What is true regarding Somatostatin?

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Last updated: December 3, 2025View editorial policy

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Somatostatin Characteristics

Option D is correct: Somatostatin inhibits gastric motility, along with endocrine, exocrine, pancreatic, and pituitary secretions, and modifies memory formation in the CNS. 1, 2

Analysis of Each Option

Option A: Production by Antral G Cells - INCORRECT

  • Somatostatin is not produced by antral G cells 3
  • G cells produce gastrin, while somatostatin actually inhibits gastrin release from these cells 3
  • Somatostatin is produced by D cells in the gastric antrum and functions as part of the gastrin-acid feedback loop 2

Option B: Half-life of 30 Minutes - INCORRECT

  • Native somatostatin has an extremely short half-life of 1-3 minutes, not 30 minutes 4, 5
  • This very short half-life necessitates continuous intravenous infusion for clinical use 2, 4
  • The synthetic analogue octreotide has a longer half-life of approximately 1.5-2 hours 1, 4

Option C: Increases Portal Blood Flow - INCORRECT

  • Somatostatin decreases splanchnic blood flow, not increases it 1, 4
  • Octreotide reduces splanchnic blood flow in healthy volunteers and hepatic venous pressure in cirrhotic patients 4
  • This property is utilized therapeutically in variceal bleeding management 2

Option D: Inhibits Gastric Motility and Multiple Secretions - CORRECT

  • Somatostatin has broad inhibitory effects on gastrointestinal motility 1, 2
  • It inhibits gastric, biliary, and pancreatic secretions 1
  • It suppresses endocrine secretions including growth hormone, insulin, glucagon, gastrin, and thyroid-stimulating hormone 1, 6, 4
  • It inhibits exocrine pancreatic function (amylase, trypsin, lipase) 4
  • It has effects on CNS function and memory formation 2, 7

Clinical Significance

The comprehensive inhibitory profile of somatostatin explains its therapeutic utility in multiple conditions 2, 5:

  • Neuroendocrine tumors: Controls hormone hypersecretion syndromes 1
  • High-output jejunostomy: Reduces fluid and electrolyte losses 1
  • Variceal bleeding: Decreases splanchnic blood flow 2
  • Acromegaly: Suppresses growth hormone secretion 1, 6

The short half-life limitation has been overcome with long-acting formulations (octreotide LAR, lanreotide) that can be administered every 4 weeks 1.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Somatostatin: physiology and clinical applications.

Bailliere's clinical endocrinology and metabolism, 1994

Guideline

Somatostatin's Role in Gastrin Release Inhibition

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

[Somatostatin, a new hormone? (author's transl)].

Diabete & metabolisme, 1976

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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