What is the pathophysiology of myopia?

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Pathophysiology of Myopia

Myopia develops primarily through excessive axial elongation of the eyeball, resulting from a complex interplay between genetic predisposition and environmental factors, with near work activities and limited outdoor time being key environmental triggers for this abnormal eye growth. 1, 2

Anatomical Changes

  • Axial elongation: The fundamental structural change in myopia is an increase in the axial length of the eyeball 3
  • This elongation causes light rays to focus in front of the retina rather than directly on it, resulting in blurred distance vision
  • The severity of myopia correlates with the degree of axial elongation

Etiology: Gene-Environment Interaction

Genetic Factors

  • More than 450 common genetic loci have been associated with myopia development 1
  • Higher prevalence in individuals with myopic parents, strongly suggesting genetic predisposition 4
  • Genetic factors influence susceptibility to environmental triggers

Environmental Factors

  • Near work activities: Extended periods of close-up visual tasks (reading, screen time) 1
  • Limited outdoor time: Reduced exposure to natural light 1, 5
  • Educational intensity: Higher levels of education correlate with increased myopia prevalence 1

Interaction Mechanism

  • Environmental factors like near work create optical blur that triggers signaling cascades
  • These signals influence eye growth and remodeling in genetically susceptible individuals
  • Early onset of myopia is associated with faster progression and higher risk of severe myopia 5

Progression Factors

  • Age of onset: Earlier onset correlates with faster progression and higher final myopia 5
  • Hyperopia ≤0.5 diopters in children is a predictor of future myopia development 5
  • Educational intensity: Higher educational demands increase progression rates
  • Screen time: Increased digital device use contributes to progression

Pathological Consequences

High myopia (≥-6 diopters) can lead to sight-threatening complications:

  • Retinal detachment
  • Choroidal neovascularization
  • Early cataracts
  • Glaucoma
  • Macular atrophy 5

Epidemiological Significance

  • Reaching epidemic proportions globally, especially in East Asia (80-90% prevalence) 5, 2
  • Approximately 20% of myopic individuals develop high myopia with risk of complications 5
  • Ranked as the sixth leading cause of blindness globally 4
  • The rapid increase in prevalence cannot be explained by genetics alone, highlighting the critical role of environmental factors 4

Prevention and Management Implications

The understanding of myopia pathophysiology has led to effective interventions:

  • Increased outdoor activity to delay onset 5
  • Reduction in prolonged near work 5
  • Optical interventions (defocusing lenses, orthokeratology) 2, 3
  • Pharmacological approaches (low-dose atropine) 3

Understanding the pathophysiological mechanisms of myopia is crucial for developing effective strategies to prevent its onset, slow its progression, and reduce the risk of vision-threatening complications.

References

Research

Gene-environment interaction in myopia.

Ophthalmic & physiological optics : the journal of the British College of Ophthalmic Opticians (Optometrists), 2023

Research

Myopia.

Nature reviews. Disease primers, 2020

Research

[Myopia in children].

Medecine sciences : M/S, 2020

Research

Epidemiology of Myopia.

Asia-Pacific journal of ophthalmology (Philadelphia, Pa.), 2016

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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