Management of Acute Supraventricular Tachycardia in a Hypotensive Patient
Proceed directly to synchronized cardioversion for any patient with supraventricular tachycardia presenting with hypotension, as this represents hemodynamic instability requiring immediate electrical therapy. 1, 2
Defining Hemodynamic Instability
Your patient meets criteria for hemodynamic instability based on hypotension alone. Additional signs that would also mandate immediate cardioversion include: 2
- Systolic blood pressure <90 mmHg (your patient)
- Syncope or altered mental status
- Signs of myocardial ischemia (chest pain with ST-segment changes)
- Acute pulmonary edema or severe heart failure
- Signs of shock
Immediate Management Algorithm
Step 1: Prepare for Synchronized Cardioversion
Synchronized cardioversion is the Class I, Level B recommendation for hemodynamically unstable SVT, with initial energy of 50-100 J. 2 This achieves near 100% termination of SVT in unstable patients. 3, 2
- Have defibrillator and emergency equipment immediately available 4
- Provide procedural sedation if the patient is conscious 2
- Ensure continuous ECG monitoring and frequent blood pressure measurement 4
Step 2: Consider One Dose of Adenosine (Optional, Only If Specific Criteria Met)
If the tachycardia is regular with a narrow QRS complex and the patient is not in frank shock, you may attempt one dose of adenosine 6 mg rapid IV push before cardioversion. 2, 5 However, this is optional and should not delay cardioversion. 1, 3
Critical safety requirement: Cardioversion equipment must be immediately available at bedside because adenosine can precipitate atrial fibrillation with rapid ventricular response, potentially triggering ventricular fibrillation. 2, 5
Adenosine achieves 90-95% success for AVNRT and orthodromic AVRT even in unstable patients. 2, 5 If you attempt adenosine:
- Administer 6 mg rapid IV push through a large proximal vein 3, 2
- Follow immediately with 20 mL saline flush 3
- If no conversion within 1-2 minutes, give 12 mg IV push 2
- Do not give a third dose in an unstable patient—proceed to cardioversion 2
Step 3: What NOT to Do in Hypotensive SVT
Never administer calcium channel blockers (diltiazem, verapamil) or beta-blockers to hypotensive patients—these can precipitate cardiovascular collapse. 3, 2
- Diltiazem and verapamil are absolutely contraindicated in hemodynamically unstable SVT 2
- IV metoprolol will worsen hypotension 2
- These agents are only appropriate for hemodynamically stable patients 1, 4
Special Diagnostic Considerations
Rule Out Pre-Excited Atrial Fibrillation
If the ECG shows irregular wide-complex tachycardia or you suspect Wolff-Parkinson-White syndrome with atrial fibrillation: 2
- Proceed immediately to synchronized cardioversion without attempting adenosine 2
- Do not use any AV nodal blocking agents (adenosine, calcium channel blockers, beta-blockers, digoxin) as these may enhance accessory pathway conduction and precipitate ventricular fibrillation 2
Obtain 12-Lead ECG
A 12-lead ECG during tachycardia is essential to differentiate SVT from ventricular tachycardia and identify pre-excitation. 3 A narrow QRS complex confirms supraventricular origin (likely AVNRT or orthodromic AVRT). 2
Post-Cardioversion Management
After successful cardioversion, anticipate: 2
- Premature atrial or ventricular complexes that may reinitiate tachycardia 2
- Have antiarrhythmic drugs ready to prevent acute reinitiation if recurrence occurs 2
- Continue continuous cardiac monitoring 3
Common Pitfalls to Avoid
Do not attempt vagal maneuvers in hypotensive patients—these waste time and are ineffective in unstable SVT. Vagal maneuvers are only for hemodynamically stable patients. 1, 3
Do not use procainamide—this is indicated for pre-excited atrial fibrillation, not regular narrow-complex SVT, and is too slow-acting for a patient in shock. 2
Do not delay cardioversion to obtain additional history, perform extensive workup, or attempt multiple medication trials. 1, 2
Do not confuse "low blood pressure" with "stable but anxious"—true hypotension (<90 mmHg systolic) mandates immediate cardioversion regardless of patient appearance. 2