Which patients are at high risk of thrombosis?

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Last updated: October 15, 2025View editorial policy

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High Thrombotic Risk Patient Identification

Patients at high risk of thrombosis include those with immobility, cancer, previous thromboembolism, and specific genetic or acquired thrombophilias. 1

Stroke and Immobility-Related Risk Factors

  • Patients unable to move one or both lower limbs 1
  • Patients unable to mobilize independently 1
  • Previous history of venous thromboembolism 1
  • Dehydration 1
  • Comorbidities such as cancer 1

Cancer-Related Risk Factors

  • Patients with advanced disease of the brain, lung, uterus, bladder, pancreas, stomach, and kidney have the highest 1-year incidence rate of venous thromboembolism (VTE) 1
  • Metastatic disease increases VTE risk 4-13 times compared to localized disease 1
  • Patients receiving chemotherapy, particularly cisplatin, fluorouracil, and immunomodulatory drugs (thalidomide, lenalidomide) 1, 2
  • Patients with multiple myeloma receiving immunomodulatory drugs combined with glucocorticoids (10-40% increased risk) 2
  • Bevacizumab (anti-VEGF therapy) increases arterial thrombotic risk and possibly venous thrombotic risk 1, 2

Cancer Risk Assessment Models

  • Khorana score identifies high-risk ambulatory cancer patients based on:

    • Cancer site (very high risk: stomach, pancreas; high risk: lung, lymphoma, gynecological, genitourinary; low risk: breast, colorectal, head and neck) 1
    • Pre-chemotherapy platelet count ≥350 × 10^9/L 1
    • Hemoglobin <10 g/dL or use of erythropoiesis-stimulating agents 1
    • Leukocyte count >11 × 10^9/L 1
    • BMI ≥35 kg/m^2 1
  • High-risk patients (score ≥3) have a 6.7% incidence of VTE 1

  • For multiple myeloma patients receiving immunomodulatory drugs, specific risk assessment models (SAVED or IMPEDE VTE) are recommended 1

Genetic and Acquired Thrombophilias

  • Factor V Leiden mutation (most common genetic cause) 1
  • Prothrombin G20210A mutation (second most common genetic cause) 1
  • Antiphospholipid syndrome (most common acquired cause) 1
  • Deficiencies of coagulation inhibitors (antithrombin III, protein C, or protein S) 1
  • Homozygous factor V Leiden carriers have significantly higher risk than heterozygous carriers 1

Cardiovascular and Cerebrovascular Risk Factors

  • Patients with mechanical heart valves, especially:

    • Older caged-ball valves (most thrombogenic) 1
    • Mitral position valves (22% annual risk without anticoagulation) 1
    • Valves with other risk factors (prior embolic event, severe left ventricular dysfunction) 1
  • Patients with non-valvular atrial fibrillation with high CHADS₂ scores (5-6) 1

  • Patients with suspected cardioembolic stroke mechanism 1

  • Patients with patent foramen ovale and cryptogenic stroke (increased paradoxical embolism risk) 1

Perioperative and Procedural Risk

  • Major surgery and laparoscopic procedures with pneumoperitoneum and reverse Trendelenburg position 1
  • Major cardiovascular surgery, orthopedic surgery, head and neck cancer surgery, urologic surgery 1
  • Surgeries lasting ≥45 minutes 1

Special Populations

  • Patients with myeloproliferative neoplasms, particularly polycythemia vera (28.6% prevalence of thrombosis) 1
  • Patients with acute lymphoblastic leukemia receiving L-asparaginase (increased CNS thrombosis risk) 2
  • Patients with thrombocytopenia from antiphospholipid antibodies (paradoxically higher thrombotic than bleeding risk) 3

Risk Stratification Approach

  • For stroke patients: assess mobility status, previous VTE history, and comorbidities 1
  • For cancer patients: use validated risk assessment models (Khorana score, SAVED, IMPEDE VTE) 1
  • For surgical patients: consider procedure type, duration, and patient-specific factors 1
  • For patients with atrial fibrillation: use CHADS₂ score 1

Important Caveats

  • Mild to moderate thrombocytopenia (>50,000/μL) should not interfere with thrombosis risk assessment, while severe thrombocytopenia (<50,000/μL) requires individualized assessment 3
  • Patients with liver cirrhosis may have bleeding abnormalities but paradoxically remain at high risk for portal and venous thromboses 3
  • Aspirin treatment alone is not as effective as heparins for VTE prevention 3
  • Genetic screening for thrombophilia is not recommended for primary prevention in asymptomatic individuals 1, 4

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Thrombotic Risk from Chemotherapy and Other Cancer Therapies.

Cancer treatment and research, 2019

Guideline

Treatment Recommendations for Prothrombin 2 (PT20210) Mutation

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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