What is lymphoma?

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What is Lymphoma?

Lymphoma is a group of malignant neoplasms arising from clonal proliferation of lymphocytes within the lymphatic system, comprising over 90 distinct subtypes that are broadly classified into two main categories: Hodgkin lymphoma (HL) and non-Hodgkin lymphoma (NHL). 1, 2

Classification and Subtypes

Lymphoma is traditionally divided into two major categories based on the WHO classification 3:

Hodgkin Lymphoma (HL)

  • Classical Hodgkin lymphoma (CHL) accounts for approximately 95% of all HL cases and is characterized by the presence of Reed-Sternberg cells (large malignant cells expressing CD15+/CD30+/CD20-) in an inflammatory background 3
  • CHL includes four histologic subtypes: lymphocyte-rich, nodular sclerosis, mixed cellularity, and lymphocyte-depleted 3
  • Nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL) represents approximately 5% of cases, characterized by lymphocyte-predominant cells (L & H cells or "popcorn cells") with a distinct immunophenotype (CD15-/CD30-/CD20+) 3

Non-Hodgkin Lymphoma (NHL)

  • Represents approximately 90% of all lymphomas and includes over 30 unique subtypes 1, 2
  • B-cell lymphomas account for approximately 90% of NHL cases 4
  • T-cell and NK-cell lymphomas comprise the remaining cases 3

Major B-Cell NHL Subtypes 3:

  • Diffuse large B-cell lymphoma (DLBCL): accounts for 30-40% of adult NHL, representing the most common aggressive subtype 5
  • Follicular lymphoma: second most frequent nodal lymphoid malignancy in Western Europe 3
  • Chronic lymphocytic leukemia/small lymphocytic lymphoma
  • Mantle cell lymphoma
  • Marginal zone lymphomas (MALT type, nodal, splenic)
  • Burkitt's lymphoma

T-Cell and NK-Cell NHL Subtypes 3:

  • Peripheral T-cell lymphomas
  • Extranodal NK/T-cell lymphoma, nasal type
  • Mycosis fungoides/Sézary syndrome
  • Anaplastic large cell lymphoma

Epidemiology

Incidence

  • Hodgkin lymphoma: crude incidence of 2.3 cases per 100,000 population per year in the European Union, with mortality of 0.4 per 100,000 per year 5
  • DLBCL: crude incidence of 3.8 cases per 100,000 per year in Europe 5
  • Approximately 82,000 new lymphoma cases are diagnosed annually in the United States 1
  • NHL incidence has increased worldwide by approximately 30% in the 5 years prior to 2010 5

Age Distribution

  • HL demonstrates a bimodal age distribution with peaks in young adults aged 20-40 years and in individuals aged 55 years and older 3, 5
  • Most HL patients are diagnosed between 15 and 30 years of age 3
  • NHL can affect all age groups but incidence increases with age 1

Gender

  • Slightly more men than women are diagnosed with HL 5

Pathophysiology

Cellular Origin

  • Hodgkin lymphoma: malignant Reed-Sternberg cells represent only 0.1-1% of the tumor mass, with the remainder consisting of a heterogeneous inflammatory infiltrate including lymphocytes, histiocytes, eosinophils, plasma cells, and fibroblasts 3
  • NHL: arises from clonal proliferation of lymphocytes at various stages of differentiation 2
  • Most lymphomas are of B-cell origin, though T-cell and NK-cell variants exist 4

Immunophenotyping

Critical for diagnosis and classification 3:

  • Classical HL: CD15+, CD30+, CD20- 3
  • NLPHL: CD15-, CD30-, CD20+ 3
  • B-cell NHL: typically CD20+, CD79a+ with variable expression of other markers 3
  • T-cell NHL: variable expression of CD2, CD3, CD4, CD5, CD7, CD8 3
  • NK-cell lymphomas: CD2+, surface CD3-, cytoplasmic CD3ε+, CD56+, EBV-EBER+ 3

Clinical Presentation

Common Presenting Features

  • Painless lymphadenopathy is the most common presentation, with over 60% of HL patients initially observing enlarged cervical lymph nodes 3, 1
  • Lymphoma typically presents as painless adenopathy in accessible lymph node regions 1

