What are the definitions, diagnostic criteria, risk factors, and management options for preterm labor, preterm prelabor rupture of membrane (PPROM), placenta previa, placental abruptio, preeclampsia/eclampsia, and HELLP (Hemolysis, Elevated Liver enzymes, and Low Platelet count) syndrome in high-risk pregnant women with potential previous pregnancy complications, hypertension, or underlying medical conditions?

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High-Risk Obstetric Emergencies: A Clinical Guide for PA Students

Preterm Labor

Definition and Diagnosis

Preterm labor is defined as regular uterine contractions with cervical change occurring before 37 weeks of gestation. 1

  • Diagnostic criteria include:
    • Regular contractions (≥4 in 20 minutes or ≥8 in 60 minutes) 2
    • Progressive cervical dilation ≥2 cm or effacement ≥80% 2
    • Gestational age between 20 0/7 and 36 6/7 weeks 1

Risk Factors

  • Maternal factors: Previous preterm birth, multiple gestations, short cervical length (<25mm), uterine anomalies 2
  • Pregnancy complications: Infection (chorioamnionitis), placental abruption, PPROM 2
  • Medical conditions: Hypertension, diabetes, systemic lupus erythematosus 1
  • Behavioral factors: Smoking >10 cigarettes/day, substance abuse 1

Management Algorithm

For threatened preterm labor <34 weeks with intact membranes:

  • Tocolysis: Use to delay delivery 48 hours for corticosteroid administration 2
  • Corticosteroids: Betamethasone 12mg IM q24h x2 doses or dexamethasone 6mg IM q12h x4 doses for fetal lung maturity if delivery anticipated <35 weeks 3
  • Magnesium sulfate: Administer for neuroprotection if delivery expected <32 weeks 3
  • Antibiotics: Only indicated if PPROM present (see below) 1

For preterm labor ≥34 weeks:

  • Proceed with delivery as neonatal outcomes are favorable 2

Preterm Prelabor Rupture of Membranes (PPROM)

Definition and Incidence

PPROM is membrane rupture before labor onset occurring before 37 weeks of gestation, complicating <1% of pregnancies but causing one-third of all preterm births. 1, 4

Diagnostic Approach

Diagnosis requires sterile speculum examination—never perform digital cervical exam until labor is established to reduce infection risk. 4, 5

  • Pooling test: Visualize amniotic fluid pooling in posterior vaginal fornix 5
  • Nitrazine test: pH >6.5 turns paper blue (90% sensitivity) 4, 5
  • Ferning test: Microscopic crystallization pattern on dried fluid 4, 5
  • Combination accuracy: Two positive tests = 93.1% diagnostic accuracy 4
  • Modern tests: AmniSure or Actim tests for equivocal cases 5

Gestational Age-Based Management

For previable/periviable PPROM (20 0/7 to 23 6/7 weeks):

  • All patients must receive individualized counseling about maternal risks (sepsis, hemorrhage) and fetal outcomes (5-6% survival <23 weeks with 98-100% severe morbidity among survivors). 1
  • Both abortion care and expectant management should be offered; abortion care must be available to all patients. 1
  • Antibiotics can be considered but are not mandatory at this gestational age 1

For PPROM 24 0/7 to 31 6/7 weeks:

  • Expectant management with hospitalization is standard. 2
  • Antibiotics are mandatory: 7-day course reduces neonatal morbidity 1, 2
    • Ampicillin 2g IV q6h + erythromycin 250mg IV q6h x48h, then amoxicillin 250mg PO q8h + erythromycin base 333mg PO q8h x5 days 2
  • Corticosteroids: Administer for fetal lung maturity 1, 3
  • Magnesium sulfate: Give for neuroprotection if delivery imminent 1, 3
  • Monitor for: Chorioamnionitis (fever, maternal/fetal tachycardia, uterine tenderness, purulent discharge), placental abruption, cord compression 2

For PPROM 32 0/7 to 33 6/7 weeks:

  • Consider delivery versus expectant management based on lung maturity assessment 2

For PPROM ≥34 weeks:

  • Proceed with delivery as benefits clearly outweigh risks. 2

Contraindications to Expectant Management

Immediate delivery indicated for:

  • Clinical chorioamnionitis 2
  • Non-reassuring fetal status 2
  • Placental abruption 2
  • Advanced labor 2

Placenta Previa

Definition and Classification

Placenta previa is placental implantation that covers or lies within 2cm of the internal cervical os.

