What is the half-life of intraarterial verapamil in an adult patient?

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Half-Life of Intraarterial Verapamil

The half-life of intraarterial verapamil is 2 to 5 hours, based on the terminal elimination phase following intravenous administration. 1

Pharmacokinetic Profile

The FDA-approved drug label for verapamil specifies that intravenously administered verapamil exhibits bi-exponential elimination kinetics with two distinct phases: 1

  • Rapid distribution phase (alpha): approximately 4 minutes half-life 1
  • Terminal elimination phase (beta): 2 to 5 hours half-life 1

Since intraarterial administration is functionally equivalent to intravenous administration in terms of systemic pharmacokinetics (both bypass first-pass hepatic metabolism), the same elimination half-life applies. 1

Supporting Research Evidence

Multiple clinical studies confirm this elimination half-life range:

  • Intravenous verapamil studies in healthy volunteers demonstrate an elimination half-life of 2.0 hours after IV bolus administration (0.1 mg/kg). 2
  • Pharmacokinetic analyses in normal subjects show a beta-phase half-life ranging from 3.7 to 4.8 hours after intravenous doses. 3
  • Conventional formulation studies report a half-life (t1/2 beta) of 3-5 hours for intravenous verapamil. 4

Clinical Implications

Peak therapeutic effects occur within 3 to 5 minutes after bolus injection, reflecting the rapid distribution phase. 1 This rapid onset is why verapamil is effective for acute management of supraventricular arrhythmias when given intravenously or intraarterially. 5, 6

The relatively short elimination half-life means that:

  • Drug effects are transient after single bolus administration 1
  • Repeat dosing may be needed for sustained therapeutic effect 5
  • Continuous infusion (5-15 mg/h) may be required for ongoing rate control 5

Important Caveats

The elimination half-life can be significantly prolonged in specific patient populations:

  • Elderly patients: Aging may prolong the elimination half-life 1
  • Liver cirrhosis: Half-life increases four-fold (from 3.7 hours to 14.2 hours) in cirrhotic patients 7
  • Hepatic dysfunction: Severe liver disease can dramatically reduce verapamil clearance by 66% and increase half-life 8

Dose adjustments are critical in these populations to prevent accumulation and adverse effects, particularly hypotension and bradycardia. 9, 7

References

Research

Systemic availability of oral verapamil and effect on PR interval in man.

British journal of clinical pharmacology, 1981

Research

Pharmacokinetics of conventional and slow-release verapamil.

British journal of clinical pharmacology, 1986

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Management of Paroxysmal Supraventricular Tachycardia (PSVT)

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Guideline

Verapamil Dosage for Rate Control

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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