What is the treatment for metoprolol (beta blocker) toxicity?

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Last updated: August 27, 2025View editorial policy

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Treatment of Metoprolol Toxicity

The treatment of metoprolol toxicity requires aggressive management with high-dose insulin and vasopressors as first-line therapies, followed by additional supportive measures based on clinical presentation.

Initial Assessment and Management

  • Assess for bradycardia, hypotension, altered mental status, and hypoglycemia
  • Secure airway, breathing, and circulation
  • Obtain ECG, continuous cardiac monitoring, and blood glucose levels
  • Consider activated charcoal for recent ingestions (within 1-2 hours) if airway is protected 1

First-Line Pharmacological Interventions

Vasopressors (Class 1, C-LD)

  • Start immediately for hypotension to maintain adequate perfusion
  • Options include epinephrine, norepinephrine, and dopamine
  • Titrate to effect based on blood pressure response 2, 1

High-Dose Insulin Euglycemic Therapy (HIET) (Class 1, B-NR)

  • Administer 1 U/kg IV bolus followed by infusion starting at 1 U/kg/hour
  • Titrate up to clinical effect (may require higher doses)
  • Monitor glucose every 15-30 minutes initially
  • Supplement with dextrose as needed to maintain euglycemia
  • Continue for 24-36 hours in severe cases 2, 1, 3

Second-Line Interventions

Glucagon (Class 2a, C-LD)

  • Administer 5-10 mg IV bolus over 3-5 minutes
  • Follow with continuous infusion of 1-5 mg/hour
  • Be prepared for potential vomiting as a side effect 2, 1

Atropine (Class 2b, C-LD)

  • Use for symptomatic bradycardia: 0.5-1 mg IV
  • May repeat to maximum of 3 mg
  • Note: Often has limited efficacy in beta-blocker overdose 2, 1

Calcium (Class 2b, C-LD)

  • Consider calcium chloride (10-20 mL IV) or calcium gluconate (30-60 mL IV)
  • May repeat every 10-20 minutes for 3-4 doses if beneficial 1

Advanced Interventions for Refractory Cases

Extracorporeal Life Support (Class 2a, C-LD)

  • Consider VA-ECMO for life-threatening poisoning with cardiogenic shock refractory to pharmacological interventions
  • Early consultation with ECMO team recommended 2, 1

Cardiac Pacing (Class 2b, C-LD)

  • Consider for persistent symptomatic bradycardia unresponsive to medical therapy
  • May have limited efficacy due to myocardial depression 2, 1

Hemodialysis (Class 2b, C-LD)

  • May be considered for severe metoprolol toxicity, particularly with renal impairment
  • Most effective for water-soluble beta-blockers with low protein binding 2

Intravenous Lipid Emulsion (ILE) (Class 3: No Benefit, C-LD)

  • Not recommended as routine therapy for beta-blocker poisoning
  • Limited evidence for efficacy despite some case reports 2, 3

Monitoring and Support

  • Continuous cardiac monitoring
  • Frequent blood pressure measurements
  • Serial ECGs
  • Blood glucose and potassium levels
  • Lactate levels to assess tissue perfusion 1

Special Considerations

  • Metoprolol toxicity can cause direct myocardial depression leading to bradycardia, hypotension, and cardiovascular collapse 3
  • Severe toxicity may require multiple treatment modalities simultaneously
  • Patients with co-ingestions (especially other cardiovascular medications) may have worse outcomes 4
  • Symptoms typically appear within 2-6 hours of ingestion but may be delayed with sustained-release formulations 4

References

Guideline

Management of Beta-Blocker Overdose

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Clinical toxicology of beta-blocker overdose in adults.

Basic & clinical pharmacology & toxicology, 2019

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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