What is the antidote for intra-arterial injection of bupivacaine (local anesthetic) or lidocaine (xylocaine) during a brachial block?

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Antidote for Intra-arterial Bupivacaine or Lidocaine Injection During Brachial Block

The antidote for local anesthetic systemic toxicity (LAST) from intra-arterial injection of bupivacaine or lidocaine during brachial block is 20% intravenous lipid emulsion (ILE), which should be administered immediately upon recognition of toxicity. 1

Immediate Management Algorithm

Recognition of Toxicity

Local anesthetic systemic toxicity can present with:

  • CNS symptoms first: perioral numbness, metallic taste, tinnitus, facial tingling, confusion, agitation, or seizures 1
  • Cardiovascular collapse: profound bradycardia, junctional rhythms, asystole, or refractory hypotension—often occurring suddenly and may be the first manifestation with intra-arterial injection 2, 3, 4
  • Bupivacaine is particularly cardiotoxic and can cause sudden cardiac arrest even at doses below the traditional maximum recommended dose 5, 4

Lipid Emulsion Therapy Protocol

The American Heart Association provides specific dosing recommendations 1:

Initial bolus: 1.5 mL/kg of 20% lipid emulsion over 1 minute (approximately 100 mL for a 70 kg patient) 1

Continuous infusion: 0.25 mL/kg/min for 30-60 minutes 1

Repeat bolus: May repeat the 1.5 mL/kg bolus once or twice for persistent cardiovascular collapse 1

Maximum total dose: 10 mL/kg over the first hour 1

Concurrent Resuscitation Measures

  • Standard ACLS protocols should continue alongside lipid emulsion therapy 1
  • Maintain adequate ventilation and oxygenation 6
  • Control seizures with benzodiazepines or thiopental if they occur 6
  • Avoid or use reduced doses of epinephrine (maximum 1 mcg/kg), as there are complex pharmacodynamic interactions between lipid emulsion and vasopressors 1
  • Continue CPR if cardiac arrest has occurred 1

Critical Clinical Considerations

Why Lipid Emulsion Works

Lipid emulsion creates a lipid compartment in the serum that sequesters lipophilic local anesthetics (especially bupivacaine) away from cardiac tissue, and also increases cardiac inotropy through other mechanisms 1

Evidence Quality

  • All 21 published cases of lipid emulsion use for bupivacaine-induced LAST demonstrated clinical improvement (return of spontaneous circulation, relief of hypotension, or resolution of dysrhythmia) 1
  • Animal studies consistently show benefit of lipid emulsion in bupivacaine toxicity 1
  • The 2023 American Heart Association guidelines give this a Class IIa recommendation (reasonable to use) 1

Bupivacaine-Specific Concerns

Bupivacaine is uniquely dangerous because 7, 5, 6:

  • It causes more severe and refractory cardiac toxicity than other local anesthetics 6
  • Cardiac arrest can occur suddenly without preceding CNS symptoms 5, 4
  • Dysrhythmias may be refractory to standard ACLS medications 6
  • Toxicity has been reported at doses as low as 1.1 mg/kg IV 5
  • The FDA explicitly warns against using bupivacaine for IV regional anesthesia (Bier block) due to reports of cardiac arrest and death 7

Common Pitfalls and How to Avoid Them

Preparation is Essential

  • Lipid emulsion 20% must be immediately available wherever regional anesthesia is performed 1, 8
  • Staff should know the exact location of lipid emulsion storage 1
  • Insufficient quantities may be problematic, as toxicity can recur 40+ minutes after initial lipid rescue, requiring additional doses 3

Recognition Delays

  • Do not wait for confirmatory testing—treat immediately based on clinical presentation 1
  • Intra-arterial injection can cause immediate cardiovascular collapse without the typical progression of CNS symptoms first 2, 4
  • Bradycardia from bupivacaine toxicity is often refractory to atropine 4

Special Populations at Higher Risk

  • Uremic patients are at increased risk of cardiotoxicity even at lower doses 2
  • Patients with preexisting cardiac conduction abnormalities are more susceptible 2
  • Those taking beta-blockers, calcium channel blockers, or digoxin have increased risk 6

Monitoring After Treatment

  • Prolonged observation is mandatory as toxicity can recur after apparent successful resuscitation 3
  • Continue monitoring for at least several hours after lipid emulsion administration 3
  • Have additional lipid emulsion readily available for potential recurrence 3

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Cardiovascular collapse from low dose bupivacaine.

The Canadian journal of clinical pharmacology = Journal canadien de pharmacologie clinique, 2005

Guideline

Intravenous Lidocaine Administration Guidelines

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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