What is the half-life of adenosine?

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Half-Life of Adenosine

Adenosine has an extremely short half-life of less than 10 seconds in the circulation. 1, 2 This remarkably brief duration of action makes it uniquely valuable for both diagnostic and therapeutic cardiovascular applications.

Pharmacokinetics of Adenosine

Distribution and Metabolism

  • Intravenously administered adenosine distributes from circulation via cellular uptake, primarily by:
    • Erythrocytes (red blood cells)
    • Vascular endothelial cells 2
  • This process involves a specific transmembrane nucleoside carrier system that is:
    • Reversible
    • Nonconcentrative
    • Bidirectionally symmetrical 2

Elimination Pathway

  • Extracellular adenosine is primarily cleared from plasma by cellular uptake
  • Excessive amounts may be deaminated by an ecto-form of adenosine deaminase 2
  • Once inside the cell, adenosine is rapidly:
    • Phosphorylated by adenosine kinase to adenosine monophosphate, or
    • Deaminated by adenosine deaminase to inosine 2

Clinical Implications of Short Half-Life

Advantages

  • Allows for rapid titration of dosing during treatment of supraventricular tachycardia (SVT) 3
  • Adverse effects are typically transient, resolving within seconds to minutes after administration 4
  • Does not require dose adjustment in renal or hepatic impairment 2
  • Enables safe use in diagnostic procedures like stress testing 5

Administration Considerations

  • Must be administered as a rapid IV bolus followed immediately by saline flush 4
  • Should be injected into a proximal or central IV line to ensure rapid delivery to the heart 4
  • Initial dose is typically 6 mg, with subsequent doses of 12 mg if needed 4
  • Continuous ECG monitoring during administration is essential 4

Practical Applications

SVT Management

  • The ultra-short half-life makes adenosine ideal for acute termination of SVT, with success rates of 78-96% 4
  • While effective at terminating acute episodes, the short half-life means adenosine will not prevent recurrence of SVT 6
  • Additional antiarrhythmic medications should be considered if prophylaxis against recurrence is required 6

Diagnostic Applications

  • The transient AV nodal blockade produced by adenosine helps differentiate various tachyarrhythmias 4
  • Can unmask atrial flutter or atrial tachycardia by temporarily blocking AV conduction 4

Reversal of Effects

  • Due to the extremely short half-life, adverse effects typically resolve spontaneously
  • In cases of persistent adverse reactions, methylxanthines such as theophylline can be used as competitive adenosine receptor antagonists 2

The remarkably short half-life of adenosine is central to its clinical utility, allowing for both effective treatment of SVT and valuable diagnostic applications while minimizing the duration of potential adverse effects.

References

Guideline

Adenosine Therapy

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Adenosine administration for neonatal SVT.

Neonatal network : NN, 1993

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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