What are the management options for skin and soft tissue infections, including severity criteria and treatment options?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: November 22, 2025View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Management of Skin and Soft Tissue Infections

Classification Framework

SSTIs must be classified by three key characteristics: necrotizing versus non-necrotizing, anatomical depth, and presence of purulence. 1

Severity Stratification

Severity assessment determines treatment intensity and should be based on systemic inflammatory response criteria:

  • Mild (Class 1): No systemic signs, no significant comorbidities 2
  • Moderate (Class 2): Presence of SIRS criteria (temperature >38°C or <36°C, heart rate >90 bpm, respiratory rate >24/min, WBC >12,000 or <4,000 cells/μL) OR systemically well with complicating comorbidities 1, 2, 3
  • Severe (Class 3-4): Toxic appearance, treatment failure, immunocompromised status, hypotension, or signs of deeper infection (bullae, skin sloughing, disproportionate pain, rapid progression) 1, 2, 3

Infection-Specific Management

1. Impetigo

Pathogen: Staphylococcus aureus and/or β-hemolytic Streptococcus (including increasing CA-MRSA) 1, 4

Treatment:

  • Limited lesions: Topical mupirocin or retapamulin ointment twice daily 1
  • Extensive lesions: Oral antibiotics
    • Dicloxacillin 250 mg QID 1
    • Cephalexin 250 mg QID (pediatric: 25-50 mg/kg/day divided 3-4 times) 1
    • Clindamycin 300-400 mg QID (pediatric: 20 mg/kg/day divided 3 times) if MRSA suspected 1
    • Amoxicillin-clavulanate 875/125 mg BID 1

2. Erysipelas

Pathogen: Primarily Streptococcus pyogenes (Group A Streptococcus) 4

Treatment:

  • Mild cases: Oral β-lactams (cephalexin 500 mg QID or dicloxacillin 500 mg QID) 1
  • Severe cases: IV penicillin or cefazolin 1

3. Cellulitis

Pathogen: Streptococci (diffuse infection) or staphylococci (more localized) 4, 5

Treatment approach by severity:

Mild cellulitis (no systemic signs):

  • Oral β-lactams: Cephalexin 500 mg QID or dicloxacillin 500 mg QID 1
  • Penicillin-allergic: Clindamycin 300-450 mg QID 1

Moderate cellulitis (SIRS present):

  • If MRSA not suspected: Cefazolin 1 g IV every 8 hours 1
  • If MRSA suspected: Add vancomycin 30 mg/kg/day divided every 12 hours IV 1

Severe cellulitis (toxic appearance, hypotension, organ dysfunction):

  • Vancomycin 30 mg/kg/day IV PLUS piperacillin-tazobactam or carbapenem for polymicrobial coverage 2, 4

4. Abscesses and Furuncles

Pathogen: Staphylococcus aureus (including MRSA) 1, 5

Treatment:

Simple abscesses:

  • Incision and drainage is the primary treatment; antibiotics are NOT routinely needed 1, 4
  • Add antibiotics ONLY if: systemic signs present, immunocompromised, incomplete source control, or significant surrounding cellulitis (>2 cm) 4

Complex abscesses (perianal, perirectal, injection drug use sites):

  • Incision and drainage PLUS broad-spectrum antibiotics covering Gram-positives, Gram-negatives, and anaerobes 4
  • Empiric regimen: Vancomycin PLUS piperacillin-tazobactam or carbapenem 2, 4

Carbuncles with treatment failure or SIRS:

  • MRSA-active antibiotic required: vancomycin, linezolid, or daptomycin 1

5. Bite Wounds (Animal and Human)

Infection risk: Cat bites 30-50%, dog bites 5-25%, human bites 20-25% 4

Pathogens: Polymicrobial (oral flora, Pasteurella in animals, Eikenella in humans, anaerobes) 4

Treatment:

  • Early aggressive surgical debridement PLUS antibiotics 4
  • Amoxicillin-clavulanate 875/125 mg BID (first-line oral) 1
  • Severe infections: Ampicillin-sulbactam 1.5-3 g IV every 6 hours or piperacillin-tazobactam 4
  • Avoid high-pressure irrigation—this drives bacteria deeper into tissue 4

6. Traumatic Wounds

Treatment:

  • Irrigation and debridement of necrotic tissue are the most critical interventions 4
  • Prophylactic antibiotics NOT routinely recommended 4
  • Antibiotics required if: systemic signs, immunocompromised, severe comorbidities, severe/deep wounds, or significant cellulitis 4
  • Empiric regimen: Broad-spectrum covering aerobes and anaerobes (amoxicillin-clavulanate or piperacillin-tazobactam) 4

7. Necrotizing Soft Tissue Infections

Clinical red flags requiring immediate surgical consultation:

  • Pain disproportionate to physical findings 2
  • Violaceous bullae or skin sloughing 2
  • Skin anesthesia 2
  • Rapid progression 2, 3
  • Gas in tissue on imaging 2

Classification:

  • Type I: Polymicrobial (mixed aerobes and anaerobes) 4
  • Type II: Monomicrobial (Streptococcus pyogenes or MRSA) 4
  • Type III: Gas gangrene (Clostridium species) 4

