What are the diagnostic criteria and treatment options for hemiplegic migraines?

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Hemiplegic Migraine: Definition and Diagnostic Criteria

Hemiplegic migraine is a rare subtype of migraine with aura characterized by fully reversible motor weakness (hemiparesis) during attacks, always accompanied by other aura symptoms, and typically followed by headache with migraine features. 1

Diagnostic Criteria

According to the International Classification of Headache Disorders (ICHD-3), hemiplegic migraine is diagnosed based on the following criteria:

Primary Criteria

  • At least 2 attacks fulfilling the criteria for migraine with aura 1
  • Aura including motor weakness (hemiparesis) 1
  • At least one of the following fully reversible aura symptoms:
    • Visual 1
    • Sensory 1
    • Speech and/or language 1
    • Motor (defining feature) 1
    • Brainstem 1
    • Retinal 1

Aura Characteristics (at least 3 of the following)

  • At least 1 aura symptom spreads gradually over ≥5 minutes 1
  • Two or more aura symptoms occur in succession 1
  • Each individual aura symptom lasts 5-60 minutes (motor symptoms may last up to 72 hours) 1, 2
  • At least 1 aura symptom is unilateral 1
  • At least 1 aura symptom is positive (e.g., scintillations, pins and needles) 1
  • The aura is accompanied by or followed within 60 minutes by headache 1

Classification Types

  • Familial Hemiplegic Migraine (FHM): At least one first- or second-degree relative has aura including motor weakness 3, 4
  • Sporadic Hemiplegic Migraine (SHM): No family history of motor weakness aura 3, 4

Clinical Features

Common Symptoms

  • Motor weakness (hemiparesis) - defining feature 3, 2
  • Sensory symptoms (paresthesia) - very common 3
  • Visual aura (scintillations, scotoma) 1, 3
  • Speech disturbance (aphasia, dysarthria) 1
  • Basilar-type symptoms (occur in up to 70% of patients) 4
  • Headache with typical migraine features (unilateral, pulsating, moderate to severe intensity) 1

Severe Attack Features

  • Prolonged hemiplegia 3, 4
  • Confusion 3, 4
  • Fever 3
  • Seizures 3, 4
  • Coma (in severe cases) 4

Associated Conditions

  • Permanent cerebellar signs (nystagmus, ataxia, dysarthria) - especially with CACNA1A mutations 4
  • Intellectual disability (less frequent) 4, 2
  • Epilepsy (in some cases) 3, 4

Genetic Aspects

  • Three main genes identified: 3, 4, 2
    • CACNA1A (chromosome 19)
    • ATP1A2 (chromosome 1)
    • SCN1A (chromosome 2)
  • All three genes encode ion transporters, supporting the channelopathy theory 3, 4
  • Genetic testing can confirm but not rule out the diagnosis (as other unknown genes may be involved) 3

Diagnostic Approach

Required Investigations

  • Detailed clinical history focusing on aura symptoms, duration, and family history 1, 2
  • Neuroimaging (MRI preferred) to exclude secondary causes 3, 2
  • Consider EEG and CSF analysis to rule out other conditions 3, 2

Contraindicated Procedures

  • Conventional cerebral angiography (may trigger attacks) 3

Treatment Options

Acute Treatment

  • Acetaminophen and NSAIDs are first-line options for headache 3
  • Triptans are controversial in hemiplegic migraine due to theoretical vasoconstriction concerns 3, 4
  • No specific treatment exists for the aura symptoms 3

Preventive Treatment

Consider when:

  • Attack frequency exceeds 2 per month 3
  • Attacks are severe and debilitating 3

First-line Preventive Options

  • Flunarizine 3
  • Sodium valproate (avoid in women of childbearing potential) 3
  • Lamotrigine 3
  • Verapamil 3
  • Acetazolamide 3

Second-line Preventive Options

  • Topiramate 3
  • Candesartan 3
  • Propranolol (controversial but can be considered) 3, 5

Emerging Treatments

  • Anti-CGRP monoclonal antibodies (e.g., galcanezumab) show promise in reducing both headache and motor weakness in hemiplegic migraine 6

Important Considerations

  • Hemiplegic migraine attacks can mimic stroke or transient ischemic attack, requiring careful differential diagnosis 2
  • The differential diagnosis is broad and includes other conditions causing transient neurological deficits 2
  • Prognosis is generally good, though some patients may develop permanent neurological symptoms 4
  • Treatment is largely empirical due to the rarity of the condition and lack of clinical trials 3, 2

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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