Can adrenaline (epinephrine) cause leukocytosis?

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Can Adrenaline Cause Leukocytosis?

Yes, adrenaline (epinephrine) definitively causes leukocytosis through rapid mobilization of white blood cells from the marginal pool, spleen, bone marrow, and lymphatics, with effects occurring within 5 minutes of administration and typically resolving within 25 minutes. 1

Mechanism and Time Course

Adrenaline-induced leukocytosis occurs through a biphasic response:

  • Phase 1 (Immediate, <30 minutes): Rapid lymphocyte mobilization, predominantly affecting natural killer (NK) cells, with total blood leucocytes increasing to approximately 220% of baseline within 5 minutes 1, 2

  • Phase 2 (Later): Granulocyte increases with relative lymphopenia, as neutrophilic granulocytes rise to about 160% of baseline 1, 3

The response is transient, with cell concentrations returning to baseline within 25 minutes after adrenaline injection 1

Specific Cell Populations Affected

The most responsive leukocyte subsets include 4:

  • CD16+CD56dim cytotoxic NK cells (most prominent increase)
  • CCR7-CD45RA+CD8+ effector T cells
  • CD4-CD8- gamma/delta T cells
  • CD3+CD56+ NKT-like cells
  • CD14dimCD16+ proinflammatory monocytes

Both T and B lymphocytes are mobilized to approximately 230-250% of baseline concentrations 1

Sources of Mobilized Cells

Multiple anatomical compartments contribute substantially to this leukocytosis 1:

  • Marginal pool: Primary source for both lymphocytes and granulocytes 2, 5
  • Spleen: Significant contributor (splenectomized rats show markedly reduced response) 1
  • Bone marrow: Substantial contribution evidenced by increased band-nucleated granulocytes 1
  • Lymphatics: Thoracic duct drainage significantly reduces the response 1
  • Lung: 20-25% of neutrophils are retained in first pass; exercise and catecholamines accelerate their release 5

Receptor Mechanisms

The leukocytosis involves distinct adrenergic pathways 2:

  • Beta-2 adrenoceptors: Mediate lymphocyte mobilization (particularly NK cells)
  • Alpha-adrenoceptors: Mediate granulocyte increases
  • CD11a/CX3CR1: These adhesion molecules correlate strongly with adrenergic leukocytosis (r = 0.86 and 0.78, p < 0.005) 4

Propranolol (beta-blocker) blocks the polymorphonuclear leucocytosis response 3

Clinical Relevance

Important caveat: This leukocytosis is a normal physiological response and should not be confused with pathological leukocytosis 1, 2. The phenomenon represents mobilization of cytotoxic effector cells that provide immediate protection from invading pathogens as part of the fight/flight response 2, 4

The response is not blood-flow dependent, as organ perfusion remains unaltered after adrenaline injection despite significant cell mobilization 1

Repeated Exposure Effects

Repeated daily administration of exogenous adrenaline increases basal total leucocyte and polymorph counts while decreasing lymphocyte counts, suggesting sensitization of the system responsible for polymorph release from bone marrow 3

References

Research

Adrenaline-induced leucocytosis: recruitment of blood cells from rat spleen, bone marrow and lymphatics.

European journal of applied physiology and occupational physiology, 1994

Research

Effect of electroconvulsions on leucocyte counts.

Indian journal of experimental biology, 1990

Research

Selective mobilization of cytotoxic leukocytes by epinephrine.

Journal of immunology (Baltimore, Md. : 1950), 2010

Research

Leukocyte kinetics in the human lung: role of exercise and catecholamines.

Journal of applied physiology: respiratory, environmental and exercise physiology, 1984

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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