Initial Management of Congestive Heart Failure with Trouble Breathing and Edema
The initial management for a patient presenting with signs of congestive heart failure, including trouble breathing and edema, should include immediate oxygen therapy, non-invasive ventilation if respiratory distress is present, intravenous diuretics, and appropriate monitoring of vital signs and response to treatment. 1
Immediate Assessment and Monitoring
- Establish non-invasive monitoring including pulse oximetry, blood pressure, respiratory rate, and continuous ECG within minutes of patient contact 1
- Monitor dyspnea (using visual analog scale), heart rate and rhythm, urine output, and peripheral perfusion 1
- Position patient upright to reduce work of breathing 1
- Assess severity using respiratory rate (>25/min), SpO2 (<90%), and work of breathing 1
Oxygen Therapy and Ventilatory Support
- Provide oxygen therapy if oxygen saturation <90% or based on clinical judgment 1
- Initiate non-invasive ventilation (NIV) immediately in patients showing respiratory distress 1
- Use continuous positive airway pressure (CPAP) in the pre-hospital setting as it requires minimal training and equipment 1
- Consider pressure-support positive end-expiratory pressure (PS-PEEP) for patients with acidosis and hypercapnia, particularly those with COPD history 1
- Avoid hyperoxia unless specifically indicated 1
Pharmacological Management
- Administer intravenous loop diuretics promptly 1, 2:
- Consider vasodilators for patients with normal to high blood pressure (SBP >110 mmHg) 1
- Avoid routine use of opioids as they are associated with higher rates of mechanical ventilation, ICU admission, and death 1
- Avoid sympathomimetics or vasopressors except in patients with persistent hypoperfusion despite adequate filling status 1
Laboratory and Diagnostic Tests
- Measure plasma natriuretic peptide level (BNP, NT-proBNP, or MR-proANP) in all patients with acute dyspnea and suspected AHF 1
- Perform basic laboratory assessments including troponin, BUN (or urea), creatinine, electrolytes, glucose, and complete blood count 1
- Consider chest X-ray to identify pulmonary venous congestion, pleural effusions, and interstitial or alveolar edema, though it may be normal in up to 20% of cases 1
- If expertise is available, consider bedside thoracic ultrasound to visualize interstitial edema and assess cardiac function 1
- Perform immediate echocardiography in patients with cardiogenic shock; in others, it can be performed after stabilization 1
Disposition and Further Management
- Reassess clinical, biological, and psychosocial parameters regularly 1
- Consider admission to intensive care unit/coronary care unit for patients with severe respiratory distress or hemodynamic instability 1
- For stable patients, consider observation unit (<24h) or ward admission (cardiology, internal medicine, geriatrics) 1
- Once stabilized, plan for follow-up with a cardiologist within 1-2 weeks 1
Special Considerations
- For patients with heart failure and atrial fibrillation, consider intravenous cardiac glycosides for rapid control of ventricular rate 1
- Beta-blockers are preferred first-line treatment for ventricular rate control in patients with HF and AF 1
- In patients with preserved ejection fraction (HFpEF), consider other causes of dyspnea and edema, including kidney disease, liver disease, chronic venous insufficiency, or specific cardiomyopathies 1
- For long-term management after stabilization, consider ACE inhibitors (e.g., lisinopril) which have been shown to improve symptoms including edema, rales, paroxysmal nocturnal dyspnea, and jugular venous distention 3, 4
Pitfalls to Avoid
- Do not delay treatment; the "time-to-treatment" concept is important in AHF patients 1
- Avoid routine use of morphine in AHF patients 1
- Do not assume all patients with dyspnea and edema have heart failure; consider non-cardiac mimics 1
- Avoid invasive mechanical ventilation if possible by early use of non-invasive ventilation in appropriate patients 1
- Do not neglect monitoring for electrolyte imbalances during diuretic therapy 1