What is the recommended dose of Intravenous Immunoglobulin (IV IG) for the treatment of Acute Motor-Sensory Axonal Neuropathy (AMSAN)?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: September 19, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Recommended IVIG Dosing for Acute Motor-Sensory Axonal Neuropathy (AMSAN)

The recommended dose of Intravenous Immunoglobulin (IVIG) for AMSAN is 2 g/kg of ideal body weight, typically divided over 5 days (0.4 g/kg/day) as a total treatment course. 1

Dosing Protocol and Administration

AMSAN is a rare axonal variant of Guillain-Barré syndrome (GBS) that requires prompt immunomodulatory treatment. The dosing protocol follows established guidelines for similar acute inflammatory neuropathies:

  • Standard dosing: 2 g/kg total dose 1
  • Administration schedule: Typically divided over 5 days (0.4 g/kg/day) 1
  • Alternative administration: For doses exceeding 80g, consider administering over 3-5 days at 0.4 g/kg/day 2
  • Duration: Single course initially, with consideration for repeat treatment if clinical deterioration occurs after initial improvement 1

Pre-Treatment Assessment

Before administering IVIG, the following assessments should be performed:

  • Serum IgA level: Must be checked before administration as IgA deficiency may lead to severe reactions including fever, infusion reactions, and anaphylaxis 2
  • If IgA deficient: Use IVIG preparation with reduced IgA levels 2
  • Cardiac function and fluid status: Assess before administration to minimize risks 1

Monitoring During Treatment

  • Neurological assessments: Perform frequent evaluations to monitor treatment response 1
  • Pulmonary function: Monitor closely as respiratory compromise can occur rapidly in AMSAN 1
  • Autonomic function: Watch for dysautonomia which can complicate the clinical course 1

Treatment Considerations

  • Hospitalization: Patients should be admitted to a facility with capability for rapid transfer to ICU-level monitoring 1
  • Alternative treatment: If IVIG is unavailable or contraindicated, plasma exchange (200-250 ml plasma/kg in five sessions) is an acceptable alternative 1
  • Supportive care: Address complications such as neuropathic pain (with pregabalin, gabapentin, or duloxetine) and constipation/ileus if present 1

Potential Adverse Effects

IVIG therapy carries several risks that require monitoring:

  • Common: Headache (most frequent during loading dose), infusion reactions 3
  • Serious: Thromboembolic events, renal dysfunction, aseptic meningitis, hemolysis 1
  • Long-term safety: Studies in other neurological conditions suggest decreasing adverse event rates with continued treatment 3

Clinical Pearls

  • AMSAN has a worse prognosis than demyelinating forms of GBS, making prompt treatment crucial
  • The diagnosis should be confirmed with EMG and nerve conduction studies before initiating treatment 4
  • Unlike AMAN (which is often associated with Campylobacter jejuni), AMSAN has less clear triggering factors, though associations with EBV and autoimmune conditions like SLE have been reported 4, 5, 6
  • Early treatment initiation is associated with better outcomes

IVIG remains the first-line therapy for AMSAN due to its efficacy and relatively favorable side effect profile compared to alternatives, with treatment protocols following established guidelines for GBS variants.

References

Guideline

Dermatomyositis Treatment Guidelines

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Acute motor-sensory axonal neuropathy associated with systemic lupus erythematosus.

Proceedings (Baylor University. Medical Center), 2019

Research

Acute motor-sensory axonal neuropathy associated with active systemic lupus erythematosus and anticardiolipin antibodies.

Journal of clinical rheumatology : practical reports on rheumatic & musculoskeletal diseases, 2001

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.