What is the mechanism of action (MOA) of acetazolamide?

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Mechanism of Action of Acetazolamide

Acetazolamide primarily works as a potent carbonic anhydrase inhibitor that prevents the conversion of carbon dioxide and water to carbonic acid and its subsequent dissociation to bicarbonate and hydrogen ions. 1

Primary Mechanism

  • Carbonic Anhydrase Inhibition: Acetazolamide is a non-bacteriostatic sulfonamide that specifically inhibits the enzyme carbonic anhydrase, which catalyzes the reversible reaction involving the hydration of carbon dioxide and dehydration of carbonic acid 1

  • Enzyme Specificity: It acts as a noncompetitive inhibitor of multiple carbonic anhydrase subtypes in humans 2

Physiological Effects Based on Target Tissues

Kidney Effects

  • Inhibits carbonic anhydrase in the kidney, disrupting the reversible reaction of CO2 hydration/carbonic acid dehydration 1
  • Results in renal loss of bicarbonate ions (HCO3-), which carries out sodium, water, and potassium 1
  • Causes alkalinization of urine and promotes diuresis 1
  • Increases reabsorption of ammonia by renal tubules due to urinary alkalinization 1
  • Acts in the proximal convoluted tubule to reduce sodium reabsorption 3

Eye Effects

  • Decreases secretion of aqueous humor in the eye 1
  • Results in reduced intraocular pressure, beneficial in glaucoma 1

Central Nervous System Effects

  • Retards abnormal, paroxysmal, excessive discharge from central nervous system neurons 1
  • Acidifies intracellular and extracellular environments, activating acid-sensing ion channels 2
  • Increases seizure threshold in critical structures such as the hippocampus 2
  • Decreases cerebrospinal fluid (CSF) production, useful in treating raised intracranial pressure 4, 5
  • Inhibits brain/choroid plexus CA II, IV, VA, and XII isoforms, leading to decreased CSF secretion 5

Respiratory Effects

  • Creates metabolic acidosis that stimulates ventilation, particularly useful at high altitude 6
  • Offsets hyperventilation-induced respiratory alkalosis at altitude 6
  • Allows chemoreceptors to respond more fully to hypoxic stimuli 6

Clinical Applications Based on Mechanism

  • Glaucoma: Reduces intraocular pressure by decreasing aqueous humor production 1
  • Epilepsy: Controls certain convulsive disorders by inhibiting abnormal neuronal discharge 1, 2
  • Edema: Promotes diuresis in cases of abnormal fluid retention (e.g., cardiac edema) 1
  • Idiopathic Intracranial Hypertension: Reduces CSF production and controls intracranial pressure 5
  • Altitude Sickness: Multiple mechanisms including metabolic acidosis, improved ventilation, better sleep quality, and diuresis 6, 7
  • Heart Failure: Enhances decongestion when used with loop diuretics by inhibiting sodium reabsorption in the proximal tubule 3

Potential Side Effects Related to Mechanism

  • Paresthesias and altered taste due to effects on neural tissue 7, 4
  • Metabolic acidosis from renal bicarbonate loss 7, 8
  • Electrolyte imbalances from diuretic effect 7
  • Kidney stones from urinary alkalinization 7, 8
  • Periorbital myokymia (rare) through unknown mechanisms 4

Acetazolamide's diverse therapeutic applications stem from its fundamental action as a carbonic anhydrase inhibitor, with different clinical effects manifesting based on the specific tissue distribution of carbonic anhydrase isoforms throughout the body.

References

Research

Acetazolamide: Old drug, new evidence?

Epilepsia open, 2022

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Mechanisms of action of acetazolamide in the prophylaxis and treatment of acute mountain sickness.

Journal of applied physiology (Bethesda, Md. : 1985), 2007

Guideline

High Altitude Sickness Prevention and Treatment

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Acetazolamide in the treatment of seizures.

The Annals of pharmacotherapy, 1996

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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