What is Vitamin K2?

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What is Vitamin K2?

Vitamin K2, also known as menaquinone (MK), is a fat-soluble vitamin that is endogenously synthesized by intestinal bacteria and exists in several subtypes (MK-2 to MK-14) that differ in side chain length, with menaquinone-7 (MK-7) being particularly bioavailable and clinically relevant. 1, 2

Forms and Sources

Vitamin K exists in multiple forms with distinct characteristics:

  • Vitamin K1 (phylloquinone) is the predominant dietary form found in plants, green leafy vegetables, cruciferous vegetables, asparagus, prunes, peas, and parsley 3, 1
  • Vitamin K2 (menaquinone) is produced by intestinal bacteria and includes various subtypes (MK-2 through MK-14) that differ in their side chain length 1, 2
  • Vitamin K2-7 (MK-7) is absorbed more readily than K1 and demonstrates superior bioavailability 2

Biological Functions

Blood Clotting

  • Vitamin K2 serves as an essential cofactor for gamma-carboxylation of vitamin K-dependent clotting factors (II, VII, IX, X), which is critical for normal blood coagulation 4, 5

Bone Health

  • K2 acts as a cofactor to convert undercarboxylated osteocalcin (ucOC) to its carboxylated form (cOC), facilitating calcium deposition in bones 2
  • It upregulates osteoprotegerin, which acts as a decoy receptor for RANK ligand (RANKL), thereby inhibiting bone resorption 2
  • K2 promotes the transition of osteoblasts to osteocytes and limits osteoclastogenesis 1
  • Vitamin K2 has been approved for osteoporosis treatment in Japan since 1995, with evidence showing reduced fracture incidence and maintained bone mineral density 6

Cardiovascular Health

  • K2 converts undercarboxylated matrix Gla protein (ucMGP) to its carboxylated form (cMGP), preventing vascular calcification 2

Additional Roles

  • Anti-inflammatory effects through suppression of proinflammatory mediators (IL-1α, IL-1β, TNF-α) and NF-κB signal transduction 3, 2
  • Transcriptional regulation through the steroid and xenobiotic receptor (SXR), inducing expression of bone markers including alkaline phosphatase, osteoprotegerin, osteopontin, and matrix Gla protein 5
  • Potential benefits in diabetes, peripheral neuropathy, Alzheimer's disease, and cancer through various signal transduction pathways (PI3K/AKT, MAP Kinase, JAK/STAT) 2

Clinical Considerations

Deficiency

  • Common causes include fat malabsorption syndromes (celiac disease, cystic fibrosis, short bowel), malnutrition, prolonged antibiotic use, anticoagulant therapy (warfarin), and chronic cholestasis 3, 4
  • Deficiency contributes to bleeding disorders, poor bone development, osteoporosis, and increased cardiovascular disease risk 3
  • 8-31% of normal healthy adults have vitamin K deficiency based on undercarboxylated protein analysis 3

Toxicity

  • Vitamin K1 and K2 are not associated with toxicity, with no known adverse effects from supplementation 3
  • Rare anaphylactoid reactions (bronchospasm, cardiac arrest) have been reported only with IV vitamin K1 administration 3

Dosing

  • Current adequate intake recommendations are based on K1 only due to lack of data for K2: 1 mg/kg body weight per day (EFSA) or 120 mg for males and 90 mg for females (IOM) 3
  • For K2-7 supplementation, the usual adult dose is 4 capsules once daily as prescribed 7
  • In cystic fibrosis, vitamin K1 supplementation ranges from 0.3-1 mg/day for infants to 1-10 mg/day for older children and adults 3

Important Caveat

  • Patients on vitamin K antagonists (warfarin) must maintain stable vitamin K intake and require blood clotting monitoring, as sudden changes can affect anticoagulation control 3, 4

References

Research

Vitamin K and osteoporosis: Myth or reality?

Metabolism: clinical and experimental, 2017

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Distinguishing Vitamin K Deficiency from Liver Dysfunction

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

[Vitamin K2].

Clinical calcium, 2008

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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