  • Complete previa: Placenta completely covers internal os
  • Marginal previa: Placental edge within 2cm of os but doesn't cover it
  • Low-lying placenta: Placental edge within 2cm of os on transvaginal ultrasound

Risk Factors

  • Previous cesarean delivery (most significant risk factor)
  • Advanced maternal age (>35 years) 1
  • Multiparity (≥3 previous births) 1
  • Multiple gestation 1
  • Previous uterine surgery or curettage
  • Smoking 1

Clinical Presentation

Classic presentation is painless vaginal bleeding in the second or third trimester.

  • Bleeding typically occurs after 20 weeks
  • May be provoked by intercourse or contractions
  • Never perform digital cervical examination if previa suspected—can precipitate massive hemorrhage

Diagnostic Approach

  • Transvaginal ultrasound is gold standard and safe: Provides accurate placental localization
  • Transabdominal ultrasound for initial screening
  • MRI reserved for suspected placenta accreta spectrum

Management Strategy

For asymptomatic previa diagnosed <28 weeks:

  • Pelvic rest (no intercourse, no vaginal exams)
  • Activity modification
  • Repeat ultrasound at 28 and 32 weeks (many resolve with lower uterine segment development)

For bleeding episodes:

  • Hospitalize for active bleeding
  • Type and crossmatch blood
  • Large-bore IV access
  • Continuous fetal monitoring
  • Corticosteroids if <34 weeks 3
  • Consider hospitalization until delivery for recurrent bleeding

Delivery planning:

  • Scheduled cesarean delivery at 36-37 weeks for persistent previa
  • Earlier delivery for uncontrolled bleeding or non-reassuring fetal status
  • Prepare for potential massive hemorrhage and placenta accreta spectrum
  • Multidisciplinary team including anesthesia, blood bank, possible gynecologic oncology

Placental Abruption

Definition and Severity

Placental abruption is premature separation of a normally implanted placenta from the uterine wall before delivery, occurring in 0.5-1% of pregnancies.

Risk Factors

  • Hypertensive disorders: Most significant modifiable risk factor 1
  • Previous abruption (10-25% recurrence risk)
  • Maternal trauma (motor vehicle accident, domestic violence)
  • Cocaine or methamphetamine use
  • Cigarette smoking 1
  • Advanced maternal age 1
  • Multiparity 1
  • Preterm PPROM 1
  • Thrombophilias 1

Clinical Presentation

Classic triad (only present in 50% of cases):

  • Vaginal bleeding (80% of cases—may be concealed in 20%)
  • Abdominal/back pain with uterine tenderness
  • Uterine contractions/hypertonus

Additional findings:

  • Non-reassuring fetal heart rate patterns
  • Maternal hemodynamic instability (tachycardia, hypotension)
  • Coagulopathy in severe cases (DIC develops in 10%)

Diagnostic Approach

Diagnosis is primarily clinical—ultrasound has poor sensitivity (25-50%) and should not delay management.

  • Continuous fetal monitoring mandatory
  • Maternal labs: CBC, type and crossmatch, coagulation studies (PT, aPTT, fibrinogen, D-dimer), Kleihauer-Betke if Rh-negative
  • Ultrasound to assess fetal viability and rule out previa

Management Based on Severity

Mild abruption (no maternal/fetal compromise, <1/3 placental separation):

  • Hospitalization with continuous monitoring
  • Corticosteroids if <34 weeks 3
  • Expectant management may be considered if stable

Moderate to severe abruption:

  • Immediate delivery is indicated regardless of gestational age 1
  • Aggressive fluid resuscitation with crystalloid
  • Transfuse PRBCs to maintain hemoglobin >7-8 g/dL
  • Correct coagulopathy: FFP for INR >1.5, cryoprecipitate for fibrinogen <200 mg/dL, platelets for count <50,000/mm³ 3
  • Vaginal delivery acceptable if maternal/fetal status stable and labor progressing
  • Cesarean delivery for non-reassuring fetal status, failed induction, or maternal instability

Fetal demise with abruption:

  • Vaginal delivery preferred unless maternal indication for cesarean
  • Monitor closely for DIC development

