What are the symptoms of hypoaldosteronism?

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Symptoms of Hypoaldosteronism

Hypoaldosteronism primarily presents with electrolyte abnormalities including hyperkalemia, hyponatremia, and metabolic acidosis, along with symptoms of volume depletion such as fatigue, muscle weakness, postural dizziness, and hypotension. 1

Clinical Manifestations

Primary Symptoms

  • Electrolyte Abnormalities:
    • Hyperkalemia (present in 94.6% of cases) 1
    • Hyponatremia (present in 54.5% of cases) 1
    • Metabolic acidosis (present in 60.3% of cases) 1

Common Clinical Presentations

  • Fatigue and exercise intolerance
  • Muscle weakness and cramps
  • Postural dizziness
  • Low blood pressure or orthostatic hypotension
  • Weight loss
  • Increased heart rate
  • Abdominal pain
  • Low urine volume
  • Lethargy and confusion 2

Laboratory Findings

  • Elevated serum potassium (typically >5.0 mmol/L)
  • Decreased serum sodium (typically <135 mmol/L)
  • Decreased serum bicarbonate (metabolic acidosis)
  • Elevated serum creatinine (prerenal renal failure)
  • Increased plasma renin activity (in most forms of hypoaldosteronism)
  • Low or inappropriately normal aldosterone levels 2, 1

Types of Hypoaldosteronism

Isolated Hypoaldosteronism

  • Most common form in adults (95% of cases) 1
  • Often associated with hyporeninemic hypoaldosteronism
  • Frequently seen in older patients (>50 years)
  • Common in patients with diabetes mellitus and/or nephropathy 3

Primary Adrenal Insufficiency

  • Hypoaldosteronism as part of broader adrenal insufficiency
  • Additional symptoms related to cortisol deficiency
  • Less common (only 5% of hypoaldosteronism cases) 1

Pseudohypoaldosteronism

  • Presents with similar electrolyte abnormalities but due to resistance to aldosterone action
  • Type I: characterized by insensitivity of the distal nephron to aldosterone
  • Type II: associated with hyperabsorption of chloride in the distal nephron and hypertension 4

Risk Factors and Associated Conditions

  • Advanced age (median age 77 years in one study) 1
  • Diabetes mellitus
  • Chronic kidney disease
  • Interstitial nephropathy
  • Hydronephrosis
  • Use of certain medications:
    • Prostaglandin inhibitors (NSAIDs)
    • Beta-blockers
    • ACE inhibitors
    • Angiotensin receptor blockers
    • Potassium-sparing diuretics 3, 2

Diagnostic Considerations

  • Hyperkalemia may be the only presenting abnormality in some patients
  • Normal potassium levels do not rule out primary aldosteronism (the opposite condition) 5
  • In infants, high aldosterone levels may paradoxically be seen in early aldosterone deficiency, mimicking pseudohypoaldosteronism 6
  • The ratio of stimulated plasma aldosterone to serum potassium can help differentiate hypoaldosteronism (ratio <3) from normal aldosterone function 7

Complications

If left untreated, hypoaldosteronism can lead to:

  • Severe hyperkalemia causing cardiac arrhythmias
  • Worsening renal function
  • Metabolic acidosis
  • Hypovolemic shock in severe cases
  • Impaired cognitive function 2

Early recognition of these symptoms is crucial for prompt diagnosis and treatment to prevent life-threatening complications, particularly in older patients and those with underlying renal disease or diabetes.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

[Hyporeninemic hypoaldosteronism and the differential diagnosis of hyperkalemia].

Schweizerische medizinische Wochenschrift, 1982

Guideline

Primary Aldosteronism Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Aldosterone deficiency with a hormone profile mimicking pseudohypoaldosteronism.

Journal of pediatric endocrinology & metabolism : JPEM, 2020

Research

Diagnostic value of plasma aldosterone/potassium ratio in hypoaldosteronism.

Journal of the Formosan Medical Association = Taiwan yi zhi, 1995

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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