Guidelines for Managing Tachycardia
The management of tachycardia should follow a structured approach based on hemodynamic stability, with immediate synchronized cardioversion for unstable patients and a stepwise pharmacological approach for stable patients. 1, 2
Initial Assessment and Classification
Hemodynamic Stability Assessment
- Hemodynamically unstable: Hypotension, altered mental status, signs of shock, chest pain, heart failure
QRS Complex Width
- Narrow QRS complex tachycardia (<120 ms): Likely supraventricular origin
- Wide QRS complex tachycardia (≥120 ms): May be ventricular tachycardia or SVT with aberrancy
Management Algorithm for Hemodynamically Stable Narrow QRS Tachycardia
First-Line Interventions
Vagal Maneuvers (Class I, Level B-R) 1, 2
- Valsalva maneuver: Patient bears down against closed glottis for 10-30 seconds in supine position
- Carotid sinus massage: Apply steady pressure over carotid sinus for 5-10 seconds (after confirming absence of bruit)
- Success rate of combined techniques: approximately 28% 1
Adenosine IV (Class I, Level B-R) 1, 2
- Initial dose: 6 mg rapid IV push followed by saline flush
- If ineffective: 12 mg IV push, may repeat once if needed
- Terminates approximately 95% of AVNRT cases
- Contraindicated in severe bronchial asthma
- Have resuscitation equipment available
Second-Line Interventions
IV Calcium Channel Blockers (Class IIa, Level B-R) 1, 2
- Diltiazem or verapamil for hemodynamically stable patients
- Avoid in patients with pre-excited AF, hypotension, or heart failure
IV Beta Blockers (Class IIa, Level B-R) 1, 2
- Metoprolol or esmolol for hemodynamically stable patients
- Avoid in patients with pre-excited AF, severe bronchospasm, or decompensated heart failure
Synchronized Cardioversion (Class I, Level B-NR) 1
- Recommended when pharmacological therapy fails or is contraindicated
- Sedate patient if hemodynamically stable
Management of Wide QRS Complex Tachycardia
Hemodynamically Stable Wide QRS Tachycardia
- If SVT with aberrancy is confirmed: Follow narrow QRS algorithm
- If ventricular tachycardia or uncertain mechanism: 1, 2
- IV procainamide or sotalol (Class I recommendation)
- IV amiodarone is acceptable, preferred in patients with impaired LV function
- Avoid AV nodal blocking agents (adenosine, calcium channel blockers, beta blockers) if pre-excited AF is suspected
Special Considerations
- Pre-excited AF: Avoid AV nodal blockers as they can increase conduction through accessory pathway and precipitate ventricular fibrillation 2
- Digitalis toxicity-induced VT: Specific antidotes may be required 1
Long-Term Management
Pharmacological Options
- Beta blockers: First-line for long-term management 2
- Calcium channel blockers: Alternative if beta blockers contraindicated 2
- Class IC agents (flecainide, propafenone): For patients without structural heart disease 2
- Amiodarone: For refractory cases, but has significant side effect profile 2
Definitive Treatment
- Catheter ablation: Recommended for recurrent, symptomatic SVT 2
- Success rates >95% for most SVT mechanisms
- Can be considered first-line therapy to avoid lifelong medication
Common Pitfalls to Avoid
- Misdiagnosis of wide QRS tachycardia as SVT when it's VT
- Simultaneous use of IV calcium channel blockers and beta blockers can cause profound hypotension
- Using AV nodal blockers in pre-excited AF can accelerate ventricular rate
- Delaying cardioversion in patients with hemodynamic compromise
- Failure to recognize tachycardia-induced cardiomyopathy in persistent tachycardia
By following this structured approach to tachycardia management, clinicians can effectively diagnose and treat various tachyarrhythmias while minimizing complications and improving patient outcomes.