What is the most common cause of copper deficiency?

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Most Common Causes of Copper Deficiency

The most common cause of copper deficiency is malabsorption following bariatric surgery, particularly gastric bypass procedures, due to altered gastrointestinal anatomy that impairs copper absorption in the stomach and proximal small intestine. 1

Pathophysiology and Absorption

Copper is primarily absorbed in the stomach and small intestine, with the duodenum being the main site of absorption 1. Normal copper absorption is a highly regulated process, with biliary excretion being the primary route of elimination 1. Daily copper requirements range between 1.1-2 mg/day for adults, with absorption rates varying between 20-50% 1.

Major Causes of Copper Deficiency

1. Post-Surgical Causes

  • Bariatric surgery - particularly Roux-en-Y gastric bypass and biliopancreatic diversion with duodenal switch 1, 2
  • Gastrectomy - partial or total 2
  • Small bowel resections - particularly affecting the duodenum and jejunum 2
  • History of subtotal colectomy with small bowel involvement 2

2. Nutritional Causes

  • Prolonged parenteral nutrition without adequate copper supplementation 1, 3
  • Premature infants on copper-deficient diets 3
  • Malnutrition states 4

3. Other Medical Conditions

  • Continuous renal replacement therapy 1
  • Malabsorption syndromes 5
  • Protein-losing enteropathy 4
  • Zinc excess - high-dose zinc supplementation can interfere with copper absorption 1, 5

Clinical Manifestations of Copper Deficiency

Hematologic Manifestations

  • Microcytic anemia (most common) 1
  • Neutropenia 1, 2
  • Pancytopenia 6
  • Myelodysplastic-like bone marrow changes 5, 6

Neurologic Manifestations

  • Myelopathy resembling subacute combined degeneration 5
  • Peripheral neuropathy 2
  • Ataxia 2
  • Paresthesias 2

Other Manifestations

  • Delayed wound healing 1
  • Hair depigmentation 1
  • Osteoporosis 1
  • Taste changes and glossitis 1

Diagnosis and Monitoring

Copper deficiency is diagnosed through blood tests showing:

  • Serum copper levels <12 μmol/L with elevated CRP (>20 mg/L) suggests deficiency 1
  • Serum copper levels <8 μmol/L (with or without elevated CRP) confirms deficiency 1
  • Low ceruloplasmin levels may be present 4

Treatment Approach

  • Mild to moderate deficiency: Oral supplementation with 4-8 mg/day of elemental copper 4
  • Severe deficiency: Intravenous copper supplementation may be required 4
  • When supplementing both zinc and copper, maintain a ratio of 8-15 mg zinc to 1 mg copper 4

Prevention in High-Risk Patients

  • Regular monitoring of copper levels in patients with history of bariatric surgery or other risk factors 1, 6
  • Routine copper supplementation in patients on long-term parenteral nutrition 1, 3
  • Lifelong dietary supplements for bariatric patients 6

Important Clinical Considerations

  • Neurological symptoms may be irreversible if diagnosis and treatment are delayed 2, 5
  • Hematological abnormalities typically respond well to copper replacement 5
  • Copper deficiency can mimic myelodysplastic syndrome, leading to misdiagnosis 5, 6

Vigilance for copper deficiency is particularly important in patients with a history of gastrointestinal surgery, as early detection and treatment can prevent permanent neurological damage 2, 5.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Clinical Manifestations of Copper Deficiency: A Case Report and Review of the Literature.

Nutrition in clinical practice : official publication of the American Society for Parenteral and Enteral Nutrition, 2021

Research

Copper deficiency in humans.

Seminars in hematology, 1983

Guideline

Management of Wilson's Disease

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Copper deficiency myelopathy.

Journal of neurology, 2010

Research

Copper deficiency (hypocupremia) and pancytopenia late after gastric bypass surgery.

Proceedings (Baylor University. Medical Center), 2013

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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