What is the treatment for central serous chorioretinopathy (CSC)?

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Treatment of Central Serous Chorioretinopathy

For acute CSC presenting within the first 4 months, observation is the standard approach unless the patient requires rapid visual recovery for professional reasons, has recurrent episodes, or bilateral disease—in which case ICGA- and FA-guided half-dose photodynamic therapy should be performed immediately. 1, 2

Acute Central Serous Chorioretinopathy (aCSC)

Initial Management Strategy

  • Observation for 4 months is recommended for first-time acute cases with a single focal leak and minimal RPE changes (less than 1-disc area), given the high rate of spontaneous subretinal fluid resolution during this timeframe. 1, 2

  • OCT imaging is critical for diagnosis and monitoring, as subretinal fluid can be too shallow to detect on slit-lamp biomicroscopy, and residual fluid can cause photoreceptor and RPE atrophy leading to vision loss over years. 1

Indications for Immediate Treatment (No Observation Period)

Proceed directly to half-dose PDT without observation in these specific scenarios:

  • Patients requiring rapid visual recovery for professional reasons 1, 2
  • Recurrent active aCSC (previous episodes of subretinal fluid) 1, 2
  • Bilateral active disease 1, 2

Treatment Options for Acute CSC

Half-dose PDT is the treatment of choice when intervention is indicated, achieving faster subretinal fluid resolution, more rapid recovery of retinal sensitivity, and lower recurrence rates (25%) compared to spontaneous resolution (51.2% recurrence). 1, 2

Technical Specifications for PDT:

  • Use ICGA-guided half-dose PDT to optimally treat underlying choroidal abnormalities 1
  • Target hyperfluorescent areas on ICGA that correspond to focal leakage on FA and subretinal fluid on OCT 1, 2
  • Half-dose PDT is preferred over half-fluence or half-time PDT, as large RCTs demonstrate high efficacy in chronic CSC with minimized local and systemic side effects 1
  • Half-dose protocol allows one vial of verteporfin to treat two patients, reducing costs and increasing availability 1

Argon Laser Photocoagulation:

  • Use only when focal leak on FA is located at a safe distance from the fovea 1
  • Do not use thermal laser photocoagulation for underlying choroidal abnormalities 1
  • Carries significant risks: symptomatic paracentral scotoma, macular neovascularization, and chorioretinal adhesion with secondary intraretinal cystoid fluid 1, 2

Important Caveat

Opting for a short observation period of a few months does not appear to affect longer-term outcomes in aCSC, so the decision to observe versus treat early should be based on the specific clinical scenarios outlined above. 1


Chronic Central Serous Chorioretinopathy (cCSC)

Definition and Treatment Goals

  • Chronic CSC is defined by persistent subretinal fluid on OCT for longer than 4 months, with more than 1-2 disc areas of atrophic RPE changes, and focal or diffuse leakage on FA with hyperfluorescent choroidal abnormalities on ICGA. 1, 2

  • The treatment goal is to prevent irreversible photoreceptor damage and vision loss by achieving complete subretinal fluid resolution and reversing photoreceptor/RPE dysfunction, as persistent subretinal fluid leads to irreversible vision loss and reduced quality of life. 1, 2

First-Line Treatment

ICGA- and FA-guided half-dose (or half-fluence) PDT is the first-line treatment for chronic CSC, with large RCTs demonstrating 21-100% complete subretinal fluid resolution rates and superior functional improvement compared to other treatments. 1, 2

Evidence Supporting PDT Superiority:

  • The PLACE trial showed half-dose PDT was superior to high-density subthreshold micropulse laser (HSML), with complete subretinal fluid resolution in 67% versus 29% of patients at 7-8 months. 1
  • The SPECTRA trial demonstrated half-dose PDT led to significantly more complete subretinal fluid resolution and functional improvement compared to eplerenone. 1
  • PDT provides faster subretinal fluid resolution, more rapid recovery of retinal sensitivity, and higher best-corrected visual acuity compared to observation. 1, 3, 4

Treatment Algorithm for Persistent Subretinal Fluid After Initial PDT

If subretinal fluid persists after initial half-dose PDT:

  1. Re-treatment with another half-dose or half-fluence PDT guided by FA/ICGA 1, 2
  2. Full-dose with full-fluence PDT may also be considered 1
  3. High-density subthreshold micropulse laser (inferior to PDT but acceptable alternative) 1, 2
  4. Mineralocorticoid receptor antagonist (limited evidence) 1, 2
  5. Conservative observation if only small amount of residual subretinal fluid 1, 2

Alternative Treatments When PDT is Unavailable

Conventional argon laser photocoagulation can be used for chronic CSC with focal, non-central leakage on angiography when PDT is unavailable, but only when the leak is at a safe distance from the fovea. 1, 4

High-density subthreshold micropulse laser has been studied but shows inferior results compared to half-dose PDT in both focal and diffuse phenotypes of chronic CSC. 1, 2

Mineralocorticoid receptor antagonists (eplerenone, spironolactone) may be considered when PDT is unavailable or as adjunctive therapy, although the VICI trial showed non-superiority of eplerenone compared to placebo. 1, 2, 5

Selective retina therapy (SRT) has shown variable results, but the evidence quality is insufficient to recommend it over PDT. 2, 4

Anti-VEGF agents are not effective for chronic CSC in the absence of choroidal neovascularization. 4, 6


CSC with Concurrent Macular Neovascularization

When macular neovascularization is present (detected using OCT, OCT-A, FA, and ICGA):

  • Treat with half-dose/half-fluence PDT and/or intravitreal anti-VEGF injections 1
  • When a neovascular component of polypoidal choroidal vasculopathy (aneurysmal type 1 neovascularization) is present, PDT (either full-dose, half-dose, or half-fluence) can be added to anti-VEGF treatment 1

Critical Considerations

Corticosteroid Use

If the patient is currently taking corticosteroids, discuss stopping their use prior to treatment, as corticosteroids are a known risk factor for CSC. 1, 7

Post-PDT Precautions

  • Patients must avoid direct sunlight and UV radiation for 48-72 hours after receiving PDT 3
  • Wear protective clothing and stay out of the sun for 72 hours following the procedure 3

Follow-up Timing

  • Follow-up imaging at 4-6 weeks post-treatment is appropriate to assess treatment response 3
  • For observation cases, follow-up with OCT after 1-3 months to assess for complete subretinal fluid resolution 1

Potential PDT Complications

  • Significant RPE damage (with conventional subthreshold micropulse laser) 1
  • Grade 1 choroidal ischemia (3/51 eyes receiving PDT) 1
  • Photodynamic therapy-induced acute exudative maculopathy (rare) 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Treatment Approach for Central Serous Retinopathy

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Photodynamic Therapy for Chronic Central Serous Chorioretinopathy

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Interventions for central serous chorioretinopathy: a network meta-analysis.

The Cochrane database of systematic reviews, 2025

Research

Central serous chorioretinopathy.

Optometry (St. Louis, Mo.), 2008

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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