What is the classification and mechanism of action of Colistin (polymyxin antibiotic)?

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Colistin Classification and Mechanism of Action

Colistin (polymyxin E) is a positively charged polypeptide antibiotic that disrupts the outer membrane of gram-negative bacteria by binding to the lipid A moiety of lipopolysaccharides, resulting in cell death. 1

Drug Classification

  • Colistin belongs to the polymyxin class of antibiotics, specifically classified as polymyxin E 1
  • The drug is administered as colistimethate sodium (CMS), an inactive prodrug that is less toxic and preferred for parenteral administration 2, 3
  • Colistimethate sodium is a polypeptide antibiotic with an approximate molecular weight of 1750 and the empirical formula C58H105N16Na5O28S5 3

Mechanism of Action

Primary Bactericidal Effect

  • Colistin is an amphipathic molecule that interacts with lipopolysaccharide (LPS) in the bacterial outer membrane 4
  • The positively charged decapeptide structure binds specifically to the lipid A moiety of LPS, disrupting the integrity of the outer membrane of gram-negative bacteria 1
  • This binding leads to disruption of both the outer and cytoplasmic membranes, resulting in bacterial death and lysis 5

Secondary Antiendotoxic Properties

  • The endotoxic activity of lipopolysaccharides is simultaneously inhibited during colistin's bactericidal action 1
  • Polymyxins have potent antiendotoxic properties that contribute to their therapeutic effect 4

Antibacterial Spectrum

  • Colistin demonstrates potent antibacterial activity against Pseudomonas aeruginosa, Acinetobacter baumannii, Klebsiella pneumoniae, and many Enterobacteriaceae 6, 4
  • The antibiotic has resurfaced as a last-line treatment option for multidrug-resistant gram-negative organisms 6

Important Clinical Considerations

Resistance Mechanism Implications

  • Colistin resistance occurs through LPS modifications that protect the cytoplasmic membrane but not the outer membrane from damage 5
  • Resistance can develop via chromosomal mutations or acquisition of mobilized colistin-resistance (mcr) genes, both resulting in chemical modifications to LPS with positively charged moieties 5, 7
  • Resistance seems to emerge often unless colistin is combined with another antibiotic, though further studies are needed 1

Pharmacodynamic Properties

  • The ratio between the area under the curve (AUC) of free colistin and the pathogen's Minimal Inhibitory Concentration (MIC) best predicts microbiological and clinical responses 1

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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