What causes hypertension?

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Last updated: December 24, 2025View editorial policy

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Causes of High Blood Pressure

Hypertension results from two main categories: primary (essential) hypertension accounting for approximately 90% of cases with multifactorial origins, and secondary hypertension representing 10% with identifiable underlying causes. 1, 2

Primary (Essential) Hypertension

Primary hypertension develops through complex interactions between genetic predisposition, environmental factors, and behavioral influences, though the exact cause remains unknown. 1, 2

Modifiable Lifestyle Factors

Obesity is the single most important modifiable cause, responsible for 40% of all hypertension cases and up to 78% in men and 65% in women. 1, 2 The relationship between body mass index and blood pressure is continuous and almost linear with no threshold. 1, 2

Dietary sodium excess is positively associated with blood pressure and accounts for much of the age-related increase in blood pressure. 1, 2 Excessive sodium consumption is independently associated with increased risk of stroke and cardiovascular disease beyond its blood pressure effects. 1, 2

Additional dietary factors include insufficient intake of potassium, calcium, magnesium, protein, fiber, and fish fats. 1, 2

Excessive alcohol intake (≥3 standard drinks per day) shows a strong, direct relationship with blood pressure elevation. 2 Current guidelines recommend men limit consumption to no more than 2 standard drinks per day and women to 1 standard drink per day. 3

Physical inactivity and prolonged sedentary lifestyles combined with caloric intake exceeding energy expenditure lead to steep rises in blood pressure, particularly systolic, with advancing age. 4

Genetic Factors

Genetic variants collectively account for only about 3.5% of blood pressure variability, despite hypertension being a complex polygenic disorder. 1, 2 Family history of hypertension is a characteristic feature suggesting primary hypertension. 3, 1

Pathophysiological Mechanisms

  • Overactivation of the renin-angiotensin-aldosterone system (RAAS) contributes to development and maintenance of hypertension 2
  • Endothelial dysfunction impairs normal vascular regulation 2
  • Dysregulation of renal sodium handling and pressure natriuresis plays a fundamental role 2
  • Increased peripheral vascular resistance, vascular remodeling, and fibrosis with inflammation perpetuate elevated blood pressure 2

Secondary Hypertension

Secondary hypertension accounts for approximately 10% of cases overall, but increases to 10-35% in resistant hypertension populations and up to 20-40% in patients presenting with malignant hypertension. 2, 4

Renal Causes

Chronic kidney disease and renal parenchymal disease are the most common secondary causes of hypertension. 1, 2 The most common parenchymal kidney diseases include chronic glomerulonephritis, polycystic kidney disease, and hypertensive nephrosclerosis. 2

Renal artery stenosis should be suspected with onset of hypertension before age 30 or after age 55, presence of abdominal bruit, accelerated hypertension, and recurrent flash pulmonary edema. 2

Endocrine Causes

Primary aldosteronism presents with unprovoked hypokalemia, muscle cramps, and weakness. 3, 1, 2

Pheochromocytoma causes blood pressure lability, episodic pallor, dizziness, headache, palpitations, and perspiration. 3, 1, 2

Cushing's syndrome presents with central obesity, facial rounding, easy bruisability, truncal obesity, glucose intolerance, and purple striae. 3, 1, 2

Hyperthyroidism causes weight loss, palpitations, and heat intolerance. 3, 1, 2

Obstructive Sleep Apnea

Obstructive sleep apnea presents with snoring and hypersomnolence and requires continuous positive airway pressure therapy to improve blood pressure control. 1, 2

Drug-Induced Hypertension

Medications and substances that elevate blood pressure include: 3, 1, 2

  • Nonsteroidal anti-inflammatory drugs (NSAIDs)
  • Cocaine
  • Amphetamines
  • Corticosteroids
  • Calcineurin inhibitors
  • Alcohol

Vascular Causes

Aortic coarctation is characterized by decreased pressure in lower extremities or delayed/absent femoral arterial pulses. 2

Clinical Approach to Identifying the Cause

Features Suggesting Primary Hypertension

  • Gradual increase in blood pressure with slow rate of rise 3, 1, 2
  • Lifestyle factors favoring higher blood pressure (weight gain, high-sodium diet, decreased physical activity, excessive alcohol consumption) 3, 1
  • Strong family history of hypertension 3, 1

Features Suggesting Secondary Hypertension

  • Absence of family history of hypertension 3, 1
  • Sudden onset or worsening of hypertension 3, 1
  • Young age (any hypertension <20 years; needing treatment <30 years) 3
  • Resistant to multidrug regimen (≥3 drugs) 3
  • Elevated serum creatinine 3
  • Proteinuria or hematuria 3
  • Hypokalaemia with increased or high normal plasma sodium 3

Common pitfall: Poor compliance or adherence to drug treatment or lifestyle changes is the most common cause of resistant hypertension and should be evaluated before extensive workup for secondary causes. 4

References

Guideline

Causes of Hypertension

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Hypertension Causes and Mechanisms

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Hypertension Management and Complications

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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