What is myelodysplastic syndrome (MDS)?

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What is Myelodysplastic Syndrome (MDS)?

Myelodysplastic syndromes are a heterogeneous group of clonal hematopoietic stem cell disorders characterized by ineffective hematopoiesis leading to peripheral blood cytopenias despite a hypercellular bone marrow, with dysplasia of cellular elements and a risk of transformation to acute myeloid leukemia in approximately one-third of patients. 1

Core Pathophysiology

MDS represents a malignant clonal disorder originating from hematopoietic stem cells, where the bone marrow paradoxically appears hypercellular yet fails to produce adequate functional blood cells—a phenomenon termed "ineffective hematopoiesis." 1 This results in:

  • Peripheral blood cytopenias (anemia, neutropenia, and/or thrombocytopenia) despite increased marrow cellularity 1
  • Morphologic dysplasia affecting one or more myeloid lineages, including megaloblastoid erythropoiesis, nucleocytoplasmic asynchrony in early precursors, and dysmorphic megakaryocytes 1
  • Increased apoptosis of developing blood cells within the marrow, preventing their maturation and release 1

The disease develops through a multistep genetic process involving mutations affecting DNA damage responses, transcription, RNA splicing, epigenetics, and cytokine signaling. 1

Epidemiology and Risk Factors

MDS predominantly affects elderly individuals with a median age at diagnosis of approximately 70 years, though more than 10% of patients are younger than 50 years. 1, 2 The incidence is:

  • 4 per 100,000 in the general U.S. population 1, 2
  • 22-45 per 100,000 among individuals older than 70 years 1
  • Higher in males (5.4 per 100,000) compared to females (2.9 per 100,000) 2

Known etiologic factors account for only 15-20% of cases: 1

  • Prior chemotherapy exposure (especially alkylating agents, purine analogues) 1
  • Radiation therapy or ionizing radiation 1
  • Benzene and its derivatives 1
  • Tobacco smoking 1
  • Inherited predisposition (more common in pediatric cases, including Down syndrome, Fanconi anemia) 1

The etiology remains unknown in more than 80% of patients. 1

Clinical Presentation

The major clinical problems stem from cytopenias and leukemic transformation risk: 1

  • Anemia symptoms: fatigue, weakness, dyspnea
  • Thrombocytopenia: bleeding, bruising, petechiae
  • Neutropenia: recurrent infections
  • Insidious onset with symptoms developing over months 1, 2

The disease typically presents with relatively stable blood counts over several months, distinguishing it from acute processes. 1

Leukemic Transformation Risk

Approximately 30-40% of MDS patients will progress to acute myeloid leukemia, with transformation rates varying by subtype: 1

  • Lower-risk subtypes (refractory anemia, refractory anemia with ring sideroblasts): 5-15% transformation rate 1
  • Higher-risk subtypes (refractory anemia with excess blasts): 40-50% transformation rate 1

When MDS transforms to AML, these patients experience lower response rates to standard therapy compared to de novo AML. 1

Prognosis and Heterogeneity

MDS demonstrates marked heterogeneity in clinical outcomes even within morphologic subtypes. 1 Survival varies dramatically:

  • Lower-risk disease: median survival 3-10 years 2, 1
  • Higher-risk disease: median survival less than 3 years, often 5-12 months 2, 1
  • Overall 5-year survival: approximately 37% 2

The International Prognostic Scoring System (IPSS) and its revision (IPSS-R) stratify patients based on bone marrow blast percentage, cytogenetic abnormalities, and number/severity of cytopenias. 1 These scoring systems are critical for risk-based treatment selection and clinical trial design. 1

Historical Context and Nomenclature

MDS has been historically referred to by various names including oligoblastic leukemia, refractory anemia, smoldering acute leukemia, and preleukemia. 1 The French-American-British (FAB) classification system, established in 1982 and modified in 1985, remains widely used despite WHO modifications. 1 The WHO 2016 classification incorporates molecular markers (such as SF3B1 mutations) and lowered the blast threshold for AML diagnosis from 30% to 20%. 1

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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