What are the diagnostic criteria and treatment options for seronegative autoimmune encephalitis?

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Diagnostic Criteria and Treatment Options for Seronegative Autoimmune Encephalitis

Seronegative autoimmune encephalitis requires a clinical diagnosis based on specific criteria when no identifiable antibodies are present, and early immunotherapy is essential for improved outcomes despite negative antibody testing.

Diagnostic Criteria

Major Criterion (Required)

  • Altered mental status (decreased consciousness, lethargy, or personality change) lasting ≥24 hours with no alternative cause identified 1

Minor Criteria (≥3 required for probable/confirmed seronegative autoimmune encephalitis)

  • Documented fever ≥38°C within 72 hours before or after presentation 1
  • New onset seizures not attributable to preexisting seizure disorder 1
  • New focal neurological findings 1
  • CSF white blood cell count ≥5/cubic mm 1
  • Brain MRI abnormalities suggestive of encephalitis 1
  • EEG abnormalities consistent with encephalitis and not attributable to another cause 1

Additional Supportive Features

  • Polysyndromic presentation (a clinical hallmark of autoimmune encephalitis) 1
  • Preceding viral infection, fever, or viral-like prodrome 1
  • Personal or family history of autoimmune disorders 1
  • Rapid onset with progression over days to weeks 1
  • Exclusion of infectious, toxic, metabolic, and neoplastic causes 1

Diagnostic Algorithm

Step 1: Confirm focal or multifocal brain pathology

  • Brain MRI with and without contrast (may be normal in early stages) 1
  • EEG if MRI is negative or if patient is encephalopathic/having seizures 1
  • Brain FDG-PET if MRI is negative and diagnosis remains uncertain (can be more sensitive than MRI) 1

Step 2: Confirm inflammatory etiology

  • Lumbar puncture with CSF analysis including:
    • Cell count, protein, glucose 1
    • Viral PCR studies (HSV, VZV, etc.) 1
    • Oligoclonal bands and IgG index 1
    • Comprehensive autoantibody panel (despite expected negative results) 1
  • Serum autoantibody testing (despite expected negative results) 1
  • Brain biopsy if diagnosis remains uncertain after initial testing 1

Step 3: Screen for associated neoplasm

  • CT chest, abdomen, and pelvis 1
  • Mammogram/breast MRI in women 1
  • Pelvic or testicular ultrasound 1
  • Body PET if initial screen negative 1

Treatment Approach

First-Line Immunotherapy

  • High-dose corticosteroids (methylprednisolone 1g/day for 3-5 days) 2, 3
  • Intravenous immunoglobulin (IVIG) (2g/kg divided over 2-5 days) 2, 3
  • Plasma exchange (5-7 exchanges over 10-14 days) if poor response to steroids/IVIG 2, 4

Second-Line Immunotherapy (if inadequate response to first-line)

  • Rituximab (anti-CD20 monoclonal antibody) 2, 4
  • Cyclophosphamide 2
  • Tocilizumab (IL-6 receptor antagonist) for refractory cases 2

Treatment Duration and Monitoring

  • Continue immunotherapy for at least 6 months in patients with persistent disease 2
  • Monitor response using modified Rankin Scale and Clinical Assessment Scales in Autoimmune Encephalitis 2
  • Consider maintenance immunotherapy in patients with relapsing disease 2

Prognostic Factors (RAPID Score)

The following factors are associated with poorer 2-year outcomes in seronegative autoimmune encephalitis 2:

  • Refractory status epilepticus 2
  • Age of onset ≥60 years 2
  • Probable autoimmune encephalitis subtype (vs. limbic encephalitis or ADEM) 2
  • Infratentorial involvement 2
  • Delay of immunotherapy ≥1 month 2

Important Considerations and Pitfalls

  • Don't delay treatment: Initiate immunotherapy when there is strong clinical suspicion, even without antibody confirmation 3, 4
  • Watch for super-refractory status epilepticus: This is a common presentation of seronegative autoimmune encephalitis 3
  • Consider post-infectious triggers: Autoimmune encephalitis may develop following viral infections (HSV, VZV, EBV) 5
  • Monitor for brain atrophy: Development of cerebellar atrophy on follow-up MRIs indicates poor outcomes 2
  • Be aware of psychiatric presentations: Pure neuropsychiatric features can occur with normal brain MRI 6
  • Recognize that seronegative cases may have better response to first-line therapy than seropositive cases 4
  • Consider that antibodies may be discovered later: Some currently "seronegative" cases may be found to have novel antibodies in the future 7

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Dilemmas and Challenges in Treating Seronegative Autoimmune Encephalitis in Indian Children.

Indian journal of critical care medicine : peer-reviewed, official publication of Indian Society of Critical Care Medicine, 2018

Guideline

NMDA Receptor Antibody Testing in Epilepsy

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Neuropsychiatric autoimmune encephalitis without VGKC-complex, NMDAR, and GAD autoantibodies: case report and literature review.

Cognitive and behavioral neurology : official journal of the Society for Behavioral and Cognitive Neurology, 2013

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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