What is leuprolide acetate and what are its side effects?

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Leuprolide Acetate: Mechanism and Side Effects

What is Leuprolide Acetate?

Leuprolide acetate is a synthetic gonadotropin-releasing hormone (GnRH) agonist that suppresses sex hormone production by initially stimulating, then desensitizing the pituitary gland, leading to medical castration in adults and pubertal suppression in children. 1

Mechanism of Action

  • Leuprolide acts as a potent GnRH receptor agonist that initially causes a transient surge in luteinizing hormone (LH) and follicle-stimulating hormone (FSH), followed by receptor desensitization and down-regulation with continuous use 1, 2

  • Within 2-4 weeks of continuous administration, testosterone levels in males drop to castrate levels (below 50 ng/dL), and estrogen levels in premenopausal females fall to postmenopausal ranges 1, 2

  • The drug is administered as subcutaneous or intramuscular injections because it is not orally bioavailable (it's a peptide that would be degraded in the GI tract) 1

  • Common formulations include daily subcutaneous injections (5 mg/mL) or depot preparations given monthly (3.75 mg or 7.5 mg) or every 3-6 months 3, 2

Clinical Applications

  • Primary indication: Advanced prostate cancer - used as androgen deprivation therapy (ADT), either alone or in combination with antiandrogens like bicalutamide or flutamide 3

  • Endometriosis treatment - monthly depot injections of 3.75 mg show comparable efficacy to danazol 800 mg/day or buserelin 900 mcg/day, but use is limited to 6 months due to bone density loss 2

  • Central precocious puberty - doses of 3.75-15 mg monthly suppress sexual maturation and improve predicted adult height 2, 4, 5, 6

  • Fertility preservation - used as gonadotropin-releasing hormone agonist co-therapy with cyclophosphamide to prevent ovarian insufficiency in women with rheumatic diseases 3

Side Effects

Cardiovascular Effects

  • Atrial fibrillation occurs in approximately 0.73% of patients treated with leuprolide 3

  • Leuprolide combined with newer agents (abiraterone, enzalutamide) may increase cardiovascular risk, though leuprolide alone shows fewer cardiovascular effects than older hormonal therapies like diethylstilbestrol 3, 2

  • In patients with prostate cancer receiving GnRH analogues (including leuprolide), atrial fibrillation was reported in 16% of cases 3

Hormonal and Metabolic Effects

  • Tumor flare phenomenon: During the first 1-2 weeks of treatment, the initial surge in testosterone can temporarily worsen prostate cancer symptoms (bone pain, urinary obstruction, spinal cord compression) - this is why short-term antiandrogen coverage is planned for approximately 93% of patients starting leuprolide 3

  • Hot flashes and vasomotor symptoms are nearly universal in adults due to sex hormone suppression 2

  • Bone mineral density loss occurs with prolonged use, which is why endometriosis treatment is limited to 6 months 2

  • Sexual dysfunction and loss of libido are expected consequences of medical castration 2

Reproductive Effects

  • In men: Leuprolide causes infertility during treatment - sperm cryopreservation should be offered before starting therapy, as the drug causes maximum damage to post-meiosis spermatids 3

  • In women: Ovarian suppression and potential for premature ovarian insufficiency, though co-therapy with GnRH agonists may provide some ovarian protection during chemotherapy 3

Pediatric-Specific Effects

  • In children with central precocious puberty, leuprolide decreases growth velocity initially but ultimately improves predicted adult height by slowing premature bone age advancement 2, 4, 6

  • Side effects in children are generally minimal, though long-term safety data beyond 24.9 months of treatment are limited 4

Other Notable Effects

  • Injection site reactions with subcutaneous administration 1

  • The depot formulation contains benzyl alcohol (9 mg/mL) as a preservative 1

  • Minimal drug-drug interactions expected because leuprolide is degraded by peptidases rather than cytochrome P-450 enzymes, and only 43-49% protein-bound 1

Pharmacokinetics Relevant to Side Effects

  • Terminal elimination half-life is approximately 3 hours after IV bolus 1

  • Less than 5% recovered unchanged in urine, with metabolism to smaller inactive peptides 1

  • Steady-state volume of distribution is 27 L in healthy males 1

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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