What is the function of dihydrofolate reductase (DHFR) in the body?

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Dihydrofolate Reductase Function

Primary Enzymatic Function

Dihydrofolate reductase (DHFR) catalyzes the NADPH-dependent reduction of dihydrofolate to tetrahydrofolate (THF), which is essential for DNA synthesis, repair, and cellular replication. 1, 2

Critical Metabolic Roles

DNA Synthesis and Cell Proliferation

  • DHFR regenerates tetrahydrofolate by reducing dihydrofolate using NADPH as a cofactor, making it essential for maintaining the cellular folate pool 2, 3
  • Tetrahydrofolate and its one-carbon adducts are required for de novo synthesis of purines and thymidylate, which are fundamental building blocks of DNA 2, 3
  • The enzyme enables synthesis of glycine, methionine, and serine through one-carbon metabolism pathways 2
  • Dihydrofolates must be reduced to tetrahydrofolates before they can function as carriers of one-carbon groups in nucleotide synthesis 1

Folate Metabolism Integration

  • DHFR is expressed primarily in the liver and converts synthetic folic acid (pteroylglutamic acid) into biologically active tetrahydrofolate, though this enzymatic activity is relatively limited 4
  • The enzyme works in concert with other folate-dependent enzymes, particularly thymidylate synthase, which uses 5,10-methylenetetrahydrofolate to convert uracil to thymine 4
  • DHFR activity is essential for maintaining adequate intracellular concentrations of 5,10-MTHF, preventing uracil misincorporation into DNA 4

Clinical Significance

Therapeutic Target

  • DHFR inhibition depletes tetrahydrofolate, thereby slowing DNA synthesis and cell proliferation, making it an effective target for cancer chemotherapy 1, 5
  • Methotrexate inhibits DHFR, interfering with DNA synthesis, repair, and cellular replication in rapidly proliferating tissues such as malignant cells, bone marrow, and intestinal mucosa 1
  • The enzyme has been studied extensively as a drug target for bacterial, protozoal, and fungal infections, as well as neoplastic and autoimmune diseases 3

Genetic Variations

  • Polymorphisms in the DHFR gene affect mRNA levels and enzyme expression, influencing folate status, disease susceptibility (including spina bifida and cancer), and response to antifolate medications like methotrexate 5
  • Changes in DHFR expression levels can affect susceptibility to folate-dependent diseases and sensitivity to anti-cancer drugs 5

Secondary DHFR Enzyme

  • Humans possess a second dihydrofolate reductase enzyme (DHFRL1) located in mitochondria, which has lower specific activity and higher Km for dihydrofolate compared to cytoplasmic DHFR 6
  • DHFRL1 can complement DHFR-negative phenotypes but appears optimized for mitochondrial folate metabolism with its lowered substrate affinity 6

Pathophysiological Consequences of Deficiency

  • Limited DHFR activity leads to accumulation of uracil, imbalanced deoxyuridine monophosphate:deoxythymidine monophosphate ratios, and misincorporation of uracil into cellular DNA 4
  • Inadequate DHFR function results in increased DNA deletions, chromosomal instability, and impaired DNA integrity 4
  • DHFR inhibition causes disruption of purine and thymidylate biosynthesis and DNA replication, leading to cell death 2

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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