Treatment:

Immediate aggressive surgical debridement is ESSENTIAL—survival depends on it 1, 2, 4

Empiric antibiotic regimen (broad-spectrum polymicrobial coverage):

Gram-positive coverage (including MRSA):

  • Vancomycin 30 mg/kg/day IV 2, 4
  • OR Linezolid 600 mg IV/PO every 12 hours 2, 4, 6
  • OR Daptomycin 4-6 mg/kg IV daily 2, 4

PLUS Gram-negative and anaerobic coverage:

  • Piperacillin-tazobactam 3.375-4.5 g IV every 6 hours 2, 4
  • OR Carbapenem (imipenem, meropenem, or ertapenem) 2, 4
  • OR Ceftriaxone 1-2 g IV daily PLUS metronidazole 500 mg IV every 8 hours 2, 4
  • OR Fluoroquinolone PLUS metronidazole 2, 4

For confirmed Group A Streptococcus necrotizing fasciitis:

  • Clindamycin 600-900 mg IV every 8 hours PLUS penicillin G 4 million units IV every 4 hours (clindamycin suppresses toxin production) 4

Duration: Continue antibiotics until no further debridement needed, clinical improvement achieved, and afebrile for 48-72 hours 4


8. Diabetic Foot Infections

Pathogen: Often polymicrobial; evaluate for underlying osteomyelitis 2

Treatment:

  • Always assess for osteomyelitis with imaging (plain radiographs initially) 2
  • Mild infections: Amoxicillin-clavulanate 875/125 mg BID 1
  • Moderate-severe infections: Linezolid 600 mg BID OR vancomycin PLUS piperacillin-tazobactam or carbapenem 2, 6
  • More aggressive management required than non-diabetic SSTIs 2

Antibiotic Selection by Pathogen

MSSA (Methicillin-Susceptible S. aureus)

Parenteral:

  • Nafcillin or oxacillin 1-2 g IV every 4 hours (drug of choice) 1
  • Cefazolin 1 g IV every 8 hours (for penicillin allergy, except immediate hypersensitivity) 1

Oral:

  • Dicloxacillin 500 mg QID 1
  • Cephalexin 500 mg QID 1

MRSA (Methicillin-Resistant S. aureus)

Parenteral:

  • Vancomycin 30 mg/kg/day IV divided every 12 hours (drug of choice) 1, 4
  • Daptomycin 4-6 mg/kg IV daily 1, 2
  • Linezolid 600 mg IV every 12 hours 1, 2
  • Ceftaroline 600 mg IV every 12 hours 1

Oral:

  • Linezolid 600 mg PO every 12 hours 1, 6
  • Clindamycin 300-450 mg QID (if susceptible; check for inducible resistance) 1, 7
  • Doxycycline or minocycline 100 mg BID 1
  • Trimethoprim-sulfamethoxazole 1-2 double-strength tablets BID 1

Diagnostic Workup

Obtain cultures when:

  • Moderate or severe infection 1, 2
  • Treatment failure 1
  • Immunocompromised host 1
  • Systemic signs present 1

Culture methods:

  • Needle aspiration, tissue biopsy, or swab of purulent drainage 2
  • Blood cultures if bacteremia suspected 2

Imaging:

  • Plain radiographs to evaluate for gas, foreign bodies, or osteomyelitis 2
  • Consider CT or MRI for deep infections or necrotizing fasciitis 2

Treatment Duration

  • Simple SSTIs: 5-7 days (if clinical improvement) 2
  • Complicated SSTIs: 7-14 days 2
  • Necrotizing infections: Until no further debridement needed and afebrile 48-72 hours 4
  • Osteomyelitis: 4-6 weeks minimum 2

Monitoring and Reassessment

Reassess within 24-48 hours to evaluate treatment response 2

Signs of treatment failure requiring escalation:

  • Worsening erythema or pain 2
  • New systemic signs 2
  • Lack of clinical improvement 2

Consider repeat surgical debridement if no clinical improvement in necrotizing infections 2

Adjust antibiotics based on culture results and clinical response 2

Monitor for complications: bacteremia, osteomyelitis, sepsis 2


Critical Pitfalls to Avoid

  • Never delay surgical consultation for suspected necrotizing infections—mortality increases with each hour of delay 1, 2
  • Do not use antibiotics alone for abscesses—incision and drainage is mandatory 1, 4
  • Avoid high-pressure wound irrigation—it spreads bacteria into deeper tissue planes 4
  • Do not assume all cellulitis is streptococcal—MRSA coverage needed if purulence, abscess, or treatment failure 1, 3
  • Immunocompromised patients require immediate treatment and broader coverage for atypical organisms 2, 3, 5
  • Leukopenia (<4,000 cells/μL) is as concerning as leukocytosis and indicates severe infection 1, 3
  • Clindamycin resistance can be inducible in MRSA—perform D-test if using for MRSA 1
  • Consider recurrent MRSA decolonization protocols for patients with multiple episodes 2

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Management of Deep Tissue Infections

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Classification of Purulent Skin and Soft Tissue Infections

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Tratamiento de Infecciones de Tejidos Blandos

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Skin and Soft Tissue Infections.

American family physician, 2015

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.