In chronic myeloid leukaemia, what findings are classically seen on the peripheral blood film?

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Classic Peripheral Blood Film Findings in Chronic Myeloid Leukemia

The peripheral blood film in chronic phase CML classically shows marked leukocytosis with left-shifted granulopoiesis at all stages of maturation (myeloblasts through mature neutrophils), accompanied by basophilia, eosinophilia, and thrombocytosis. 1

Characteristic White Blood Cell Findings

The hallmark feature is excessive left-shifted granulopoiesis detectable at all stages of maturation, meaning you will see the entire spectrum of myeloid cells from blasts to mature neutrophils on a single smear. 1 This creates a characteristic "myelocyte bulge" where myelocytes and metamyelocytes are prominently increased. 1

  • Myelocytes and metamyelocytes are typically abundant (often 20-40% of white cells), creating the distinctive left shift that differentiates CML from reactive leukocytosis. 2
  • Blast cells are present but remain <10% in chronic phase, distinguishing it from accelerated phase (10-19% blasts) or blast crisis (≥20% blasts). 1
  • Promyelocytes are visible but usually comprise only 1-5% of cells. 2

Basophilia and Eosinophilia

Basophilia is a typical and diagnostically important feature of chronic phase CML, often comprising 3-10% or more of the differential count. 1 The absence of basophils should raise suspicion for atypical CML or other diagnoses, though rare CML cases can present without basophilia. 2

  • Eosinophilia is also commonly present, contributing to the overall granulocytic expansion. 1
  • Basophil percentage >20% indicates accelerated phase rather than chronic phase. 1

Platelet Findings

Thrombocytosis (elevated platelet count) is a typical feature of chronic phase CML at presentation, often with counts exceeding 450-600 × 10⁹/L. 1 In some cases, marked thrombocytosis may mimic essential thrombocythemia clinically, making Philadelphia chromosome testing mandatory. 3

Red Blood Cell Findings

  • Nucleated red blood cells may be present on the peripheral smear, reflecting the myeloproliferative nature of the disease. 4
  • Anemia is variable; some patients present with normal hemoglobin while others have mild to moderate anemia. 4

Critical Diagnostic Pitfall

Do not diagnose CML based solely on peripheral blood morphology—the Philadelphia chromosome (t(9;22)) or BCR-ABL1 fusion gene must be demonstrated to confirm the diagnosis. 1, 5 Approximately 5% of cases lack a visible Philadelphia chromosome on conventional cytogenetics and require FISH or RT-PCR for BCR-ABL1 detection. 1

Atypical CML (BCR-ABL1 negative) lacks basophilia and shows more pronounced dysplasia, distinguishing it from classic Philadelphia-positive CML. 1

Distinguishing CML from Leukemoid Reaction

The combination of left shift extending to myelocytes/promyelocytes, basophilia, eosinophilia, and thrombocytosis strongly favors CML over a reactive leukocytosis. 1 Reactive processes rarely produce significant basophilia or the full spectrum of myeloid maturation seen in CML. 6

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This information is intended for healthcare professionals. Any medical decision-making should rely on clinical judgment and independently verified information. The content provided herein does not replace professional discretion and should be considered supplementary to established clinical guidelines. Healthcare providers should verify all information against primary literature and current practice standards before application in patient care. Dr.Oracle assumes no liability for clinical decisions based on this content.

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