Preeclampsia/Eclampsia

Definitions

Preeclampsia is new-onset hypertension (≥140/90 mmHg) after 20 weeks gestation with proteinuria or end-organ dysfunction. 3, 6

Severe features include:

  • Blood pressure ≥160/110 mmHg 3
  • Thrombocytopenia (<100,000/mm³) 3
  • Elevated liver enzymes (AST/ALT >2x normal) 3
  • Renal insufficiency (creatinine >1.1 mg/dL or doubling of baseline) 3
  • Pulmonary edema 3
  • New-onset cerebral or visual symptoms 3

Eclampsia is new-onset grand mal seizures in a woman with preeclampsia that cannot be attributed to other causes. 3

Atypical Presentations to Recognize

Preeclampsia can occur without classic hypertension and proteinuria—15% of HELLP cases have normal blood pressure. 7, 6

  • Before 20 weeks: Consider molar pregnancy, antiphospholipid syndrome 6
  • Beyond 48 hours postpartum: Late postpartum preeclampsia/eclampsia occurs in up to 25% of cases 6
  • Isolated symptoms: Severe headache, visual changes, or epigastric pain may precede hypertension 6

Risk Factors

  • Nulliparity or new paternity
  • Previous preeclampsia (15-25% recurrence)
  • Chronic hypertension 1
  • Pregestational diabetes
  • Renal disease
  • Autoimmune disease (SLE, antiphospholipid syndrome) 1
  • Multiple gestation 1
  • Obesity (BMI >30) 1
  • Maternal age >35 or <20 years 1
  • Family history of preeclampsia

Diagnostic Workup

Essential laboratory assessment includes: 3, 8

  • Complete blood count with platelet count
  • Comprehensive metabolic panel (creatinine, liver enzymes)
  • Urinalysis with protein quantification (24-hour urine or protein/creatinine ratio)
  • Coagulation studies if platelets <100,000/mm³
  • LDH and peripheral smear if hemolysis suspected

Management of Preeclampsia Without Severe Features

Antepartum management:

  • Outpatient management acceptable with close surveillance
  • Blood pressure checks twice weekly
  • Weekly labs (CBC, liver enzymes, creatinine)
  • Fetal surveillance (NST, AFI) twice weekly starting at diagnosis
  • Patient education on warning symptoms

Antihypertensive therapy:

  • Initiate oral therapy for blood pressure 140-159/90-109 mmHg using labetalol, nifedipine, or methyldopa 3
  • Target blood pressure 130-150/80-100 mmHg

Delivery timing:

  • Deliver at 37 weeks for preeclampsia without severe features 3

Management of Preeclampsia With Severe Features

Immediate interventions:

  • Hospitalization mandatory 3
  • Severe hypertension (≥160/110 mmHg) requires urgent treatment within 30-60 minutes 3
    • IV labetalol: 20mg bolus, then 40mg at 10 min, then 80mg q10min x2 (max 220mg) 3
    • IV hydralazine: 5mg bolus, then 10mg q20-30min (max 25mg) 3
    • Oral nifedipine immediate-release: 10mg, repeat in 20 min if needed 3

Seizure prophylaxis:

  • Magnesium sulfate mandatory for all severe preeclampsia and during labor/24 hours postpartum 3
  • Loading dose: 4-6g IV over 15-20 minutes
  • Maintenance: 2g/hour continuous infusion 3
  • Monitor: Deep tendon reflexes, respiratory rate >12/min, urine output >25 mL/hour
  • Antidote: Calcium gluconate 1g IV for magnesium toxicity

Delivery timing:

  • Deliver at 34 weeks for severe preeclampsia 3
  • Immediate delivery indicated for: 3
    • Eclampsia
    • Pulmonary edema
    • Placental abruption
    • Non-reassuring fetal status
    • Persistent severe symptoms despite treatment

Expectant management <34 weeks (highly selective):

  • Only if maternal/fetal status stable
  • Corticosteroids for fetal lung maturity 3
  • Continuous inpatient monitoring
  • Daily labs and fetal surveillance

Management of Eclampsia

Acute seizure management:

  • Magnesium sulfate is first-line: 4-6g IV bolus over 5 minutes, then 2g/hour maintenance 3
  • Protect airway, supplemental oxygen
  • Left lateral positioning
  • If seizures persist: Additional 2g magnesium bolus or consider lorazepam 2-4mg IV
  • Deliver after maternal stabilization regardless of gestational age 3

