What is Hallervorden-Spatz syndrome (Pantothenate Kinase-Associated Neurodegeneration, PKAN)?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: February 11, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

From the Guidelines

Hallervorden-Spatz syndrome, also known as Pantothenate Kinase-Associated Neurodegeneration (PKAN), is a rare autosomal recessive disease characterized by excess iron deposition in the basal ganglia with progressive neuronal degeneration.

Key Characteristics

  • Annual incidence of 1/1,000 1
  • Classically presents in the first decade with slowly progressive gait disturbances, dystonia, dysarthria, spasticity, and pyramidal tract signs 1
  • NBIA type 1 is the most common subtype of Neurodegeneration with Brain Iron Accumulation (NBIA) 1

Diagnosis

  • MRI is the preferred imaging modality for suspected NBIA due to its sensitivity to basal ganglia iron accumulation 1
  • CT Head may be helpful to distinguish between calcium and iron deposition in the brain, but its utility is limited 1
  • A combination of imaging techniques, including structural imaging and advanced MRI techniques, may be needed for complete evaluation of the patient 1

From the Research

Definition and Characteristics

  • Hallervorden-Spatz syndrome (HSS), also known as pantothenate kinase-associated neurodegeneration (PKAN), is a rare inherited neurodegenerative disorder with childhood, adolescent, or adult onset 2.
  • PKAN is characterized by a progressive movement disorder and prominent iron deposition in the globus pallidus 3.
  • The disorder is associated with a combination of motor symptoms, including dystonia, parkinsonism, choreoathetosis, corticospinal tract involvement, optic atrophy, pigmentary retinopathy, and cognitive impairment 2.

Clinical Features

  • Patients with PKAN may exhibit a range of clinical features, including misalignment of teeth, a high arched palate, hollow feet, slight cognitive decline, and apparent executive dysfunction 4.
  • The presence of a mutation in the PANK2 gene is associated with younger age at onset and a higher frequency of dystonia, dysarthria, intellectual impairment, and gait disturbance 2.
  • Parkinsonism is seen predominantly in adult-onset patients, whereas dystonia seems more frequent in earlier-onset cases 2.

Diagnosis and Imaging

  • The characteristic MRI brain pattern of PKAN shows the "eye-of-the-tiger" sign 5.
  • Brain perfusion single photon emission computed tomography (SPECT) may show decreased regional cerebral blood flow in the bilateral frontoparietal lobes, the globus pallidus, the striatum, and around the ventriculus quartus 4.

Genetic Basis

  • PKAN is caused by a defect in the pantothenate kinase 2 (PANK2) gene, which is responsible for iron metabolism in the brain 6.
  • Mutations in the PANK2 gene are associated with the development of PKAN, and different mutations may result in varying clinical phenotypes 2, 4.

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.