Is serum cortisol elevated in patients with Cushing's disease?

Medical Advisory BoardAll articles are reviewed for accuracy by our Medical Advisory Board
Educational purpose only • Exercise caution as content is pending human review
Article Review Status
Submitted
Under Review
Approved

Last updated: January 21, 2026View editorial policy

Personalize

Help us tailor your experience

Which best describes you? Your choice helps us use language that's most understandable for you.

Serum Cortisol in Cushing's Disease

Yes, serum cortisol is elevated in Cushing's disease, though the elevation is best demonstrated through specific testing patterns rather than random serum measurements alone. 1, 2

Understanding Cortisol Elevation Patterns

Cushing's disease is characterized by chronic hypercortisolism with loss of normal circadian rhythm, which is the hallmark diagnostic feature. 1 The key abnormality is not simply that cortisol is high, but that it remains inappropriately elevated when it should be suppressed—particularly at night. 1

Specific Cortisol Patterns in Cushing's Disease

  • Morning serum cortisol levels may appear deceptively normal or only mildly elevated in some patients, as the normal reference range is 5-23 μg/dL (138-635 nmol/L). 3 This overlap with normal values makes random morning cortisol a poor screening test.

  • Late-night cortisol levels are the most diagnostically useful, as they should normally be <1.8 μg/dL (<50 nmol/L) in sleeping individuals. 3 Values of 290 and 275 nmol/L at night are significantly elevated and highly suggestive of Cushing's syndrome with loss of normal circadian rhythm. 1

  • 24-hour urinary free cortisol is markedly elevated, with mean total daily cortisol production reaching 46 mg (range 13.4-154 mg) in Cushing's disease patients compared to 12.8 mg (range 6.7-20 mg) in healthy controls. 4

Mechanism of Cortisol Elevation

The cortisol elevation in Cushing's disease results from both amplified pulsatile secretion and dramatically increased basal secretion rates. 4 Specifically:

  • Basal cortisol secretion rates are increased 7-fold above normal values in Cushing's disease patients. 4

  • Pulsatile cortisol secretion shows increased mass per pulse due to higher maximal secretion rates, with patients having more cortisol pulses per 24 hours (24 vs. 17 in controls). 4

  • The underlying cause is ACTH hypersecretion from a pituitary corticotroph adenoma, which drives the adrenal glands to overproduce cortisol. 2, 5

Diagnostic Testing Algorithm

To confirm elevated cortisol in suspected Cushing's disease, use the following approach:

  1. Screen with at least 2-3 of these tests (never rely on a single test): 1, 3

    • Late-night salivary cortisol (>3.6 nmol/L abnormal, sensitivity >90%) 3
    • 24-hour urinary free cortisol (>100 μg/24h diagnostic in symptomatic patients) 3
    • Overnight 1-mg dexamethasone suppression test (cortisol ≥1.8 μg/dL abnormal, >5 μg/dL indicates overt disease) 3
  2. Measure morning plasma ACTH once hypercortisolism is confirmed to determine if it's ACTH-dependent (>5 ng/L) or ACTH-independent (suppressed). 1, 6 In Cushing's disease, ACTH is detectable or elevated, with levels >29 ng/L having 70% sensitivity and 100% specificity for pituitary disease. 1, 6

  3. Proceed to pituitary MRI if ACTH is elevated, followed by bilateral inferior petrosal sinus sampling if imaging is inconclusive. 1, 6

Critical Pitfalls to Avoid

  • Do not rely on random morning serum cortisol alone, as values may overlap with normal ranges and miss the diagnosis. 3 The loss of circadian rhythm is more diagnostic than absolute cortisol levels.

  • Beware of false positives from oral contraceptives (which increase cortisol-binding globulin and falsely elevate total cortisol), severe obesity, depression, and alcoholism. 1, 3

  • Always obtain multiple screening tests (2-3 collections of each modality) due to substantial intra-patient variability and the possibility of cyclic Cushing's syndrome. 1, 3

  • Measure dexamethasone levels concomitantly during suppression testing to confirm adequate drug absorption and exclude false-positive results from CYP3A4 inducers. 1, 3

Rare Exception

In extremely rare cases, a patient with Cushing's disease may have a defect in 11β-hydroxysteroid dehydrogenase type 1 activity, which impairs cortisone-to-cortisol conversion and increases cortisol clearance, potentially masking the typical biochemical picture. 7 However, this is exceptionally uncommon and should not alter standard diagnostic approaches.

References

Guideline

Cushing's Syndrome Diagnosis and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Cortisol Levels and Diagnostic Guidelines

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Research

Cushing's disease.

Best practice & research. Clinical endocrinology & metabolism, 2009

Guideline

Cushing's Syndrome Classification and Diagnosis

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

Have a follow-up question?

Our Medical A.I. is used by practicing medical doctors at top research institutions around the world. Ask any follow up question and get world-class guideline-backed answers instantly.