What investigations are recommended for a patient with suspected sepsis and acute kidney injury (AKI)?

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Investigations for Suspected Sepsis with Acute Kidney Injury

For patients with suspected sepsis and acute kidney injury (AKI), a comprehensive diagnostic workup should include blood tests, urinalysis, cultures, imaging, and biomarkers to determine etiology, severity, and guide management.

Initial Laboratory Investigations

Blood Tests

  • Complete blood count with differential (leukocytosis with left shift suggests infection) 1
  • Serum creatinine and blood urea nitrogen (BUN) to stage AKI severity using KDIGO criteria 2
  • Serum electrolytes (sodium, potassium, bicarbonate) - monitor at least every 48 hours 1
  • Liver function tests (bilirubin, transaminases, alkaline phosphatase)
  • Coagulation profile (PT, INR, aPTT) to assess for coagulopathy 1
  • Arterial blood gas analysis to evaluate acid-base status 1
  • Lactate levels (marker of tissue hypoperfusion in sepsis) 1
  • Blood glucose monitoring every 1-2 hours until stable 1

Cultures and Infection Workup

  • Blood cultures (at least two sets from different sites) before antibiotic administration 1
  • Urine culture 1
  • Cultures from suspected sources of infection (respiratory, wound, etc.) 1
  • Procalcitonin (may help differentiate sepsis from non-infectious causes) 2

Urinalysis and Kidney-Specific Tests

  • Urinalysis with microscopy (for casts, cells, proteinuria, hematuria) 1
  • Urine sodium and fractional excretion of sodium (FENa) to differentiate prerenal from intrinsic causes 1
  • Urine osmolality 2
  • Protein-to-creatinine ratio or albumin-to-creatinine ratio 1
  • Consider novel biomarkers when available:
    • Neutrophil gelatinase-associated lipocalin (NGAL) 1, 2
    • Cystatin C (more accurate GFR estimation than creatinine) 1
    • Proenkephalin-A (for prediction of kidney recovery) 1
    • C-C motif chemokine ligand 14 (for prediction of persistent AKI) 1

Imaging Studies

  • Renal ultrasound (first-line imaging for all AKI cases) to assess:
    • Kidney size and echogenicity
    • Presence of hydronephrosis
    • Renal perfusion with color Doppler 1, 2
  • Chest X-ray to evaluate for pneumonia or pulmonary edema 1
  • Consider CT scan without contrast if ultrasound is inconclusive 1, 2

Specialized Investigations for Cirrhotic Patients

  • Diagnostic paracentesis (mandatory in cirrhotic patients with ascites and AKI) to rule out spontaneous bacterial peritonitis 1
  • Ascitic fluid cell count, culture, and albumin level 1
  • If pleural effusion is present, diagnostic thoracentesis 1
  • Urinary NGAL (can help distinguish acute tubular necrosis from hepatorenal syndrome) 2

Cardiac Assessment

  • ECG to assess for cardiac involvement
  • Consider echocardiography to evaluate cardiac function and volume status in hemodynamically unstable patients 2

Monitoring Parameters

  • Vital signs with early warning scores (e.g., NEWS2) 1
  • Fluid balance (intake and output) and daily weight 1
  • Urine output (hourly in critically ill patients) 2
  • Hemodynamic parameters in unstable patients 1

Timing Considerations

  • Initial investigations should be performed immediately upon presentation
  • Serum creatinine and electrolytes should be monitored at least every 48 hours or more frequently in critically ill patients 1
  • Cultures should be obtained before antibiotic administration to maximize yield 1

Pitfalls and Caveats

  • Point-of-care glucose testing using capillary blood may be inaccurate in critically ill patients; arterial blood is preferred if an arterial line is available 1
  • FENa may be unreliable in patients who have recently received diuretics 1
  • Contrast agents should be avoided for imaging when possible to prevent contrast-induced nephrotoxicity 1
  • Biomarkers should be interpreted within specific clinical contexts and not in isolation 2
  • Delay in obtaining cultures before antibiotic administration increases mortality by approximately 10% for every hour in septic shock 1

By systematically applying these investigations, clinicians can rapidly identify the cause of AKI in sepsis, determine appropriate interventions, and monitor response to therapy, ultimately improving patient outcomes.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Acute Kidney Injury Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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