Maintenance of alkalosis: This reflects the inability of the kidney to excrete excess bicarbonate due to factors such as hypovolemia, chloride depletion, hypokalemia, hyperaldosteronism, renal failure, or a combination of these factors 3, 4, 5.
  • Clinical states associated with metabolic alkalosis: These include vomiting, aldosterone or cortisol excess, licorice ingestion, chloruretic diuretics, excess calcium alkali ingestion, and genetic diseases such as Bartter syndrome, Gitelman syndrome, and cystic fibrosis 4, 6, 5.
  • Categories of metabolic alkalosis: These can be divided into chloride depletion alkalosis, mineralocorticoid excess syndromes, apparent mineralocorticoid excess syndromes, and excess alkali administration 6, 7.

Key Factors Contributing to Metabolic Alkalosis

Some key factors that contribute to the development and maintenance of metabolic alkalosis include:

  • Volume contraction 3, 4, 5
  • Low glomerular filtration rate 4, 5
  • Potassium deficiency 3, 4, 5
  • Hypochloremia 3, 4, 7
  • Aldosterone excess 3, 4, 5
  • Elevated arterial carbon dioxide 4

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

The patient with metabolic alkalosis.

Acta clinica Belgica, 2019

Research

Metabolic Alkalosis Pathogenesis, Diagnosis, and Treatment: Core Curriculum 2022.

American journal of kidney diseases : the official journal of the National Kidney Foundation, 2022

Research

Metabolic alkalosis.

Journal of nephrology, 2006

Research

Approach to metabolic alkalosis.

Emergency medicine clinics of North America, 2014

Research

[Water-electrolyte and acid-base disorders. VII. Metabolic alkalosis].

Boletin medico del Hospital Infantil de Mexico, 1990

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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