Postpartum Management

  • Continue magnesium sulfate 24 hours postpartum 3
  • Monitor blood pressure q4h for 72 hours, then daily until normalized
  • Antihypertensive therapy as needed (avoid methyldopa postpartum—use labetalol, nifedipine, or enalapril)
  • Discharge education on warning signs (headache, visual changes, right upper quadrant pain)
  • Follow-up within 7-10 days postpartum

Prevention in High-Risk Women

Low-dose aspirin (81-150mg daily) reduces preeclampsia risk by 15-20% when started before 16 weeks and continued until 36 weeks or delivery. 3, 8

Indications for aspirin prophylaxis:

  • Previous preeclampsia, especially with adverse outcome
  • Multifetal gestation
  • Chronic hypertension
  • Type 1 or 2 diabetes
  • Renal disease
  • Autoimmune disease
  • ≥2 moderate risk factors (nulliparity, obesity, family history, age ≥35, low socioeconomic status, African American race)

Calcium supplementation (1.5-2g elemental calcium daily) reduces preeclampsia risk in women with low dietary calcium intake (<800mg/day). 3, 8


HELLP Syndrome

Definition and Diagnostic Criteria

HELLP syndrome is a severe manifestation of preeclampsia characterized by Hemolysis, Elevated Liver enzymes, and Low Platelets, occurring in 0.5-0.9% of all pregnancies and 10-20% of severe preeclampsia cases. 3, 8, 7

The American College of Obstetricians and Gynecologists requires all three components for diagnosis: 8

Hemolysis:

  • Peripheral smear with schistocytes and fragmented RBCs 8
  • LDH >295 U/L (reflects both hemolysis and hepatic dysfunction) 8
  • Indirect bilirubin ≥18 µmol/L 8
  • Elevated fibrin degradation products (>80 µg/L) 8

Elevated Liver enzymes:

  • AST >70 U/L (Tennessee Classification) or >200 U/L in severe cases 8
  • ALT >159 U/L 8

Low Platelets:

  • <100,000/mm³ (Tennessee Classification threshold) 8

Pathophysiology

Defective placental perfusion leads to endothelial dysfunction and thrombotic microangiopathy. 7

  • Fibrin deposition in hepatic sinusoids causes sinusoidal obstruction and hepatic ischemia 7
  • Can progress to subcapsular hematomas, parenchymal hemorrhage, and hepatic rupture 7
  • 15% of HELLP cases occur without hypertension—do not exclude diagnosis based on normal blood pressure alone 7, 6

Clinical Presentation

Classic symptoms (present in 90%):

  • Right upper quadrant or epigastric pain (most common—65-90%)
  • Nausea/vomiting (45-85%)
  • Malaise (90%)
  • Headache (33-61%)

Physical examination:

  • Right upper quadrant tenderness
  • Hypertension (85% of cases)
  • Edema
  • Jaundice (rare, indicates severe disease)

Timing:

  • 70% develop antepartum (27-37 weeks most common)
  • 30% develop postpartum (usually within 48 hours but can occur up to 7 days)

Comprehensive Laboratory Workup

Initial assessment must include: 3, 8

  • Complete blood count with peripheral smear examination for schistocytes 8
  • Comprehensive metabolic panel: AST, ALT, LDH, total and indirect bilirubin 8
  • Coagulation studies: PT, aPTT, fibrinogen, fibrin degradation products 8
  • Renal function tests 8
  • Type and crossmatch (anticipate transfusion needs)

Serial monitoring every 6-12 hours for 24-48 hours postpartum 8

Management Priorities

Immediate stabilization:

  • Hospitalization in high-dependency or intensive care unit mandatory 3, 8
  • Large-bore IV access (two sites)
  • Continuous maternal and fetal monitoring

Blood pressure management:

  • Non-severe hypertension (140-159/90-109 mmHg): Oral labetalol, nifedipine, or methyldopa 3, 8
  • Severe hypertension (≥160/110 mmHg): Urgent IV treatment 3, 8
    • IV labetalol: 20mg bolus, then 40mg at 10 min, then 80mg q10min x2 (max 220mg) 3
    • IV hydralazine: 5mg bolus, then 10mg q20-30min (max 25mg) 3

Seizure prophylaxis:

