What is the half-life of paroxetine (Selective Serotonin Reuptake Inhibitor)?

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Paroxetine Half-Life

Paroxetine has an elimination half-life of approximately 21 hours (with a coefficient of variation of 32%) after steady-state dosing, making once-daily administration appropriate. 1

Pharmacokinetic Details

Half-Life Characteristics

  • The mean elimination half-life is 21 hours at steady state after 30 days of daily dosing with 30 mg tablets 1
  • There is substantial interindividual variability, with reported ranges from 7 to 65 hours in healthy young subjects 2
  • This 24-hour approximate half-life supports once-daily dosing and allows steady-state concentrations to be achieved within a reasonable timeframe 3

Time to Steady State

  • Steady-state plasma concentrations are generally achieved within 7 to 14 days, though it may take substantially longer in occasional patients 1, 2
  • Approximately 10 days is typical for most subjects to reach steady state 1

Clinical Implications of Paroxetine's Half-Life

Comparison to Other SSRIs

  • Paroxetine's half-life is significantly shorter than fluoxetine (which has a 2-4 day half-life with an active metabolite lasting 7-15 days), making dose adjustments more predictable 3
  • The half-life is similar to sertraline (approximately 24 hours) but paroxetine exhibits nonlinear pharmacokinetics at higher doses due to saturable metabolism, unlike sertraline 4, 3
  • This half-life range (21-36 hours among SSRIs excluding fluoxetine) is consistent across most SSRIs except fluoxetine 4

Therapeutic Drug Monitoring Considerations

  • Blood samples for therapeutic drug monitoring should be collected at steady state (at least 5 half-lives after dose changes), which for paroxetine means approximately 1 week after stable dosing 5
  • Sampling should occur 12-16 hours after the last dose (or 24 hours if dosed once daily) during the terminal elimination phase 5

Important Caveats

  • The half-life becomes longer at higher doses due to paroxetine's inhibition of its own clearance via saturable CYP2D6 metabolism 3, 1
  • Elderly patients show significantly slower elimination with higher plasma concentrations than younger subjects, though with wide interindividual variation and overlap 1, 2
  • Severe renal impairment (creatinine clearance <30 mL/min) results in approximately 4-fold higher plasma concentrations, and hepatic impairment causes approximately 2-fold increases, both affecting effective half-life 1

References

Research

A review of the metabolism and pharmacokinetics of paroxetine in man.

Acta psychiatrica Scandinavica. Supplementum, 1989

Research

Pharmacokinetics of selective serotonin reuptake inhibitors.

Pharmacology & therapeutics, 2000

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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