B Symptoms

Present in more advanced disease and include 3, 1:

  • Fever (>38°C)
  • Unexplained weight loss (>10% body weight in 6 months)
  • Drenching night sweats

Site-Specific Presentations

  • Extranodal NK/T-cell lymphoma: predominantly affects the upper aerodigestive tract (nasal cavity, nasopharynx, paranasal sinuses), but can involve skin, testis, and gastrointestinal tract 3
  • Primary cutaneous lymphomas: present with patches, plaques, nodules, or tumors on the skin 3
  • Other lymphoid organs that can be involved include spleen, liver, bone marrow, and lung 3

Diagnosis

Tissue Biopsy Requirements

  • Excisional lymph node biopsy is the gold standard for diagnosis of lymphoma 3, 1
  • Core needle biopsies should only be performed when easily accessible lymph nodes are not available (e.g., retroperitoneal masses) 3
  • Fine needle aspiration is inappropriate for reliable diagnosis 3
  • For NK/T-cell lymphomas, biopsy specimens should include edges of lesions to increase odds of obtaining viable tissue, as necrosis is very common 3

Pathologic Evaluation

The histological report must include 3:

  • Diagnosis according to WHO classification
  • Histologic grade (for follicular lymphoma: grade 1-2 with ≤15 blasts/high-power field; grade 3 with >15 blasts) 3
  • Specification if more than one histologic type is present (composite lymphoma)
  • Statement regarding specimen adequacy
  • Results of ancillary studies (immunohistochemistry, molecular diagnostics) 3

Essential Immunophenotyping

Must specify 3:

  • All markers investigated (both positive and negative)
  • CD nomenclature for antigen identification
  • Which cell population expresses each antigen
  • Percentage of positive cells when relevant
  • Whether expression is focal or diffuse

Molecular Studies

  • Molecular analysis to detect clonal T-cell receptor (TCR) gene rearrangements for T-cell lymphomas 3
  • EBV viral load by quantitative PCR is important for diagnosis and monitoring of NK/T-cell lymphomas; lack of normalization suggests persistent disease 3
  • Clonality assessment may be helpful but cannot definitively distinguish low-grade B-cell lymphomas from reactive processes 3

Staging and Risk Assessment

Staging System

  • Ann Arbor staging system (modified by Cotswolds classification) is used for HL 3
  • Lugano Classification has modernized staging recommendations for both HL and NHL, formally incorporating FDG-PET/CT 5

Risk Stratification for HL 3:

Early-stage favorable (Stage I-II without risk factors)

Early-stage unfavorable (Stage I-II with any of the following):

  • Bulky mediastinal disease (mediastinal mass ratio >0.33) or bulky disease >10 cm
  • B symptoms
  • ESR >50 (or >30 without B symptoms)
  • More than 3 nodal sites of disease
  • Extranodal disease

Advanced-stage (Stage III-IV):

  • International Prognostic Score (IPS) determines prognosis based on number of adverse factors present at diagnosis 3

Essential Staging Workup for HL 3:

  • History and physical examination with attention to all node-bearing areas including Waldeyer's ring
  • Assessment for B symptoms
  • Performance status
  • Complete blood count with differential
  • Comprehensive metabolic panel, LDH, uric acid
  • Bone marrow biopsy and aspirate
  • CT scans of chest, abdomen, and pelvis with contrast or PET/CT scan
  • Echocardiogram or MUGA scan if anthracycline-containing regimens planned

Staging for NK/T-Cell Lymphoma 3:

  • ENT evaluation of nasopharynx
  • Dedicated CT or MRI of nasal cavity, hard palate, anterior fossa, nasopharynx
  • Calculation of Prognostic Index of Natural Killer Lymphoma (PINK)
  • EBV viral load by quantitative PCR
  • Bone marrow biopsy showing EBER-1 positive lymphoid aggregates indicates involvement

