What is the treatment for cytokine release syndrome (CRS)?

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Last updated: August 24, 2025View editorial policy

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Treatment of Cytokine Release Syndrome (CRS)

The primary treatment for cytokine release syndrome is tocilizumab (anti-IL-6 receptor antibody), with corticosteroids added for refractory cases, while providing appropriate supportive care based on CRS severity. 1

CRS Definition and Grading

CRS is a systemic inflammatory response characterized by fever, tachycardia, hypoxia, hypotension, and potential multi-organ dysfunction caused by the release of cytokines from activated immune cells. It commonly occurs following CAR T-cell therapy, bispecific antibodies, and other immunotherapies.

The American Society for Transplantation and Cellular Therapy (ASTCT) consensus grading system classifies CRS into four grades:

Grade Characteristics
Grade 1 Fever ≥38°C without hypotension or hypoxia
Grade 2 Fever with hypotension responsive to fluids/low-dose vasopressors OR hypoxia requiring low-flow oxygen
Grade 3 Fever with hypotension requiring vasopressors OR hypoxia requiring high-flow oxygen
Grade 4 Fever with hypotension requiring multiple vasopressors OR hypoxia requiring mechanical ventilation

Management Algorithm

Initial Evaluation and Monitoring

  • Complete blood count, comprehensive metabolic panel, magnesium, phosphorus, CRP, LDH, uric acid, fibrinogen, PT/PTT, and ferritin 2
  • Blood and urine cultures, chest radiograph if fever present
  • Continuous cardiac telemetry and pulse oximetry for grade ≥2 CRS
  • Consider echocardiogram for severe CRS to assess cardiac function 2

Grade 1 CRS Management

  • Antipyretics, IV hydration, and symptomatic management
  • Consider tocilizumab 8 mg/kg IV (not exceeding 800 mg) for prolonged symptoms >24 hours or in high-risk patients 2, 1
  • Consider empiric broad-spectrum antibiotics if neutropenic 2

Grade 2 CRS Management

  • Continue supportive care plus IV fluid boluses and supplemental oxygen
  • Administer tocilizumab 8 mg/kg IV (not exceeding 800 mg) 2
  • Repeat tocilizumab every 8 hours if no improvement, maximum 3 doses in 24 hours or 4 doses total
  • For persistent hypotension after 1-2 doses of tocilizumab, add dexamethasone 10 mg IV every 12 hours 2
  • Escalate to Grade 3 management if no improvement within 24 hours 2

Grade 3 CRS Management

  • Transfer to ICU
  • Continue tocilizumab as in Grade 2
  • Add dexamethasone 10 mg IV every 6 hours 2
  • Vasopressors as needed
  • Obtain echocardiogram and perform hemodynamic monitoring
  • Escalate to Grade 4 management if refractory 2

Grade 4 CRS Management

  • Continue ICU care with mechanical ventilation as needed
  • Continue tocilizumab as in Grade 2
  • Initiate high-dose methylprednisolone 500-1000 mg IV every 12 hours for 3 days, followed by a taper 2
  • For refractory cases, consider alternative therapies such as anakinra (IL-1 receptor antagonist), siltuximab, ruxolitinib, or other immunosuppressants 2, 1

Special Considerations

Pediatric Dosing

  • For children <30 kg, administer tocilizumab at 12 mg/kg IV 1

Steroid Use Caution

  • While necessary for severe CRS, corticosteroids may potentially impact the efficacy of immunotherapy 1
  • Strongly consider antifungal prophylaxis in patients receiving steroids for CRS 2

Concurrent Conditions

  • Monitor for concurrent immune effector cell-associated neurotoxicity syndrome (ICANS), which may require additional management 2, 1
  • Avoid medications that cause CNS depression in patients with neurological symptoms 1

Refractory CRS

  • For cases not responding to tocilizumab and steroids, consider:
    • Anakinra (IL-1 receptor antagonist)
    • Siltuximab (alternative IL-6 antagonist)
    • Continuous renal replacement therapy (CRRT) for cytokine removal in severe cases 3

Common Pitfalls and Caveats

  1. Delayed recognition: Early identification of CRS is critical for timely intervention. Fever is typically the first sign.

  2. Underestimating progression: CRS can rapidly progress from mild to severe; close monitoring is essential even with mild symptoms.

  3. Infection vs. CRS: Symptoms of CRS can mimic infection. Always perform infection workup but don't delay CRS treatment while awaiting results.

  4. Tocilizumab timing: Delaying tocilizumab administration in moderate-severe CRS can lead to worse outcomes. Don't hesitate to administer for grade ≥2 CRS.

  5. Steroid concerns: While there are concerns about steroids potentially reducing CAR T-cell efficacy, the benefit of treating life-threatening CRS outweighs this risk in severe cases.

  6. Overlooking organ dysfunction: CRS can affect multiple organ systems; comprehensive monitoring of cardiac, renal, hepatic, and pulmonary function is essential.

  7. Forgetting that fever may be masked: In patients receiving antipyretics, CRS grading is driven by hypotension and/or hypoxia rather than fever 2.

References

Guideline

Cytokine Release Syndrome Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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