What are the causes of Alternating Hemiplegia of Childhood (AHC) disease?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: October 13, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Causes of Alternating Hemiplegia of Childhood (AHC)

Alternating Hemiplegia of Childhood (AHC) is primarily caused by mutations in the ATP1A3 gene, with approximately 70-80% of cases attributed to these genetic mutations. 1, 2

Primary Genetic Cause

  • ATP1A3 gene mutations are responsible for approximately 74-82% of AHC cases 2, 1
  • ATP1A3 encodes the neuronal Na+/K+ ATPase transmembrane ion transporter, which is critical for maintaining neuronal function 3

Most Common ATP1A3 Mutations in AHC

  • D801N mutation - accounts for approximately 40% of AHC cases 2
  • E815K mutation - accounts for approximately 26% of AHC cases 2
  • G947R mutation - accounts for approximately 8% of AHC cases 2
  • Most AHC cases are caused by one of seven recurrent ATP1A3 mutations 1

Genotype-Phenotype Correlations

  • E815K mutation is associated with more severe clinical manifestations compared to other mutations:

    • Earlier age of onset
    • More severe motor impairment
    • Higher prevalence of status epilepticus 2
  • D801N mutation generally presents with a less severe phenotype than E815K 2

Clinical Manifestations

  • Paroxysmal hemiplegic episodes (alternating between sides of the body) 3, 4
  • Dystonia 4
  • Seizures (occurs in 40-50% of individuals with AHC) 4
  • Motor dysfunction including:
    • Impaired coordination
    • Abnormal gait and balance
    • Low grip strength 4
  • Behavioral abnormalities:
    • Hyperactivity
    • Anxiety-like behaviors 4
  • Sleep disturbances (reduced REM sleep) 4
  • Neurodevelopmental delays 3

Pathophysiological Mechanisms

  • AHC-causing mutations in ATP1A3 lead to consistent reductions in ATPase activity without affecting protein expression levels 1
  • These mutations disrupt the function of the neuronal Na+/K+ ATPase, affecting ion transport across neuronal membranes 3
  • Neuroinflammation may play a role in disease pathogenesis 3

Inheritance Pattern

  • Most cases are sporadic, occurring due to de novo mutations 1
  • Familial cases with inherited mutations are rare but have been reported 2, 1

Important Clinical Considerations

  • AHC is distinct from other motor neuron diseases listed in your original question (ALS, PMA, PLS, PBP, SMA, poliomyelitis, and Kennedy's disease) 3, 4, 2
  • The condition typically presents in infancy or early childhood 2
  • Premature mortality can occur, particularly following seizures 4

The classification in your original question incorrectly categorized AHC as part of motor neuron disease spectrum. AHC is a distinct neurodevelopmental disorder caused primarily by ATP1A3 mutations affecting ion transport rather than a motor neuron disease.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.