Evaluation and Management of Decreased Left Upper Extremity Muscle Strength
The patient requires immediate neurological assessment to determine if this represents an acute stroke, followed by structured rehabilitation including strengthening exercises if stroke is confirmed. 1
Initial Evaluation
Acute Stroke Assessment
- Administer the National Institutes of Health Stroke Scale (NIHSS) immediately upon presentation or within 24 hours to assess stroke severity and guide acute treatment decisions 1
- The NIHSS should include assessment of upper extremity motor function, with an additional item examining finger extension (20 degrees wrist extension, 10 degrees finger extension) even though this doesn't contribute to the total score 1
- Obtain urgent neuroimaging (MRI with diffusion-weighted imaging) to identify the lesion location and type 2
Localization Considerations
Upper extremity monoparesis can result from several distinct anatomical locations:
- Cortical infarctions in the parietal lobe or central sulcus region (most common for isolated arm weakness, <1% of all strokes) 2
- Pontine lesions in the paramedian area (extremely rare cause of isolated brachial monoparesis) 3
- Lateral medullary infarction affecting corticospinal tract fibers (rare) 4
Detailed Motor Assessment
- Measure specific muscle group strength using manual muscle testing or hand-held dynamometry, focusing on:
- Assess both magnitude and time-dependent properties of muscle contraction, as both peak torque and speed of torque generation are impaired after stroke 6
- Evaluate the contralateral ("unaffected") arm as well, since peak torque and time to develop torque are often impaired bilaterally, with the affected arm showing approximately 39% overall weakness compared to normal 6, 5
Functional Assessment
- Apply standardized functional scales including:
Range of Motion and Contracture Assessment
- Examine upper extremity joints systematically: elbow, wrist, and long finger flexors for emerging hypoextensibility and contractures that could contribute to functional deterioration 1
Management Strategy
Acute Phase Rehabilitation
Strengthening exercises should be included immediately in acute rehabilitation for patients with muscle weakness after stroke. 1 This recommendation is based on the positive relationship between muscle strength, function, and prevention of falls, despite limited research specifically during the acute rehabilitation phase 1
Constraint-Induced Movement Therapy (CI Therapy)
- Consider CI therapy only for highly selected patients who meet strict criteria:
- This intensive therapy involves forced use of the affected extremity while discouraging use of the unaffected arm 1
Functional Electrical Stimulation
- FES may be considered as a time-limited intervention during the first several weeks after stroke, applied to cause muscle contraction 1
Therapeutic Exercise Principles
- Challenge patients to improve both strength AND speed of muscle contraction, as both magnitude and time-dependent properties are impaired 6
- Engage the patient in tasks promoting normal movement patterns, good alignment, and functional use:
- Avoid strategies that increase attention to the limb or promote compensatory patterns 1
Monitoring and Reassessment
- Reassess using NIHSS at acute care discharge 1
- Schedule routine clinic appointments every 6 months with specialist physical and occupational therapy assessments every 4 months 1
- Monitor for complications: contractures, learned non-use, pain, and functional decline 1
Critical Pitfalls to Avoid
- Do not misdiagnose as peripheral nervous system disorder simply because pyramidal tract signs, facial involvement, or leg weakness are absent—isolated arm weakness can be cortical 2
- Do not focus solely on the affected limb—the "unaffected" arm also demonstrates impairments in peak torque and contraction speed 6
- Do not use splinting routinely, as it may increase attention to the area, promote compensatory movements, cause muscle deconditioning, and lead to learned non-use 1
- Do not emphasize spasticity management at the expense of addressing underlying weakness, as traditional facilitation models have overemphasized spasticity 1