Modern Staging Considerations

  • FDG-PET/CT has been formally incorporated into standard staging for FDG-avid lymphomas 5
  • Bone marrow biopsy is no longer indicated for routine staging of HL and most diffuse large B-cell lymphomas 5

Treatment Principles

Hodgkin Lymphoma

  • Chemotherapy or combined modality therapy (chemotherapy plus radiotherapy) is standard initial treatment 3
  • Early-stage favorable disease: 2 cycles of ABVD followed by 30 Gy involved field radiotherapy 3
  • Advanced-stage disease: ABVD, Stanford V, or BEACOPP chemotherapy regimens with or without radiotherapy 1
  • Relapsed/refractory disease: brentuximab vedotin (CD30-directed antibody-drug conjugate) has shown encouraging results 3
  • High-dose chemotherapy with autologous stem cell transplantation for recurrent disease 6

Non-Hodgkin Lymphoma

Aggressive NHL (e.g., DLBCL) 7, 1:

  • R-CHOP (rituximab, cyclophosphamide, doxorubicin, vincristine, prednisone) is standard first-line therapy
  • Treated with curative intent due to chemosensitivity despite aggressive behavior 4

Indolent NHL (e.g., follicular lymphoma) 3:

  • Stage I-II: radiotherapy (30-40 Gy involved or extended field) has curative potential
  • Stage III-IV: chemotherapy initiated only upon occurrence of symptoms, B symptoms, hematopoietic impairment, bulky disease, or rapid progression
  • Rituximab in combination with chemotherapy (CHOP, CVP, or purine analog-based regimens) for complete remission
  • Generally incurable, requiring balance between quality of life and treatment toxicity 4

Pediatric mature B-cell NHL and B-cell acute leukemia 7:

  • Rituximab in combination with chemotherapy for previously untreated advanced-stage disease

Treatment Considerations

  • Long-term toxicities include neuropathy, cardiotoxicity, and secondary cancers (lung, breast) that must be discussed in shared decision-making 1
  • Potential long-term treatment effects remain an important consideration requiring long-term follow-up 3

Prognosis

Hodgkin Lymphoma

  • HL is now curable in at least 80% of patients 3
  • 5-year survival rates have improved dramatically over the past four decades 3
  • Mortality rate is 0.4 per 100,000 per year in the European Union 5

Non-Hodgkin Lymphoma

  • Prognosis varies widely depending on subtype and stage 2
  • Aggressive lymphomas are more dangerous if untreated but more chemosensitive, allowing curative treatment 4
  • Indolent lymphomas are largely incurable but have prolonged survival with appropriate management 4
  • Smoking, alcohol consumption, and obesity before diagnosis are associated with poorer overall and lymphoma-specific survival 5

Risk Factors

Modifiable Risk Factors 5, 1:

  • Tobacco use
  • Obesity (particularly high body mass as a young adult)
  • Certain occupational exposures
  • Environmental factors including pesticide exposure

Non-Modifiable Risk Factors 5:

  • Family history of lymphoma
  • Autoimmune diseases
  • HIV infection
  • Hepatitis C virus (HCV) seropositivity
  • Infectious diseases (including EBV for certain subtypes)

Surveillance and Follow-Up

Post-Treatment Monitoring

  • Routine surveillance is essential to monitor for complications and relapse 1
  • PET/CT restaging using Deauville criteria (5-point scale) to assess treatment response 3
  • Age-appropriate cancer screenings per U.S. Preventive Services Task Force recommendations 1

Immunizations

  • 13-valent pneumococcal conjugate vaccine followed by 23-valent pneumococcal polysaccharide vaccine at least 8 weeks later 1
  • Additional age-appropriate vaccinations due to immunosuppressive nature of lymphoma 1
  • Household contacts should maintain current immunization status 1

References

Research

Lymphoma: Diagnosis and Treatment.

American family physician, 2020

Research

Overview of lymphoma diagnosis and management.

Radiologic clinics of North America, 2008

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Lymphoma.

Primary care, 2016

Guideline

Epidemiology of Lymphoma

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Hodgkin lymphoma.

Nature reviews. Disease primers, 2020

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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