What causes eczema?

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Causes of Eczema

Eczema occurs primarily due to an interplay between genetic, immunological, and environmental factors that lead to skin barrier dysfunction and immune system dysregulation. 1

Pathophysiological Mechanisms

Skin Barrier Dysfunction

  • Genetic factors: Abnormalities in filaggrin and other stratum corneum constituents compromise the skin barrier 2
  • Tight junction defects: Disruption in epidermal tight junctions further impairs barrier function 2
  • Vicious cycle: Skin inflammation deteriorates barrier function by downregulating essential barrier-maintaining molecules 2

Immunological Factors

  • Dendritic cell involvement: Multiple dendritic cell subtypes play key roles in eczema development:
    • Langerhans cells
    • Inflammatory dendritic epidermal cells
    • Plasmacytoid dendritic cells 3
  • T-cell responses: Various T-cell subsets contribute to inflammation:
    • Th1/Tc1 or Th2/Tc2 cells
    • Th17, Th22, and/or Tc22 cells infiltrate the upper dermis 4
  • Innate lymphoid cells: These cells are involved in eczema formation 4

Inflammatory Process

  • Acute phase: Characterized by:
    • Spongiosis associated with hyaluronic acid overproduction
    • Secretion of self-protective galectin-7
    • Decreased expression of E-cadherin 4
  • Chronic phase: Features include:
    • Periostin contribution to inflammatory skin remodeling
    • Dermal fibrosis
    • Epidermal melanogenesis and dermal pigment deposition leading to hyperpigmentation 4

Environmental Triggers

External Factors

  • Allergens: Common allergens can trigger or worsen eczema 3
  • Microbial factors: Bacterial and viral stimuli influence the course and severity of eczema 3
  • Irritants: Various substances can act as irritants:
    • "Wet" irritants: detergents, organic solvents, soaps, weak acids and alkalis
    • "Dry" irritants: low humidity air, heat, powders and dusts 1

Contact Mechanisms

Eczema can develop through different contact mechanisms:

  • Irritant contact: Direct damage to the skin barrier without immune involvement
  • Allergic contact: T cell-mediated immune response to specific allergens
  • Phototoxic/photoallergic: Reactions triggered or worsened by light exposure 1

Pruritus (Itching) Mechanisms

  • Neurological factors: Hyperinnervation of the epidermis contributes to intense itching
  • Chemical mediators: Increased pruritogens in the skin
  • Central sensitization: Heightened sensitivity to itch signals in the central nervous system
  • Vicious cycle: Scratching damages skin, promoting further inflammation and itching 2

Clinical Manifestations

  • Acute eczema: Characterized by erythema, papules, seropapules, vesicles, pustules, scales, and crusts
  • Chronic eczema: Features lichenification, hyperpigmentation, and sometimes hypopigmentation
  • Common symptoms: Multiple pinpoint lesions, polymorphic appearance, and intense itching 4

Comorbidities

Eczematous diseases are associated with increased risk of:

  • Other allergic conditions (asthma, rhinoconjunctivitis, food allergies)
  • Coronary heart disease
  • Mental health issues such as depression 4, 5

Understanding these complex pathophysiological mechanisms has led to the development of targeted therapies for eczema, particularly for severe cases that don't respond to conventional treatments 5.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

The pathophysiology of atopic eczema.

Clinical and experimental dermatology, 2006

Research

What is "eczema"?

The Journal of dermatology, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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