What is the half-life of paroxetine (selective serotonin reuptake inhibitor)?

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Paroxetine Half-Life

The elimination half-life of paroxetine is approximately 21 hours (with a coefficient of variation of 32%) after steady-state dosing, which supports once-daily administration. 1

Pharmacokinetic Profile

Steady-state concentrations are typically achieved by approximately 10 days for most patients, though it may take substantially longer in occasional patients. 1

  • At steady state following 30 mg daily dosing for 30 days, the mean elimination half-life was 21.0 hours (CV 32%). 1
  • The 24-hour half-life permits once-daily dosing in clinical practice. 2
  • Paroxetine has no active metabolites, as the principal metabolites have no more than 1/50 the potency of the parent compound at inhibiting serotonin uptake. 1

Clinical Implications of Half-Life

For therapeutic drug monitoring, blood samples should be collected at steady state (approximately 1 week after stable dosing), with sampling occurring 12-16 hours after the last dose or 24 hours if dosed once daily. 3

Nonlinear pharmacokinetics occur with paroxetine due to saturation of CYP2D6-mediated metabolism:

  • Steady-state drug exposure (AUC0-24) is about 8 times greater than predicted from single-dose data. 1
  • At steady state, Cmax and Cmin values are about 6 and 14 times what would be predicted from single-dose studies. 1
  • This excess accumulation results from saturation of one of the enzymes that metabolizes paroxetine. 1

Special Population Considerations

Elderly patients show 70-80% greater Cmin concentrations compared to non-elderly subjects at equivalent doses, necessitating initial dose reduction. 1

Renal impairment: Patients with creatinine clearance below 30 mL/min have approximately 4-fold greater mean plasma concentrations than normal volunteers, while those with creatinine clearance 30-60 mL/min show about 2-fold increases. 1

Hepatic impairment: Patients with hepatic functional impairment demonstrate about a 2-fold increase in plasma concentrations (AUC, Cmax). 1

Metabolism and Excretion

  • Approximately 64% of an oral dose is excreted in urine (2% as parent compound, 62% as metabolites) over 10 days post-dosing. 1
  • About 36% is excreted in feces (mostly as metabolites, less than 1% as parent compound). 1
  • Paroxetine metabolism is mediated in part by CYP2D6, and paroxetine itself is both a substrate and potent inhibitor of this enzyme. 1, 4

References

Research

An overview of paroxetine.

The Journal of clinical psychiatry, 1992

Guideline

Paroxetine Pharmacokinetics and Clinical Implications

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2025

Research

Paroxetine: a review.

CNS drug reviews, 2001

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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