What androgens do the adrenal glands produce?

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Adrenal Androgens Production

The adrenal glands primarily produce dehydroepiandrosterone (DHEA), DHEA sulfate (DHEAS), androstenedione, and 11β-hydroxyandrostenedione (11OHA4) as their main androgens. 1, 2

Primary Adrenal Androgens

The zona reticularis of the adrenal cortex is the specific anatomical site responsible for androgen synthesis 2, 3. The key androgens produced include:

  • DHEA - The most abundant circulating hormone in the human body, though biologically inactive until converted peripherally 2, 4
  • DHEAS - The sulfated form of DHEA, representing the most reliable measure of adrenal androgen production 1, 2
  • Androstenedione - A direct precursor of testosterone that can be produced in peripheral tissues from DHEA 1
  • 11β-hydroxyandrostenedione (11OHA4) - The second most abundant unconjugated androgen produced by human adrenals after DHEA 4

Emerging Understanding of 11-Oxygenated Androgens

Recent evidence has identified a class of adrenal-derived androgens termed "11-oxyandrogens" that share an oxygen atom on carbon 11 4. These include:

  • 11-ketotestosterone - A downstream metabolite of 11OHA4 produced mostly in peripheral tissues, with potency equivalent to testosterone 4
  • 11-ketodihydrotestosterone - The 5α-reduced product of 11-ketotestosterone, with potency equivalent to dihydrotestosterone 4

These 11-oxyandrogens have emerged as major components in disorders of androgen excess and, unlike traditional androgens, do not demonstrate age-dependent decline 4.

Clinical Relevance in Adrenal Insufficiency

In primary adrenal insufficiency (PAI), patients become deficient in all adrenal androgen secretion, particularly affecting female patients who lack gonadal androgen production 1. When monitoring adrenal androgen replacement:

  • DHEAS, androstenedione, and testosterone levels should be measured in the morning prior to DHEA ingestion and maintained in the normal range 1
  • DHEA replacement (10-50 mg, typically 25 mg daily) can be offered to female patients with persistent lack of libido or low energy despite optimized glucocorticoid and mineralocorticoid replacement 1

Regulation of Adrenal Androgen Production

ACTH is the major regulator of adrenal androgen secretion 5. However, production is also influenced by:

  • Gonadal sex steroids, insulin, growth hormone, and prolactin 5
  • The sympathoadrenal and immunoadrenal systems 5
  • Key enzymes including cytochrome b5, DHEA sulfotransferase, and 3β-hydroxysteroid dehydrogenase 2

Diagnostic Testing for Androgen Excess

When evaluating suspected adrenal androgen excess or virilization, testing should include 1:

  • DHEAS and testosterone as primary markers 1
  • Extended panel for suspected adrenocortical carcinoma: 17β-estradiol, 17-OH progesterone, androstenedione, 17-OH pregnenolone, 11-deoxycorticosterone, progesterone, and estradiol 1

Higher androgen levels suggest greater disease burden, particularly in adrenocortical carcinoma where androgen hypersecretion occurs in more than half of cases 1.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Research

Dissecting human adrenal androgen production.

Trends in endocrinology and metabolism: TEM, 2002

Research

The role for long-term use of dehydroepiandrosterone in adrenal insufficiency.

Current opinion in endocrinology, diabetes, and obesity, 2022

Research

11-Oxygenated androgens in health and disease.

Nature reviews. Endocrinology, 2020

Research

Adrenal androgens regulation and adrenopause.

Endocrine regulations, 2001

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Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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