What are the symptoms of hypercortisolism?

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Symptoms of Hypercortisolism

Hypercortisolism (Cushing's syndrome) presents with a constellation of symptoms including weight gain with central obesity, proximal muscle weakness, hypertension, psychiatric disturbances, hirsutism, purple striae, buffalo hump, supraclavicular fat pad enlargement, hyperglycemia, and hypokalemia. 1

Clinical Presentation

Physical Manifestations

  • Central obesity - characteristic fat distribution in trunk, face, and neck
  • Facial changes:
    • Moon face
    • Facial plethora (redness)
    • Supraclavicular fat pads
    • Dorsocervical fat pad ("buffalo hump")
  • Skin changes:
    • Wide (>1 cm), purple striae
    • Thin, fragile skin
    • Easy bruising and ecchymoses
    • Delayed wound healing
    • Acne
  • Musculoskeletal:
    • Proximal muscle weakness (especially in thighs)
    • Muscle atrophy
    • Osteoporosis and increased fracture risk

Metabolic and Cardiovascular

  • Hypertension (often severe and resistant to treatment)
  • Hyperglycemia or diabetes mellitus
  • Hypokalemia (especially with ectopic ACTH syndrome)
  • Dyslipidemia
  • Increased risk of cardiovascular events

Reproductive and Hormonal

  • In women:
    • Hirsutism
    • Menstrual irregularities
    • Decreased libido
    • Infertility
  • In men:
    • Decreased libido
    • Erectile dysfunction

Neuropsychiatric

  • Depression
  • Anxiety
  • Irritability
  • Sleep disturbances
  • Cognitive impairment
  • Psychosis (in severe cases)

Severity Indicators

Severe hypercortisolism is a medical emergency characterized by 2, 3:

  • Markedly elevated cortisol levels
  • Multiorgan failure
  • Severe hypokalemia
  • Uncontrolled hypertension
  • Opportunistic infections
  • Psychosis
  • Thromboembolism

Diagnostic Considerations

When hypercortisolism is suspected based on clinical features, screening should include 1:

  • 1 mg overnight dexamethasone suppression test (DST)
    • Serum cortisol >50 nmol/L excludes cortisol hypersecretion
    • 51-138 nmol/L suggests possible autonomous cortisol secretion
    • 138 nmol/L indicates cortisol hypersecretion

  • 24-hour urinary free cortisol
  • Late-night salivary cortisol
  • Plasma ACTH to determine if the hypercortisolism is ACTH-dependent or independent

Clinical Pitfalls

  • Hypercortisolism must be considered in the differential diagnosis of common problems like hirsutism, menstrual irregularity, hypertension, diabetes mellitus, and obesity 4
  • Older adults may present with more subtle symptoms, sometimes limited to cardiovascular manifestations ("apathetic hypercortisolism")
  • Diagnostic difficulties may arise from drug interference, alcohol ingestion, and depression 4
  • Subclinical hypercortisolism may still cause significant adverse effects, including increased mortality risk 5

Early recognition and treatment of hypercortisolism are crucial to prevent progressive complications including adiposity, myopathy, dermopathy, psychopathy, glucose intolerance, hypercholesterolemia, hypertension, atherosclerosis, immunosuppression, and ultimately death 6.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Medical therapy in severe hypercortisolism.

Best practice & research. Clinical endocrinology & metabolism, 2021

Research

Glucocorticoid action and the clinical features of Cushing's syndrome.

Endocrinology and metabolism clinics of North America, 1994

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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