How should I study PLS3 (Plastin 3) mutations?

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How to Study PLS3 Mutations

I'm sorry, but the provided evidence does not address how to study PLS3 mutations in a clinical or research context.

The evidence you've provided primarily discusses:

  • NASH/MASLD genetic targets (PNPLA3, HSD17B13, MTARC1, MBOAT7, GPAM, PSD3, CIDEB) 1, 2
  • POLE and POLD1 mutations in familial colorectal cancer 1
  • Clinical phenotypes and case reports of PLS3 mutations causing X-linked osteoporosis 3, 4, 5, 6, 7

None of these sources provide methodological guidance on how to study, investigate, or analyze PLS3 mutations.

What the Evidence Does Tell Us About PLS3

Clinical Context of PLS3 Mutations

  • PLS3 mutations cause X-linked early-onset osteoporosis with bone fragility, recurrent peripheral fractures, low bone mineral density, and vertebral compression fractures 3, 4, 6, 7
  • Hemizygous males are more severely affected than heterozygous females 3, 4
  • The protein is involved in actin bundle formation in the cytoskeleton 3, 5

Types of Mutations Identified

  • Splice site mutations (e.g., c.73-24T>A) that cause frameshift and premature termination 3
  • Whole gene deletions 4
  • Nonsense mutations (e.g., c.745G>T, p.E249X) 5
  • Frameshift deletions (e.g., c.994_995delGA; p.Asp332*) 7
  • Missense mutations (e.g., c.1433T>C; p.Leu478Pro) 7

Diagnostic Methods Mentioned (But Not Detailed)

  • Whole-exome sequencing was used in one family 3
  • Sanger sequencing for confirmation 3
  • Targeted gene sequencing 5
  • cDNA sequencing to confirm splice effects 3

What You Actually Need

To properly answer "how to study PLS3 mutations," you would need evidence covering:

  • Genetic testing methodologies and sequencing strategies
  • Functional assays for PLS3 protein activity
  • Cell culture models for studying PLS3 function
  • Animal models (zebrafish were mentioned briefly 6 but not detailed)
  • Bioinformatic prediction tools for variant pathogenicity
  • Bone histomorphometry protocols specific to PLS3-related disease
  • Clinical phenotyping protocols and diagnostic criteria

The current evidence set does not provide this information.

References

Guideline

Guideline Directed Topic Overview

Dr.Oracle Medical Advisory Board & Editors, 2025

Guideline

Role of PNPLA3 Gene in MASLD/MASH Development and Management

Praxis Medical Insights: Practical Summaries of Clinical Guidelines, 2026

Research

A novel splice mutation in PLS3 causes X-linked early onset low-turnover osteoporosis.

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, 2015

Research

PLS3 mutations in X-linked osteoporosis with fractures.

The New England journal of medicine, 2013

Research

Osteoporosis caused by mutations in PLS3: clinical and bone tissue characteristics.

Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research, 2014

Professional Medical Disclaimer

This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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