What are the types of congenital hypothyroidism?

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Types of Congenital Hypothyroidism

Congenital hypothyroidism (CH) can be classified into three main categories: thyroid dysgenesis, thyroid dyshormonogenesis, and central hypothyroidism, with thyroid dysgenesis accounting for approximately 85% of permanent cases.

Primary Congenital Hypothyroidism

1. Thyroid Dysgenesis (85% of cases)

Abnormal thyroid gland development during embryogenesis:

  • Thyroid ectopy: Most common form - thyroid tissue located in abnormal position (usually sublingual)
  • Thyroid aplasia/athyreosis: Complete absence of thyroid tissue
  • Thyroid hypoplasia: Reduced size but normal location of thyroid gland

Genetic causes of thyroid dysgenesis:

  • Mutations in transcription factors: PAX8, TTF-2 (FOXE1), NKX2-1 (TTF-1)
  • TSH receptor loss-of-function mutations (can cause familial athyreosis)
  • Most cases (85%) are sporadic with unknown etiology 1

2. Thyroid Dyshormonogenesis (10-15% of cases)

Defects in thyroid hormone synthesis despite normal gland structure:

  • Sodium/iodide symporter (NIS) defects
  • Thyroid peroxidase (TPO) deficiency
  • Thyroglobulin synthesis defects
  • Iodotyrosine deiodinase deficiency
  • Pendred syndrome (associated with sensorineural hearing loss)
  • Hydrogen peroxide generation defects

These conditions typically have autosomal recessive inheritance 2, 3

3. Transient Primary Congenital Hypothyroidism

Temporary forms that resolve over time:

  • Maternal thyrotropin receptor blocking antibodies (TRB-Ab)
  • Maternal antithyroid drug exposure
  • Iodine deficiency or excess
  • Prematurity-associated hypothyroxinemia

Secondary/Central Congenital Hypothyroidism

1. Isolated TSH Deficiency

  • Rare genetic mutations affecting pituitary TSH production

2. Combined Pituitary Hormone Deficiencies

  • Often part of congenital hypopituitarism
  • May present with multiple hormone deficiencies 4

Syndromic Forms of Congenital Hypothyroidism

1. DiGeorge Syndrome (22q11.2 Deletion Syndrome)

  • Most common genetic cause of thymic aplasia/hypoplasia
  • Features: congenital heart defects, hypoparathyroidism, facial dysmorphism
  • Thymic aplasia occurs in <1% of individuals with 22q11.2DS 5

2. CHARGE Syndrome

  • Second most common genetic cause of DiGeorge-like phenotype
  • Caused by CHD7 mutations
  • Features: coloboma, heart defects, choanal atresia, growth retardation, genital abnormalities, ear anomalies 5

3. Nude SCID (FOXN1 Deficiency)

  • Autosomal recessive condition
  • Features: congenital alopecia totalis, nail dystrophy, thymic aplasia 5

4. Other Genetic Syndromes

  • PAX1 deficiency (Otofaciocervical syndrome type 2)
  • TBX1/TBX2 deficiency
  • FOXI3 haploinsufficiency 5

Environmental Causes of Congenital Thymic Aplasia/Hypoplasia

  • Diabetic embryopathy
  • In utero alcohol exposure
  • In utero retinoic acid exposure (excess or deficiency) 5

Clinical Presentation and Diagnosis

Most infants (>95%) with congenital hypothyroidism have few or no clinical manifestations at birth due to maternal thyroid hormone transfer 6, 4. When present, symptoms may include:

  • Decreased activity and increased sleep
  • Feeding difficulties
  • Constipation
  • Prolonged jaundice
  • Myxedematous facies
  • Large fontanels
  • Macroglossia
  • Distended abdomen with umbilical hernia
  • Hypotonia

Diagnosis is primarily made through newborn screening programs using:

  • Primary TSH with backup T4 or primary T4 with backup TSH measurements
  • Confirmatory serum thyroid function tests (elevated TSH, low T4/free T4)

Additional diagnostic tests to determine etiology:

  • Thyroid scintigraphy (99mTc or 123I)
  • Thyroid ultrasound
  • Serum thyroglobulin determination 6, 1

Management

Early treatment is crucial to prevent neurodevelopmental impairment:

  • Levothyroxine is the treatment of choice
  • Recommended starting dose: 10-15 mcg/kg/day
  • Goal: Rapidly raise serum T4 above 130 nmol/L (10 μg/dL) and normalize TSH
  • Frequent monitoring: Every 1-2 months in first 6 months, every 3-4 months thereafter 6

Prognosis is excellent with early detection and treatment, with IQs similar to sibling or classmate controls. Poorer outcomes may occur with:

  • Late treatment initiation (>30 days of age)
  • Lower initial levothyroxine doses
  • More severe hypothyroidism at diagnosis 6

References

Research

Congenital hypothyroidism: etiologies, diagnosis, and management.

Thyroid : official journal of the American Thyroid Association, 1999

Research

Genetics of Primary Congenital Hypothyroidism.

Pediatric endocrinology reviews : PER, 2018

Research

Athyreosis, dysgenesis, and dyshormonogenesis in congenital hypothyroidism.

Pediatric endocrinology reviews : PER, 2006

Research

Evaluation and management of the child with hypothyroidism.

World journal of pediatrics : WJP, 2019

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Congenital hypothyroidism.

Orphanet journal of rare diseases, 2010

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This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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