  • Magnesium sulfate mandatory for all HELLP patients with co-existing severe hypertension 3, 8
  • Standard dosing: 4-6g IV load, then 2g/hour maintenance 3
  • Continue 24-48 hours postpartum 3

Management of coagulopathy:

  • Platelet transfusion indicated when count <100,000/mm³ due to increased risk of abnormal coagulation and adverse outcomes 3, 8
  • For cesarean section: Transfuse if platelets <50,000/mm³ 3, 8
  • Fresh frozen plasma for INR >1.5 or active bleeding
  • Cryoprecipitate for fibrinogen <200 mg/dL

Corticosteroid management:

  • Do NOT give corticosteroids to improve maternal HELLP outcomes—no proven benefit 3, 8
  • DO give betamethasone or dexamethasone for fetal lung maturity if delivery anticipated before 35 weeks 3, 8

Delivery decision-making:

  • Prompt delivery indicated once maternal coagulopathy and severe hypertension corrected 3, 8
  • Immediate delivery mandatory for: 8
    • Severe thrombocytopenia with progressive decline
    • DIC
    • Hepatic hematoma or rupture
    • Renal failure
    • Placental abruption
    • Non-reassuring fetal status
    • Eclampsia

Mode of delivery:

  • Vaginal delivery acceptable if cervix favorable and maternal/fetal status stable
  • Cesarean section for obstetric indications or maternal instability
  • Regional anesthesia contraindicated if platelets <70,000-80,000/mm³

Postpartum Monitoring and Recovery

Intensive monitoring required for 24-48 hours minimum: 3, 8

  • Central venous pressure monitoring 3
  • Hourly urine output 3
  • Continuous blood pressure and ECG monitoring 3
  • Oxygen saturation 3
  • Serial labs every 6-12 hours 8

Expected recovery timeline:

  • Platelet count nadirs 24-48 hours postpartum, then rises
  • LDH peaks at 24-48 hours postpartum
  • Liver enzymes normalize within days to weeks 3
  • Platelet count normalizes within 6-11 days 3

Hepatic Complications

Subcapsular hematoma and hepatic rupture are rare but life-threatening:

  • Perform abdominal ultrasound if severe right upper quadrant pain, shoulder pain, or hemodynamic instability 3, 8
  • Be especially vigilant with severe thrombocytopenia 3, 8
  • CT or MRI for definitive diagnosis
  • Surgical consultation mandatory
  • Management ranges from conservative (stable hematoma) to surgical intervention or hepatic artery embolization

Prevention in Subsequent Pregnancies

Women with prior HELLP syndrome have 19-27% recurrence risk and require high-risk obstetric care: 3, 8

  • First-trimester screening (11-14 weeks) to assess early-onset preeclampsia risk 3, 8
  • Aspirin prophylaxis 150mg nightly started before 16 weeks and continued until 36 weeks, delivery, or HELLP diagnosis 3, 8
  • Calcium supplementation (1.5-2g elemental calcium daily) if dietary intake <800mg/day 3, 8
  • Serial growth ultrasounds and antenatal testing
  • Delivery planning at tertiary care center

Key Clinical Pitfalls to Avoid

  • Do not dismiss symptoms in normotensive patients—15% of HELLP occurs without hypertension 7, 6
  • Do not delay delivery for corticosteroid administration in unstable patients 3
  • Do not use corticosteroids to treat maternal HELLP syndrome—only for fetal lung maturity 3, 8
  • Do not perform neuraxial anesthesia with platelets <70,000-80,000/mm³ 3
  • Do not discharge patients early—disease can worsen postpartum 8

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Preterm premature rupture of membranes: diagnosis, evaluation and management strategies.

BJOG : an international journal of obstetrics and gynaecology, 2005

Guideline

Management of Pre-eclampsia and HELLP Syndrome

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Continuing challenges in treating preterm labour: preterm prelabour rupture of the membranes.

BJOG : an international journal of obstetrics and gynaecology, 2006

Research

Preeclampsia: Diagnosis and management of the atypical presentation.

The journal of maternal-fetal & neonatal medicine : the official journal of the European Association of Perinatal Medicine, the Federation of Asia and Oceania Perinatal Societies, the International Society of Perinatal Obstetricians, 2006

Guideline

Pathophysiology and Clinical Characteristics of HELLP Syndrome

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Guideline

HELLP Syndrome Diagnostic and Management Guidelines

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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