What are the primary causes of embolic stroke?

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Last updated: July 15, 2025View editorial policy

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Primary Causes of Embolic Stroke

Cardiogenic embolism (20% of ischemic strokes), large-artery atherosclerosis (20%), and small-vessel disease (25%) are the main causes of embolic stroke, with approximately 30% remaining cryptogenic despite thorough evaluation. 1

Cardiac Sources of Embolism

Atrial Fibrillation

  • Most common cardiac cause of embolic stroke
  • Risk increases with additional factors such as:
    • Prior stroke/TIA
    • Hypertension
    • Heart failure
    • Diabetes mellitus
    • Age ≥75 years

Valvular Heart Disease

  • Rheumatic mitral valve disease significantly increases embolic risk even without AF 1
  • Mitral stenosis with prior embolic event or left atrial thrombus requires anticoagulation 1
  • Mitral valve prolapse increases stroke risk (RR 2.2), especially with:
    • Mitral valve thickening
    • Development of AF
    • Older age 1
  • Mitral annular calcification increases stroke risk (RR 2.1-3.1) 1
  • Prosthetic heart valves, especially mechanical valves, pose significant embolic risk 1

Other Cardiac Conditions

  • Myocardial infarction with left ventricular thrombi
  • Cardiomyopathy with reduced ejection fraction
  • Endocarditis (infective and non-bacterial thrombotic)
    • 20-40% of endocarditis patients suffer embolic events 1
    • Risk factors include vegetation size, mitral valve involvement, and S. aureus infection
  • Cardiac tumors
    • Myxomas (most common cardiac tumor, 30-40% embolize)
    • Papillary fibroelastomas (stroke/TIA is presenting symptom in 50%) 1

Patent Foramen Ovale (PFO) and Atrial Septal Aneurysm (ASA)

  • Can allow paradoxical embolism from venous sources
  • Particularly significant when both PFO and ASA are present 1

Non-Cardiac Sources

Large-Artery Atherosclerosis (20% of ischemic strokes)

  • Extracranial or intracranial segments of carotid or vertebrobasilar arteries 1
  • Mechanism: artery-to-artery embolism or hemodynamic insufficiency 1
  • Even "non-stenotic" carotid plaques may constitute important sources of cerebral embolism 1
  • Aortic arch atherosclerosis

Small-Vessel Disease (25% of ischemic strokes)

  • Causes lacunar or subcortical strokes
  • Associated with diabetes or hypertension
  • Not usually caused by atherosclerosis 1

Other Uncommon Causes

  • Fat embolism after trauma
  • Tumor embolism
  • Gas embolism
  • Nonatherosclerotic vasculopathies
  • Hypercoagulable states
  • Hematologic disorders 1, 2

Cryptogenic Stroke (30% of ischemic strokes)

  • Embolic strokes of undetermined source (ESUS)
  • Likely thromboembolic in nature
  • Potential sources include:
    • Minor-risk or covert cardiac sources
    • Paradoxical embolism via PFO
    • Non-occlusive atherosclerotic plaques 3

Clinical Implications and Pitfalls

Important Diagnostic Considerations

  • Timing of symptom onset is crucial for treatment decisions 1
  • Multiple small cortical infarcts in middle cerebral artery territory suggest embolic source 1
  • Silent cerebral infarctions on imaging may indicate increased stroke risk 1

Treatment Considerations

  • Anticoagulation is indicated for:
    • Atrial fibrillation (XARELTO or warfarin) 4, 5
    • Mechanical heart valves (warfarin with target INR 2.5-3.5) 1
    • Mitral stenosis with prior embolic event or left atrial thrombus 1
  • Antiplatelet therapy for:
    • Non-cardioembolic stroke sources
    • Mitral valve prolapse with TIA 1

Common Pitfalls

  • Failing to identify the embolic source may lead to inappropriate secondary prevention
  • Anticoagulants are contraindicated in infective endocarditis unless indicated for other cardiovascular conditions 1
  • Novel oral anticoagulants (factor Xa inhibitors and direct thrombin inhibitors) are not indicated for mechanical heart valves 1
  • Assuming emboli from carotid sources only travel ipsilaterally - they can reach contralateral hemispheres through collateral circulation 6

Understanding the specific cause of embolic stroke is essential for implementing appropriate secondary prevention strategies to reduce morbidity and mortality.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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