What is the treatment for a Salter Harris fracture?

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Last updated: December 9, 2025View editorial policy

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Treatment of Salter-Harris Fractures

Salter-Harris fractures require treatment based on fracture type, location, and degree of displacement, with most minimally displaced fractures managed conservatively with casting, while displaced fractures (≥3mm) require closed reduction and potentially surgical intervention if reduction fails or soft-tissue interposition is present.

Initial Management and Imaging

  • Avoid manipulation prior to obtaining radiographs unless neurovascular compromise or critical skin injury is present, as premature manipulation may complicate subsequent management 1
  • Standard three-view radiographs (anteroposterior, lateral, and mortise for ankle fractures) are the initial imaging study of choice 1
  • MRI can help exclude Salter-Harris type I fractures in the pediatric population when radiographs are negative but clinical suspicion remains high 1

Treatment Algorithm by Displacement

Minimally Displaced Fractures (<2-3mm)

  • Cast immobilization is the primary treatment for fractures with displacement less than 3mm 2
  • Non-weight-bearing long-leg cast is appropriate for distal tibia Salter-Harris II fractures with minimal displacement 2, 3
  • Rigid immobilization is preferred over removable splints for displaced fractures requiring nonsurgical treatment 1

Moderately Displaced Fractures (2-4mm)

  • Initial closed reduction with conscious sedation in the emergency department is indicated for displacement ≥3mm 2
  • If closed reduction achieves <2mm residual displacement, proceed with cast immobilization 2, 3
  • Surgical management (open reduction and internal fixation) does NOT reduce the risk of premature physeal closure compared to casting for residual displacement of 2-4mm, but may be necessary for anatomic alignment 4

Severely Displaced Fractures (>4mm)

  • Open reduction and internal fixation is indicated when closed reduction fails to achieve adequate alignment or when initial displacement exceeds 4mm 2, 4
  • Surgical exploration should remove any interposed soft tissue, though this does not eliminate the risk of premature physeal closure 4

Special Considerations by Location

Distal Tibia Salter-Harris II Fractures

  • Most (88%) are minimally displaced and do not require reduction 2
  • High-grade injury mechanisms and initial displacement ≥4mm increase the odds of premature physeal closure by 12-14 fold, regardless of treatment method 3
  • Overall complication rate is 2% for minimally displaced fractures but increases to 17% for displaced fractures 2
  • Premature physeal closure occurs in 24-55% of cases depending on initial displacement, with higher rates in more severely displaced fractures 3, 4

Distal Phalanx Salter-Harris Fractures

  • Any clinical finding suggestive of nail-bed laceration mandates surgical exploration, including subungual hematoma, nail plate subluxation, or eponychial fold laceration 5
  • When explored, 82% have confirmed nail-bed laceration and 47% have soft-tissue interposition requiring removal 5
  • Treatment consists of open reduction with nail-bed repair and either splinting or percutaneous pinning 5

Critical Pitfalls and Caveats

  • Surgical fixation does not reduce premature physeal closure rates in Salter-Harris II distal tibia fractures and may actually increase the need for subsequent procedures (epiphysiodesis, osteotomy) 4
  • The overall premature physeal closure rate for displaced distal tibia Salter-Harris II fractures is 43%, regardless of treatment method 4
  • Patient age, sex, mechanism of injury, time to management, and number of reduction attempts do not significantly influence premature physeal closure risk 4
  • Initial fracture displacement and high-energy mechanisms are the primary predictors of complications, not the treatment method chosen 3, 4
  • Follow-up radiographs should be performed for a minimum of 6 months to assess for premature physeal closure, with CT imaging if clinical concern develops 4

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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