What nutritional deficiencies are associated with atrophic gastritis?

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Nutritional Deficiencies in Atrophic Gastritis

Patients with atrophic gastritis, particularly corpus-predominant disease, develop iron and vitamin B-12 deficiency as the primary nutritional deficiencies, with iron deficiency typically manifesting earlier than B-12 deficiency. 1

Primary Micronutrient Deficiencies

Iron Deficiency

  • Iron deficiency occurs in up to 50% of patients with corpus-predominant atrophic gastritis and presents much earlier in the disease course than vitamin B-12 deficiency 1
  • The mechanism is reduced gastric acid secretion, which impairs iron absorption from dietary sources 1
  • In autoimmune gastritis specifically, iron deficiency occurs in approximately 29-33% of patients, significantly higher than control populations 2

Vitamin B-12 Deficiency

  • Vitamin B-12 deficiency develops due to both reduced gastric acid secretion and loss of intrinsic factor production from parietal cell destruction 1
  • Approximately 13% of autoimmune gastritis patients develop frank vitamin B-12 deficiency, compared to only 1.5% in H. pylori-related gastritis 2
  • Autoimmune gastritis patients have an 11.5-fold increased risk of vitamin B-12 deficiency compared to controls 2
  • This deficiency leads to megaloblastic anemia and can cause severe neurological manifestations if untreated 3, 4

Secondary Micronutrient Deficiencies

Vitamin D Deficiency

  • Vitamin D deficiency is the most prevalent micronutrient deficiency in chronic atrophic autoimmune gastritis patients, affecting the majority of patients 5
  • Vitamin D levels correlate directly with vitamin B-12 levels, suggesting shared pathogenic mechanisms 5
  • Vitamin D levels are significantly lower in patients with macronodular ECL cell hyperplasia compared to those with linear or micronodular patterns 5

Other Deficiencies

  • Folic acid deficiency occurs less commonly but has been documented in atrophic gastritis patients 3, 5
  • Vitamin C deficiency has been increasingly recognized in chronic atrophic autoimmune gastritis 3
  • Calcium malabsorption can occur secondary to hypochlorhydria and vitamin D deficiency 3

Clinical Approach to Screening

Mandatory Screening

  • All patients with atrophic gastritis, regardless of etiology, should be evaluated for iron and vitamin B-12 deficiency, especially if corpus-predominant disease is present 1
  • Conversely, patients presenting with unexplained iron or vitamin B-12 deficiency should be evaluated for atrophic gastritis as the underlying cause 1

Risk Stratification by Etiology

  • Autoimmune gastritis carries higher risk for micronutrient deficiencies than H. pylori-related atrophic gastritis 2
  • Autoimmune gastritis patients have a 2.9-fold increased risk of iron deficiency compared to controls 2
  • Age, sex, and H. pylori status do not significantly affect the occurrence of these deficiencies once atrophic gastritis is established 2

Important Clinical Pitfalls

Multiple Deficiencies

  • Multiple vitamin deficiencies commonly coexist in atrophic gastritis patients and can lead to severe hematological, neurological, and skeletal manifestations 3
  • In one study, 52 of 76 patients with nutritional deficiencies had multiple deficiencies rather than isolated ones 5

Timing of Deficiencies

  • Iron deficiency manifests earlier than vitamin B-12 deficiency because the body has substantial hepatic stores of vitamin B-12 that can last years, whereas iron stores deplete more rapidly 1

Rare Presentations

  • Severe vitamin B-12 deficiency can present as hemolytic anemia mimicking thrombotic microangiopathy (pseudothrombotic microangiopathy), which is critical to recognize to avoid unnecessary plasmapheresis 6
  • Combined deficiencies can cause polyneuropathy and severe neurological dysfunction 4

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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